Complications of Pregnancy Flashcards
Characteristic Causes of High Risk Pregnancies
Can relate to the pregnancy itself
Can occur because the woman has a medical condition or injury that complicates the pregnancy
Can result from environmental hazards that can affect the mother or her fetus
Can arise from maternal behaviour or lifestyles that have a negative effect on the mother or fetus
Danger Signs in Pregnancy
Sudden gush of fluid from the vagina Vaginal bleeding Abdominal pain Persistent vomiting Epigastric pain Edema of face and hands Severe, persistent headache Blurred vision or dizziness Chills with fever over 38ºC Painful urination or reduced urine output
Pregnancy- Related Complications
Hyperemesis Gravidarum
Bleeding disorders: Abortion, ectopic pregnancy, placenta previa and abruptio
Hypertension
Blood incompatibility between the woman and fetus - Erythroblastosis Fetalis
Diabetes
Infections
Hyperemesis Gravidarum: Manifestations
Excessive N/V Significant weight loss Dehydration Electrolyte and Acid-Base imbalances Reduced delivery of blood, oxygen, and nutrients to the fetus Psychological Factors Can affect fetal growth
Types of Abortion: Spontaneous, Induced
Spontaneous (non intentional): - Threatened - Inevitable - Incomplete - Complete - Missed - Recurrent Induced: - Therapeutic - Elective
Ectopic Pregnancy
95% occur in fallopian tube Scarring or tubal deformity may result from: - Hormonal abnormalities - Inflammation - Infection - Adhesions - Congenital defects - Endometriosis
Ectopic Pregnancy: Manifestations
Lower abdominal pain, may have light vaginal bleeding
If tube ruptures:
- May have sudden severe lower abdominal pain
- Vaginal bleeding
- Signs of hypovolemic shock
- Shoulder pain may also be felt
S&S of Hypovolemic Shock
Changes in fetal heart rate (increased, decreased, less fluctuation)
Rising, weak pulse (tachycardia)
Rising respiratory rate (tachypnea)
Shallow, irregular respirations (air hunger)
Falling BP (hypotension)
Decreased, or absent urinary output (less than 30 mL/hr)
Pale skin of pale mucous membranes
Cold, clammy skin
Faintness
Thirst
Bleeding Disorders of late pregnancy
Placenta previa
- Abnormal implantation of the placenta
- Bright bleeding occurs when cervix dilates resulting in PAINLESS bleeding
Abruptio placentae
- Normal implantation of placenta
- Dark bleeding with PAIN, and enlarging uterus suggests blood is accumulating in the cavity
Complications or Risks: Placenta previa
Infection, because of vaginal organisms
Postpartum hemorrhage, because if lower segment of uterus is the site of attachment, then there are fewer muscle fibres so weaker contractions may occur
Complications or Risks: Abruptio placentae (Predisposing Factors)
Hypertension Cocaine or alcohol use Cigarette smoking and poor nutrition Blows to the abdomen Prior history of abruptio placentae Folate deficiency *MAY BE ACCOMPANIED BY DIC - A SERIOUS CLOTTING DISORDER
Hypertension During Pregnancy
Gestational hypertension (GH previously called toxemia)
- BP >140/90 in a normotensive woman after 20 weeks
Preeclampsia
- GH plus renal involvement with proteinuria
Eclampsia
- Preeclampsia plus CNS involvement with seizures/ convulsions, serious liver and coagulation issues
Chronic Hypertension
- Hypertension present before 20 weeks
Preeclampsia with superimposed chronic hypertension
- New occurrence of proteinuria or thrombocytopenia before pregnancy
Risk Factors for GH
- First Pregnancy
- Obesity
- Family hx of GH
- Age >40 or >19
- Multifetal pregnancy
- Chronic hypertension
- Chronic renal disease
- DM
Manifestations of GH
- Hypertension
- Edema above the waste
- Sudden, excessive weight gain
- Proteinuria
- CNS
- Eyes
- Urinary tract
- Respiratory system
- GI system and liver
- Blood clotting; HELLP Syndrome
Bleeding Incompatibilities
Rh- negative blood type is an autosomal recessive trait
Rh - positive blood type is a dominant trait
Rh - incompatibility can only occur if the woman is Rh- negative and the fetus is Rh- positive
Isoimmunization
The leaking of fetal Rh- positive blood into the Rh-negative mother’s circulation, causing her body to respond by making antibodies to destroy the Rh- positive erythrocytes. Therefore, with subsequent pregnancy, the woman’s antibodies against Rh- positive blood cross the placenta and destroy the petals Rh- positive erythrocytes before the infant is born
Erythroblastosis Fetalis
Occurs when the maternal anti-Rh antibodies cross the placenta and destroy fetal erythrocytes
Requires RhoGAM to be given at 28 weeks and within 72 hours of delivery to the mother
- Also given after amniocentesis, and if woman experiences bleeding during pregnancy
Fetal assessment tests must be done throughout pregnancy
An intrauterine transfusion may be done for the severely anemic fetus
Diabetes Mellitus
Inadequate insulin to move glucose into the blood
Type 1 - insulin dependency
Type 2 - insulin resistance
GDM is classified as preceding or occurring during pregnancy
Diabetes Mellitus
In order to dilute glucose in the blood, thirst is increased (polyphasia)
Fluid moves from tissue into blood (to dilute)
Tissue dehydration and increased urine output occurs (polyuria with glucosuria)
Lose weight despite eating large amounts of food (polyphagia)
Fatigue and lethargy occurs (cell starvation)
Diabetes Mellitus
The pancreas - little or no insulin
Cells starve as they cannot obtain insulin
To compensate the body metabolizes protein and fat for energy which causes ketones and acids to accumulate (ketoacidosis)
Diabetes Mellitus
Gestational Diabetes Mellitus
Glucose intolerance with onset during pregnancy
In true GDM, glucose usually returns to normal 6 weeks postpartum