clinical neurology soal 191-215 Flashcards
antinuclear antibodies and malar rash A. Cogan's syndrome B. polyarteritis nodosa C. systemic lupus erythematosus D. Takayasu's syndrome E. temporal arteritis F. Wegener's granulomatosis
C. systemic lupus erythematosus
visual loss and claudication with chewing A. Cogan's syndrome B. polyarteritis nodosa C. systemic lupus erythematosus D. Takayasu's syndrome E. temporal arteritis F. Wegener's granulomatosis
E. temporal arteritis
visual loss and loss of peripheral pulses A. Cogan's syndrome B. polyarteritis nodosa C. systemic lupus erythematosus D. Takayasu's syndrome E. temporal arteritis F. Wegener's granulomatosis
D. Takayasu’s syndrome
mononeuritis multiplex, kidney involvement, and skin purpura A. Cogan's syndrome B. polyarteritis nodosa C. systemic lupus erythematosus D. Takayasu's syndrome E. temporal arteritis F. Wegener's granulomatosis
B. polyarteritis nodosa
deafness and keratitis A. Cogan's syndrome B. polyarteritis nodosa C. systemic lupus erythematosus D. Takayasu's syndrome E. temporal arteritisF. Wegener's granulomatosis
A. . Cogan’s syndrome
Wernicke’s encephalopathy consists of all of the following except
A. defect in retentive memory out of proportion to other cognitive functions
B. gait ataxia
C. gaze palsy
D. mental confusion
E. nystagmus
A. defect in retentive memory out of proportion to other cognitive functions
V&A pp. 1206-1207. Defects in learning and memory out of proportionto other cognitive functions is a feature of Korsakoffs psychosis, notWernicke’s encephalopathy.
Which of the following is least suggestive of a parietal lobe lesion? A. astereognosis B. loss of position sense C. loss of temperature sensation D. loss of two-point discrimination E. atopognosia
C. loss of temperature sensation
V&A pp. 483-487. Parietal lobe lesions are characterized by loss of positionsense, impaired ability to localize touch and pain stimuli (atopognosia),astereognosis, and impairment of two-point discrimination. Perception ofpain, touch, pressure, vibratory, and thermal stimuli is relatively intact.
The purest form of achromatopsia is caused by a lesion involving the
A. left calcarine cortex
B. left superior occipitotemporal region
C. right inferior occipitotemporal region
D. right occipital cortex and angular gyrus
E. right superior calcarine cortex
C. right inferior occipitotemporal region
V&A p. 269. A lesion in the right inferior occipitotemporal region sparing theoptic radiation and striate cortex causes the purest form of achromatopsia.
Failure of a miotic pupil to dilate after instilling 2 to 10% cocaine followed by1% hydroxyamphetamine indicates a
A. first-order Horner’s syndrome
B. second-order Horner’s syndrome
C. third-order Horner’s syndrome
D. first- or second-order Horner’s syndrome
E. second- or third-order Horner’s syndrome
C. third-order Horner’s syndrome
V&A pp. 298-299. Horner’s syndrome can be confirmed by the failure of themiotic pupil to dilate in response to 2 to 10% cocaine drops. If the later applicationof the adrenergic mydriatic hydroxyamphetamine has no effect,then the lesion localizes to the third-order neuron (p ostganglionic part). A first- or second-order lesion is indicated by a failure of the miotic pupilto dilate to cocaine drops, followed by dilation (after 24 hours) with 1%hydroxyamphetamine.
Somnambulism occurs in which stage of sleep? A. stage 1 B. stage 2 C. stage 4 D. REM E. all of the above
C. stage 4
The most effective treatment of enuresis is A. clonopin B. clonidine C. haloperidol (Haldol) D. imipramine (Tofranil) E. methylphenidate (Ritalin)
D. imipramine (Tofranil)
In most caws, section of the corpus callosum causes A. apraxia of both hands to command B. apraxia of the left hand to command C. apraxia of the right hand to command D. obiect agnosia E. no deficit
B. apraxia of the left hand to command
Broca’s aphasia
A. good comprehension, fluent speech, poor repetition
B. good comprehension, nonfluent speech, good repetition
C. good comprehension, nonfluent speech, poor repetition
D. poor comprehension, fluent speech, good repetition
E. poor comprehension, fluent speech, poor repetition
F. poor comprehension, nonfluent speech, poor repetition
C. good comprehension, nonfluent speech, poor repetition
conduction aphasia
A. good comprehension, fluent speech, poor repetition
B. good comprehension, nonfluent speech, good repetition
C. good comprehension, nonfluent speech, poor repetition
D. poor comprehension, fluent speech, good repetition
E. poor comprehension, fluent speech, poor repetition
F. poor comprehension, nonfluent speech, poor repetition
A. good comprehension, fluent speech, poor repetition
global aphasia
A. good comprehension, fluent speech, poor repetition
B. good comprehension, nonfluent speech, good repetition
C. good comprehension, nonfluent speech, poor repetition
D. poor comprehension, fluent speech, good repetition
E. poor comprehension, fluent speech, poor repetition
F. poor comprehension, nonfluent speech, poor repetition
F. poor comprehension, nonfluent speech, poor repetition
