Classical Pt 2 Flashcards

1
Q

Where is the classical lesion location for Broca’s aphasia

A

posterior inferior frontal convolution and adjacent inferior aspects of pre central gyrus

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2
Q

Where is the classical lesion location for Wernicke’s aphasia

A

posterior half of superior temporal gyrus and possibly adjacent cortex

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3
Q

Where is the classical lesion location for Anomic aphasia

A

inferior parietal lobe or connections between parietal lobe and temporal lobe

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4
Q

Where is the classical lesion location for global aphasia

A

large portion of the perisylvian association cortex

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5
Q

Where is the classical lesion location for conduction aphasia

A

lesion in arcuate fasiculus and/or corticocortical connections between temporal and frontal lobes

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6
Q

Where is the classical lesion location for transcortical motor aphasia

A

white matter tracts deep to broca’s area

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7
Q

Where is the classical lesion location for transcortical sensory aphasia

A

white matter tracts connecting parietal lobe to temporal lobe or in portions of inferior parietal lobe

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8
Q

Where is the classical lesion location for subcortical aphasia

A
  1. thalamus

2. caudate, putamen, internal capsule, basal ganglia

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9
Q

In the Wernicke-lichtheim model, auditory input -> auditory word-impressions -> elaboration of concepts =?

A

auditory comprehension

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10
Q

In the Wernicke-lichtheim model, elaboration of concepts -> kinaesthetic word-impressions -> motor output to articulators =

A

speech

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11
Q

In the Wernicke-lichtheim model, auditory input -> auditory word-impressions -> kinaesthetic word-impressions -> motor output to articulators =

A

repetition

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12
Q

In the Wernicke-lichtheim model, damage at the auditory word-impression level leads to what?

A

wernicke’s aphasia

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13
Q

In the Wernicke-lichtheim model, damage at the kinaesthetic word impressions level leads to what?

A

broca’s aphasia

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14
Q

In the Wernicke-lichtheim model, damage between auditory word-impressions and kinesthetic word impressions leads to what?

A

conduction aphasia

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15
Q

Which type of aphasia is associated with the poorest prognosis?

A

Global/severe aphasia

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16
Q

In individuals with global aphasia, this most likely to improve are those with…

A
  • lesions that are mainly subcortical
  • an absence of damage to Wernicke’s area
  • and an early corresponding improvement in auditory comprehension.
17
Q

In individuals with global aphasia, rapid changes in _______________ in conversation predicted whether a patient remained globally aphasic or not

A

verbal expression

18
Q

In individuals with global aphasia, pockets of improvement can occur in auditory comprehension and gesturing with treatment, between _______ months

A

6-12

19
Q

In individuals with Wernicke’s aphasia, individuals with higher test scores and less ______ progress better over time

A

jargon

20
Q

In general in individuals with Wernicke’s aphasia, the larger the lesion in Wernicke’s area and surrounding area, the _______ the prognosis

A

poorer

21
Q

Which type of aphasia is associated with the greatest amount of recovery

A

-Broca’s aphasia

22
Q

In individual’s with conduction aphasia, the amount of recovery is comparable to _________ aphasia

A

Broca’s

23
Q

Most anomic, conduction and transcortical aphasics had ______________ outcomes

A

excellent

24
Q

Some patients with conduction aphasia evolve to ______ aphasia

A

anomic