Circulation Flashcards
fluids in cavities
effusions
anasarca
severe generalized edema, especially of subcutaneous tissue
dependent edema
distribution by gravity
pitting edema
finger pressure leaves a depression
transudate
fluid of low protein content
exudate
fluid of high protein content
ascites
excessive peritoneal fluid
hydrothorax
excessive pleural fluid
hydrarthorsis
excessive joint fluid
two mechanisms that cause generalized edema
1 insufficient serum protein b/c of decreased manufacture or loss (renal disease)
2 increased hydrostatic pressure in blood capillaries
three mechanisms that cause localized edema
1 increased hydrostatic pressure in blood capillaries - venous
2 obstructoins of lymphatics
3 increased permeability of blood capillaries- inflammatoin
what does edema look like on a slide
eosinophilic separation of tisue elements
hyperemia (congestion)
increased blood volume in vascular space
active vs hyperemia
increased inflow v decreased outflow
hyperemia morphology
vascular engorgement
hemorrhage
flow of blood out of the vascular compartment
petechiae
minute hemorrhages in the skin, muscus membranes, or serosal surfaces
purpura
slightly larger hemorrhages of surfaces
ecchymosis
large hemorrhages of surfaces
hematoma
pool of extravascular blood trapped in the tissues
hemoptysis
coughing up blood arising from larynx, trachea, bronchi, or lung
hematemesis
vomiting blood
melena
evacuation of tar like stools of altered blood
causes of local hemorrhage
trauma
infections
degeneration
neoplasia
causes of general hemorrhage
hemorrhagic diathesis (coagulatoin or vascular defects)
anemia
reduciotn in number/volume of erythrocytes per unit volume of blood caused by decreased production or increased loss of RBCs
ischemia vs infarct
ishemia- partial to complete reduction of blood resulting in hypoxia or anoxia
infarct- focus of necrosis, usually coagulative, resulting from ischemia
factors that affect the development of ischemia/infarct
1 supply of blood/oxygen
2 vascular pattern
3 rate of decrease of blood flow
4 tissue vulnerablity
types of infarct
anemic- white/pale
hemorrhagic- red
morphology of ischemia/infarct
muscle necrosis
neutrophil/macrophage
fibroblasts/collagen
heart failure
failure of pump action of heart
backward failure vs forward
failure to pump out of veins
failure to pump blood to meet needs of body
congestive heart failure
“backwards” failure
right sided- RV fails to empty veins, resulting in an increase in pressure in the atria, venous system, and capillaries
left sided- LV fails to empty pulmonic veins, so pressure builds in pulmonic circulation in addition to above
signs of left sided v right sided heart failure
left- dilated left ventricle, pulmonary edema
right- dilated right ventricle, systemic edema
pleural effusions in congestive heart failure
can result from either left or right sided heart failure