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NASM-CPT > Chapter 7: Flexibility Training Concepts > Flashcards

Chapter 7: Flexibility Training Concepts Flashcards

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1
Q

flexibility

A

the normal extensibility of all soft tissues that allows the full range of motion of a joint

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2
Q

extensibility

A

capability to be elongated or stretched

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3
Q

dynamic range of motion

A
  • optimal control of movement throughout a joint’s entire range of motion
  • the combination of flexibility and the nervous system’s ability to control this range of motion efficiently
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4
Q

neuromuscular efficiency

A

the ability of the nervous system to recruit the correct muscles (agonists, antagonists, synergists, and stabilizers) to produce force (concentrically), reduce force (eccentrically), and dynamically stabilize (isometrically) the body’s structure in all three planes of motion

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5
Q

postural distortion patterns

A
  • predictable patterns of muscle imbalances

- patterns that develop if one or more segments of the HMS are misaligned and not functioning properly

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6
Q

relative flexibility

A
  • the tendency of the body to seek the path of least resistance during functional movement patterns
  • aka altered movement patterns
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7
Q

muscle imbalance

A

alteration of muscle length surrounding a joint

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8
Q

reciprocal inhibition

A
  • the simultaneous contraction of one muscle and the relaxation of its antagonist to allow movement to take place
  • a naturally occurring phenomenon that allows movement to take place
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9
Q

altered reciprocal inhibition

A
  • the concept of muscle inhibition, caused by a tight agonist, which inhibits its functional antagonist
  • caused by a tight agonist muscle decreasing the neural drive to its functional antagonist
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10
Q

synergistic dominance

A

the neuromuscular phenomenon that occurs when inappropriate muscles take over the function of a weak or inhibited prime mover

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11
Q

arthrokinematics

A

the motions of joints in the body

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12
Q

arthrokinetic dysfunction

A
  • altered forces at the joint that result in abnormal muscular activity and impaired neuromuscular communication at the joint
  • biomechanical and neuromuscular dysfunction leading to altered joint motion
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13
Q

autogenic inhibition

A
  • the process by which neural impulses that sense tension are greater than the impulses that cause muscles to contract, providing an inhibitory effect to the muscle spindles
  • “autogenic” because the contracting muscle is being inhibited by its own receptors
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14
Q

pattern overload

A

consistently repeating the same pattern of motion, which may place abnormal stresses on the body

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15
Q

Davis’s law

A

states that soft tissue models along the lines of stress

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16
Q

static stretching

A

the process of passively taking a muscle to the point of tension and holding the stretch for a minimum of 30 seconds

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17
Q

active-isolated stretch

A

the process of using agonists and synergists to dynamically move the joint into a range of motion

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18
Q

dynamic stretch

A

the active extension of a muscle, using force production and momentum, to move the joint through the full available range of motion

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19
Q

what is range of motion dictated by?

A

the normal extensibility of all soft tissues surrounding it

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20
Q

what is an important characteristic of soft tissue?

A

it will only achieve efficient extensibility if optimal control of movement is maintained throughout the entire ROM

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21
Q

factors that can influence flexibility

A
  • genetics
  • connective tissue elasticity
  • composition of tendons or skin surrounding the joint
  • joint structure
  • strength of opposing muscle groups
  • body composition
  • sex
  • age
  • activity level
  • previous injuries or existing medical issues
  • repetitive movements (pattern overload)
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22
Q

what must individuals have to allow for optimal neuromuscular efficiency?

A
  • proper flexibility in all three planes of motion

- this allows for the freedom of movement needed to perform everyday activities effectively

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23
Q

flexibility requires ___, which requires ___, which requires _____

A

flexibility requires extensibility, which requires dynamic range of motion, which requires neuromuscular efficiency

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24
Q

flexibility training must use a ____ approach, which integrates various flexibility techniques to achieve optimal soft tissue extensibility in all planes of motion

A

multifaceted

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25
Q

agonist in a cable pulldown exercise

A

latissimus dorsi

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26
Q

what does the agonist concentrically accelerate in a cable pulldown exercise?

A

shoulder extension, adduction, and internal rotation

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27
Q

synergists in a cable pulldown exercise

A

middle and lowerr trapezius and rhomboids

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28
Q

what do the synergists do in a cable pulldown exercise?

A

perform downward rotation of the scapulae

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29
Q

stabilizers in a cable pulldown exercise

A

rotator cuff musculature

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30
Q

what do the stabilizers do in a cable pulldown exercise?

A

dynamically stabilize the glenohumeral (shoulder) joint throughout the motion

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31
Q

human movement system (HMS)

A
  • also known as the kinetic chain

- comprises the muscular, skeletal, and nervous systems

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32
Q

latissimus dorsi - sagittal movement

A

must have proper extensibility to allow for proper shoulder flexion

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33
Q

latissimus dorsi - frontal movement

A

must have proper extensibility to allow for proper shoulder abduction

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34
Q

latissimus dorsi - transverse movement

A

must have proper extensibility to allow for proper external humerus rotation

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35
Q

biceps femoris - sagittal movement

A

must have proper extensibility to allow for proper hip flexion, knee extension

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36
Q

biceps femoris - frontal movement

A

must have proper extensibility to allow for proper hip adduction

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37
Q

biceps femoris - transverse movement

A

must have proper extensibility to allow for proper hip and and knee internal rotation

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38
Q

gastrocnemius - sagittal movement

A

must have proper extensibility to allow for proper dorsiflexion of ankle

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39
Q

gastrocnemius - frontal movement

A

must have proper extensibiltiy to allow for proper inversion of calcaneus

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40
Q

gastrocnemius - transverse movement

A

must have proper extensibility to allow for proper internal rotation of femur

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41
Q

muscle imbalances lead to ___, which lead to ___, which lead to ___

A

muscle imbalances lead to poor posture, which lead to improper movement, which lead to injury

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42
Q

what are postural distortion patterns represented by?

A

a lack of structural integrity, resulting from decreased functioning of one (or more) components of the HMS

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43
Q

what can a lack of structural integrity result in?

A
  • altered length-tension relationships (altered muscle lengths)
  • altered force-couple relationships (altered muscle activation)
  • altered arthrokinematics (altered joint motion)
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44
Q

maximal neuromuscular efficiency can only exist if what?

A

all components (muscular, skeletal, and neural) function optimally and interdependently

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45
Q

what is the ultimate goal of the HMS?

A

to maintain homeostasis (or dynamic postural equilibrium)

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46
Q

what can poor flexibility lead to?

A

the development of relative flexibility (or altered movement patterns)

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47
Q

relative flexibility example in the squat

A
  • feet externally rotated
  • because most people have tight calf muscles, they lack the proper amount of dorsiflexion at the ankle to perform a squat with proper mechanics
  • by widening the stance and externally rotating the feet, it is possible to decrease the amount of dorsiflexion required at the ankle to perform a squat using good technique
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48
Q

relative flexibility example in the overhead shoulder press

A
  • excessive lumbar extension (arched lower back)
  • individuals who possess a tight latissimus dorsi will have decreased sagittal-plane shoulder flexion (inability to life arms directly overhead), and as a result, they compensate for this lack of range of motion at the shoulder in the lumbar spine to allow them to press the load completely above their head
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49
Q

overactive muscle imbalances

A

forcing compensation to occur

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50
Q

underactive muscle imbalances

A

allowing for the compensation to occur

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51
Q

mechanisms that may cause muscle imbalance

A
  • postural stress
  • emotional duress
  • repetitive movement
  • cumulative trauma
  • poor training technique
  • lack of core strength
  • lack of neuromuscular efficiency
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52
Q

muscle imbalances may be caused by or result in the following:

A
  • altered reciprocal inhibition
  • synergistic dominance
  • arthrokinetic dysfunction
  • overall decreased neuromuscular control
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53
Q

example of reciprocal inhibition in the biceps curl

A

to perform elbow flexion during the biceps curl, the biceps brachii actively contracts while the triceps brachii (the antagonist muscle) relaxes to allow mvoement to occur

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54
Q

example of altered reciprocal inhibition in the hips

A

a tight psoas (hip flexor) would decrease neural drive of the gluteus maximus (hip extensor)

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55
Q

what does altered reciprocal inhibition alter?

A

force-couple relationships

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56
Q

what does altered reciprocal inhibition produce?

A

synergistic dominance

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57
Q

what does altered reciprocal inhibition lead to the development of?

A
  • faulty movement patterns
  • poor neuromuscular control
  • arthrokinetic (joint) dysfunction
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58
Q

example of synergistic dominance in the hips

A

if the psoas is tight, it leads to altered reciprocal inhibition of the gluteus maximus, which in turn results in increased force output of the synergists for hip extension (hamstring complex, adductor magnus) to compensate for the weakened gluteus maximus

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59
Q

result of synergistic dominance

A

faulty movement patterns, leading to arthrokinetic dysfunction and eventual injury (such as hamstring strains)

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60
Q

what can altered joint motion be caused by?

A

altered length-tension relationships and force-couple relationships, which affect the joint and cause poor movement efficiency

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61
Q

performing a squat with excessively externally rotated feet (feet turned outward) forces what to happen?

A
  • forces the tibia (shin bone) and femur (thigh bone) to also rotate externally
  • this posture alters the length-tension relationships of the muscles at the knees and hips, putting the gluteus maximus in a shortened position and decreasing its ability to generate force
  • the biceps femoris (hamstring muscle) and piriformis (outer hip muscle) becomes synergistically dominant, altering the force-couple relationships and ideal joint motion, increasing the stress on the knees and low back
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62
Q

with time, what can happen due to arthrokinetic dysfunction?

A

the stress associated with it can lead to pain, which can further alter muscle recruitment and joint mechanics

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63
Q

neuromuscular efficiency

A

the ability of the neuromuscular system to properly recruit muscles to produce force (concentrically), reduce force (eccentrically), and dynamically stabilize (isometrically) the entire kinetic chain in all
three planes of motion

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64
Q

what helps to determine muscle balance or imbalance?

A

mechanoreceptors (or sensory receptors)

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65
Q

what do mechanoreceptors include?

A

the muscle spindles and Golgi tendon organs

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66
Q

muscle spindles

A

the major sensory organ of the muscle

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67
Q

what are muscle spindles composed of?

A

microscopic fibers that lie parallel to the muscle fiber

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68
Q

what are muscle spindles sensitive to?

A
  • change in muscle length

- rate of length change

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69
Q

what is the function of the muscle spindle?

A

to help prevent muscles from stretching too far or too fast

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70
Q

what happens when a muscle on one side of a joint is lengthened (because of a shortened muscle on the opposite side)?

A
  • the spindles of the lengthened muscle are stretched
  • this information is transmitting to the brain and spinal cord, exciting the muscle spindle and causing muscle fibers of the lengthened muscle to contract
  • this often results in micro muscle spasms or a feeling of tightness
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71
Q

example - hamstring complex when the pelvis is rotated anteriorly

A
  • the anterior superior iliac spines move downward and the ischium moves upward
  • if the attachment of the hamstring complex is moved superiorly, it increases the distance between the two attachment sites and lengthens the hamstring complex
  • in this case, the hamstring complex does not need to be statically stretched because it is already in a stretched position
  • when a lengthened muscle is stretched, it increases the excitement of the muscle spindles and further creates a contraction (spasm) response
  • with this scenario, the shortened hip flexors are helping to create the anterior pelvic rotation that is causing the lengthening of the hamstring complex
  • instead, the hip flexors need to be stretched
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72
Q

example - knees adduct and internally rotate during a squat exercise

A
  • the underactive muscle is the gluteus medius (hip abductor and external rotator), and the overactive muscles include the adductors (inner thighs) and tensor fascia latae (a hip flexor and hip internal rotator)
  • thus, one would not need to stretch the gluteus medius, but instead stretch the adductor complex and tensor fascia latae, which in this case are overactive, pulling the femur into excessive adduction and internal rotation
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73
Q

where are Golgi tendon organs located?

A

within the musculotendinous junction (or the point where the muscle and the tendon meet)

74
Q

what are Golgi tendon organs sensitive to?

A
  • changes in muscular tension

- the rate of tension change

75
Q

what does the Golgi tendon organ do when a muscle is excited?

A

it causes the muscle to relax, which prevents the muscle from being placed under excessive stress, which could result in injury

76
Q

what does prolonged Golgi tendon organ stimulation provide?

A

-an inhibitory action (called autogenic inhibition) to muscle spindles (located within the same muscle)

77
Q

what does holding a stretch do?

A
  • creates tension in the muscle
  • this tension stimulates the Golgi tendon organ, which overrides muscle spindle activity in the muscle being stretched, causing relaxation in the overactive muscle and allowing for optimal lengthening of the tissue
78
Q

how long should a stretch be held?

A

long enough for the Golgi tendon organ to override the signal from the muscle spindle (approximately 30 seconds)

79
Q

reasons for flexibility training

A
  • correcting muscle imbalances
  • increasing joint range of motion
  • decreasing the excessive tension of muscles
  • relieving joint stress
  • improving the extensibility of the musculotendinous junction
  • maintaining the normal functional length of all muscles
  • improving neuromuscular efficiency
  • improving function
80
Q

what are muscular imbalances often caused by?

A

muscle imbalances

81
Q

examples of pattern overload

A
  • baseball pitching
  • long-distance running
  • cycling
  • training the same gym routine repetitively
  • a repetitive occupation lifting and loading packages all day
  • sitting for long periods of time while working on a computer
82
Q

what do poor posture and repetitive movements result in?

A
  • dysfunction within the connective tissues of the body
  • this dysfunction is treated by the body as an injury, and as a result, the body will initiate a repair process termed the cumulative injury cycle
83
Q

cumulative injury cycle

A

tissue trauma > inflammation > muscle spasm > adhesions > altered neuromuscular control > muscle imbalance

84
Q

what does trauma to the tissue of the body create?

A

inflammation

85
Q

how does the body respond to inflammation?

A

it activates the body’s pain receptors and initiates a protective mechanism, increasing muscle tension or causing muscle spasm

86
Q

what happens as a result of heightened activity of muscle spindles?

A

a microspasm

87
Q

what happens as a result of a microspasm?

A

adhesions (or knots) begin to form in the soft tissue

88
Q

what do adhesions in the soft tissue form?

A

a weak, inelastic matrix (inability to stretch) that decreases normal elasticity

89
Q

what do adhesions in the soft tissue result in?

A
  • altered length-tension relationships
  • altered force-couple relationships
  • altered arthokinetic dysfunction
90
Q

what do altered length-tension relationships lead to?

A

altered reciprocal inhibition

91
Q

what do altered force-couple relationships lead to?

A

altered force-couple relationships

92
Q

what does arthrokinetic dysfunction lead to?

A

altered joint motion

93
Q

what happens if these adhesions are left untreated?

A

they can begin to form permanent structural changes in the soft tissue that is evident by Davis’s law

94
Q

what is is soft tissue remodeled with?

A

an inelastic collagen matrix that forms in a random fashion, meaning it usually does not run

95
Q

what happens to these inelastic connective tissue fibers when the muscle fibers are lengthened?

A

they act as road blocks, preventing the muscle fibers from moving properly, which creates alterations in normal tissue extensibility and causes relative flexibility

96
Q

what happens if a muscle is in a constant shortened state?

A

it will demonstrate poor neuromuscular efficiency, which will affect joint motion and alter movement patterns

97
Q

what happens when a muscle is consistently short and moves in a pattern different from its intended function?

A

the newly formed inelastic connective tissue forms along this altered pattern, reducing the ability of the muscle to extend and move in its proper manner

98
Q

3 phases of flexibility training within the OPT model (integrated flexibility continuum)

A
  1. corrective
  2. active
  3. functional
99
Q

what does corrective flexibility include?

A
  • self-myofascial release

- static stretching

100
Q

what does active flexibility include?

A
  • self-myofascial release

- active-isolated stretching

101
Q

what does functional flexibility include?

A
  • self-myofascial release

- dynamic stretching

102
Q

what is corrective flexibility designed to do?

A
  • increase joint ROM
  • improve muscle imbalances
  • correct altered joint motion
103
Q

what principle does self-myofascial release use?

A

autogenic inhibition to cause muscle relaxation

104
Q

what principle does static stretching use?

A

autogenic inhibition or reciprocal inhibition to increase muscle length

105
Q

what phase of the OPT model is corrective flexibility appropriate for?

A

the stabilization level (phase 1)

106
Q

what is active flexibility designed to do?

A
  • improve the extensibility of soft tissue

- increase neuromuscular efficiency by using reciprocal inhibition

107
Q

what does active-isolated stretching allow the agonists and synergist muscles to do?

A

move a limb through a full range of motion while the functional antagonists are being stretched

108
Q

what phase of the OPT model is active flexibility appropriate for?

A

the strength level (phases 2, 3, and 4)

109
Q

what does dynamic stretching require?

A

integrated, multiplanar soft tissue extensibility, with optimal neuromuscular control, through the full range of motion, or essentially movement without compensations

110
Q

what should the client do if they are compensating when performing dynamic stretches during training?

A

regress to active or corrective flexibility

111
Q

what phase of the OPT model is functional flexibility appropriate for?

A

the power level (phase 5) or before athletic competition

112
Q

functional movements occur in which plane of motion?

A

all 3

113
Q

which plane of motion do injuries most often occur in?

A

the transverse plane

114
Q

true or false: exercises that have high levels of neuromuscular demand are preferred

A

TRUE

115
Q

what is self-myofascial release used for?

A
  • corrective existing muscle imbalances
  • reducing trigger points (knots within a muscle_
  • inhibiting overactive musculature
116
Q

when should self-myofascial release be used?

A

before or after exercise

117
Q

what is static stretching used for?

A
  • correcting existing muscle imbalances

- lengthening overactive (tight) musculature

118
Q

when should static stretching be used?

A

before or after exercise

119
Q

what is active stretching used for?

A

-increasing the extensibility of soft tissues through reciprocal inhibition

120
Q

what is dynamic or functional stretching used for?

A

-increasing flexibility with optimal neuromuscular control

121
Q

when should dynamic stretching be used?

A

once clients have demonstrated adequate control over motions to prevent injury

122
Q

true or false: flexibility training is progressive

A

TRUE

123
Q

self-myofascial release

A

a stretching technique that focuses on the neural system and fascial system in the body (or the fibrous tissue that surrounds and separates muscle tissue)

124
Q

what happens when you apply gentle force to an adhesion or “knot”?

A

the elastic fiber muscles altered from a bundled position (which causes the adhesion) into a straighter alignment with the direction of the muscle or fascia

125
Q

what does the gentle pressure applied during SMR result in?

A

stimulation of the Golgi tendon organ, which creates autogenic inhibition, decreasing muscle spindle excitation and releasing the hypertonicity (tension) of the underlying musculature

126
Q

requirements for SMR

A
  1. find a tender spot (which indicates the presence of muscle hypertonicity)
  2. sustain pressure on that spot for a minimum of 30 seconds
127
Q

what triggers the autogenic inhibition response during SMR?

A
  • increase in the Golgi tendon organ activity

- decrease in the muscle spindle activity

128
Q

should you perform self-myofascial release before or after stretching? why?

A

before, because breaking up fascial adhesions (knots) may potentially improve the tissue’s ability to lengthen through stretching techniques

129
Q

how does SMR help restore the body back to its optimal level of function?

A

by resetting the proprioceptive mechanisms of the soft tissue

130
Q

static stretching combines ___ force with ___ duration

A

low, longer

131
Q

what happens when you hold the muscle in a stretched position for a prolonged period?

A
  • the Golgi tendon organ is stimulated and produces an inhibitory effect on the muscle spindle (autogenic inhibition)
  • this allows the muscle to relax and provides for better elongation of the muscle
132
Q

contracting the antagonistic musculature while holding a static stretch can what?

A

reciprocally inhibit the muscle being stretched, allowing it to relax and enhancing the stretch

133
Q

static stretching should be used to ___ the muscle spindle activity of a tight muscle before and after activity

A

decrease

134
Q

static stretch - mechanisms of action

A

autogenic inhibition or reciprocal inhibition (depending on how the stretch is performed)

135
Q

static stretch - acute variables

A
  • 1-3 sets

- hold each stretch 30 seconds

136
Q

static stretch - examples

A
  • gastrocnemius stretch
  • kneeling hip flexor stretch
  • standing adductor stretch
  • pectoral wall stretch
137
Q

what does active-isolated stretching result in?

A

increased motoneuron excitability, creating reciprocal inhibition of the muscle being stretched

138
Q

when should you perform active-isolated stretches?

A

as a preactivity warm-up, as long as no postural distortion patterns are present

139
Q

when should active-isolated stretching be performed if an individual possesses muscle imbalances?

A

after SMR and static stretching for muscles determined as tight or overactive during the assessment process

140
Q

active-isolated stretch - mechanism of action

A

reciprocal inhibition

141
Q

active-isolated stretch - acute variables

A

1-2 sets, hold each stretch 1-2 seconds for 5-10 repetitions

142
Q

active-isolated stretch - examples

A
  • active supine biceps femoris stretch
  • active kneeling quadriceps stretch
  • active standing adductor stretch
  • active pectoral wall stretch
143
Q

dynamic stretch - mechanism of action

A

reciprocal inhibition

144
Q

dynamic stretch - acute variables

A

1-2 sets, 10-15 repetitions, 3-10 exercises

145
Q

dynamic stretch - examples

A
  • prisoner squats
  • multiplanar lunges
  • single-leg squat touchdowns
  • tube walking
  • medicine ball lift and chop
146
Q

examples of controversial stretches

A
  1. inverted hurdler’s stretch
  2. plow
  3. shoulder stand
  4. straight leg toe touch
  5. arching quadriceps
147
Q

inverted hurdler’s stretch

A
  • believed to place high stress on the inside of the knee
  • may cause pain and stress on the kneecap
  • should not be performed by anyone with a history of knee or low-back pain
148
Q

plow

A
  • common posture from yoga (inverted)
  • placed high stress on the neck and spine
  • may place the spine at risk of injury if not performed correctly
  • should not be performed by anyone with a history of neck or back injury, or individuals with high blood pressure
149
Q

shoulder stand

A
  • common posture from yoga (inverted)
  • places high stress on the neck, shoulders, and spine
  • should be avoided in patients with hypertension or any history of neck or spine injury
150
Q

straight-leg toe touch

A
  • one of the most common stretches for the hamstring complex
  • may place the vertebrae and the cartilage discs in the low back under high stress
  • should not be performed by anyone with a history of herniated discs or nerve pain that runs in the back
  • clients with poor flexibility may attempt to hyperextend the knees during this stretch, which may place high stress on the ligaments of the knee
151
Q

arching quadriceps

A
  • designed to stretch the quadriceps and hip flexors
  • places high stretch on the kneecap and the other tissues on the front of the knee joint
  • any client with a history of knee injury should avoid this stretch
152
Q

why would anyone perform controversial stretches if they are dangerous?

A
  • some of these positions are required in certain sports or activities
  • others are traditional positions used in martial arts, gymnastics, or yoga
153
Q

anterior view checkpoints

A
  1. feet

2. knees

154
Q

lateral view checkpoints

A
  1. LPHC

2. upper body

155
Q

feet turn out - overactive muscles

A
  • soleus
  • lateral gastrocnemius
  • biceps femoris (short head)
156
Q

feet turn out - underactive muscles

A
  • medial gastrocnemius
  • medial hamstring complex
  • gracilis
  • sartorius
  • popliteus
157
Q

feet turn out - sample SMR and static stretch techniques

A
  • SMR: gastrocnemius/soleus
  • SMR: biceps femoris (short head)
  • static gastrocnemius stretch
  • static supine biceps femoris stretch
158
Q

feet turn out - sample strengthening exercises

A

single-leg balance reach

159
Q

knees move inward - overactive muscles

A
  • adductor complex
  • biceps femoris (short head)
  • tensor fascia latae
  • vastus lateralis
160
Q

knees move inward - underactive muscles

A
  • gluteus medius/maximus

- vastus medialis oblique (VMO)

161
Q

knees move inward - sample SMR and static stretch techniques

A
  • SMR: adductors
  • SMR: TFL/IT band
  • static supine biceps femoris stretch
  • static standing TFL stretch
162
Q

knees move inward - sample strengthening exercises

A

tube walking: side to side

163
Q

excessive forward lean - overactive muscles

A
  • soleus
  • gastrocnemius
  • hip flexor complex (TFL, rectus femoris, psoas)
  • abdominal complex (rectus abdominis, external obliques)
164
Q

excessive forward lean - underactive muscles

A
  • anterior tibialis
  • gluteus maximus
  • erector spinae
165
Q

excessive forward lean - sample SMR and static stretch techniques

A
  • SMR: gastrocnemius / soleus
  • SMR: quadriceps
  • static gastrocnemius stretch
  • static kneeling hip flexor stretch
166
Q

excessive forward lean - sample strength exercises

A
  • quadruped arm / opposite leg raise

- ball wall squats

167
Q

low back arches - overactive muscles

A
  • hip flexor complex (TFL, rectus femoris, psoas)
  • erector spinae
  • latissimus dorsi
168
Q

low back arches - underactive muscles

A
  • gluteus maximus
  • hamstring complex
  • intrinsic core stabilizers (transverse abdominis, multifidus, transversospinalis, internal oblique, pelvic-floor muscles)
169
Q

low back arches - sample SMR and static stretch techniques

A
  • SMR: quadriceps
  • SMR: latissimus dorsi
  • static kneeling hip flexor stretch
  • static latissimus dorsi ball stretch
170
Q

low back arches - sample strengthening exercises

A
  • quadruped arm / opposite leg raise

- ball wall squats

171
Q

arms fall forward - overactive muscles

A
  • latissimus dorsi
  • teres major
  • pectoralis major/minor
172
Q

arms fall forward - underactive muscles

A
  • mid/lower trapezius
  • rhomboids
  • rotator cuff (supraspinatus, infraspinatus, teres minor, subscapularis)
173
Q

arms fall forward - sample SMR and static stretch techniques

A
  • SMR: thoracic spine
  • SMR: latissimus dorsi
  • static latissimus dorsi ball stretch
  • static pectoral wall stretch
174
Q

arms fall forward - sample strengthening exercises

A

squat to row

175
Q

shoulders elevate (push/pulling assessment) - overactive muscles

A
  • upper trapezius
  • sternocleidomastoid
  • levator scapulae
176
Q

shoulders elevate (push/pulling assessment) - underactive muscles

A

-mid/lower trapezius

177
Q

shoulders elevate (push/pulling assessment) - SMR and static stretch techniques

A
  • SMR: upper trapezius (Thera Cane)

- static stretch upper trapezius / scalene stretch

178
Q

shoulders elevate (push/pulling assessment) - sample strengthening exercises

A

-ball cobra

179
Q

head protrudes forward (pushing/pulling assessment) - overactive muscle

A
  • upper trapezius
  • sternocleidomastoid
  • levator scapulae
180
Q

head protrudes forward (pushing/pulling assessment) - underactive muscles

A

-deep cervical flexors

181
Q

head protrudes forward (pushing/pulling assessment) - SMR and static stretch techniques

A
  • SMR: upper trapezius (Thera Cane)

- static stretch upper trapezius / scalene stretch

182
Q

head protrudes forward (pushing/pulling assessment) - sample strengthening exercises

A

-chin tuck (keep head in neutral position during all exercises)

NASM-CPT (21 decks)

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