Chapter 52-High Risk Pregnancy Flashcards
severe generalized massive edema often seen with fetal hydrops
anascara
lack of development of the caudal spine and cord (may occur in the fetus of a diabetic mother)
caudal regression syndrome
occurs when the division of the egg occurs after 13 days
conjoined twins
twins that arise from two separately fertilized ova
dizygotic
coma and seizures in the second- and third-trimester patient secondary to pregnancy-induced hypertension
eclampsia
fetal death that occurs after the fetus has reached a certain growth that is too large to resorb into the uterus
fetus papyraceous
fluid occurs in at least two areas: pleural effusion, pericardial effusion, ascites, or skin edema
hydrops fetalis
excessive vomiting that leads to dehydration and electrolyte imbalance
hyperemesis gravidarum
an antigen present in the fetus; the maternal serum is tested between 15 and 22 weeks of gestation to detect abnormal levels; can also be tested directly from the amniotic fluid during amniocentesis
maternal serum alpha-fetoprotein (MSAFP)
a blood test conducted during the second trimester (15 to 22 weeks) to identify pregnancies at a higher risk for chromosomal anomalies (trisomy 21 and trisomy 18) and neural tube defects
maternal serum quad screen
twins that arise from a single fertilized egg that divides to produce two identical fetuses
monozygotic
a group of conditions in which hydrops is present in the fetus but not a result of fetomaternal blood group incompatibility
nonimmune hydrops (NIH)
too little amniotic fluid
oligohydramnios
too much amniotic fluid
polyhydramnios
complication of pregnancy characterized by increasing hypertension, proteinuria, and edema
preeclampsia
elevation of maternal blood pressure that may put the fetus at risk
pregnancy-induced hypertension (PIH)
leaking or breaking of the amniotic membranes causing the loss of amniotic fluid, which may lead to premature delivery or infection
premature rupture of the membranes (PROM)
system of antigens that may be found on the surface of red blood cells; when the Rh factor is present, the blood type is Rh positive; when the Rh antigen is absent, the blood type is Rh negative; a pregnant woman who is Rh negative may become sensitized by the blood of an Rh-positive fetus; in subsequent pregnancies, if the fetus is Rh positive, the Rh antibodies produced in maternal blood may cross the placenta and destroy fetal cells, causing erythroblastosis fetalis
Rh blood group
overlapping of the skull bones; occurs in fetal death
Spaulding’s sign
inflammatory disease involving multiple organ systems; a fetus of a mother with SLE may develop heart block and pericardial effusion
systemic lupus erythematosus (SLE)
monozygotic twin pregnancy with single placenta and arteriovenous shunt within the placenta; the donor twin becomes anemic and growth restricted with oligohydramnios; the recipient twin may develop hydrops and polyhydramnios
twin-to-twin transfusion syndrome (TTS)