Chapter 2 Flashcards

1
Q
  1. When do the crowns of deciduous teeth develop?
  2. When do the crowns of permanent teeth develop?
  3. Changes during this time that affect the Ameloblasts can cause?
A
  1. 14th week gestation through 12 months
  2. 6 months to 15 years
  3. Tooth defects
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2
Q
  1. What is an Enamel defects seen in permanent teeth caused by periapical inflammatory disease of the overlying deciduous tooth?
  2. In which teeth is it most commonly seen?
A
  1. Turner’s Hypoplasia (Turner’s Tooth)

2. Most commonly seen in the Permanent Bicuspids because of their relationship to the overlying deciduous molars.

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3
Q
  1. What is a major concern of aesthetics of anterior teeth), from excess amounts of fluoride that can result in significant enamel defects?
  2. When is the critical period for this?
A
  1. Dental Fluorosis
  2. The critical period for clinically significant dental fluorosis is during the second and third years of life, when these teeth are developing.
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4
Q
  1. What is rare, the teeth look like a straight-edge screwdriver?
  2. What are the anterior teeth called?
  3. What are the posterior teeth called?
A
  1. Congenital Syphilis
  2. Anterior: Hutchinson’s Incisors
  3. Posterior: Mulberry Molars
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5
Q

What is an individual with Hutchinson’s teeth, 8th nerve deafness (vestibulocochlear), saddle nose and interstitial keratitis (corneal scarring)? ALSO ASSOCIATED WITH SYPHILIS????

A

Hutchinson’s Triad

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6
Q
  1. What is loss of tooth structure caused by tooth-tooth contact?
  2. What is pathologic wearing away of tooth structure (most common cause is toothbrushing)?
  3. What exhibits features of both attrition and abrasion. (chewing tobacco between opposing teeth)?
  4. What is a loss of tooth structure caused by a nonbacterial chemical process?
  5. What is erosion from dental exposure to gastric secretions?
  6. What is loss of tooth structure from occlusal stresses that create repeated tooth flexure?
A
  1. Attrition (Bruxism)
  2. Abrasion
  3. Demastication
  4. Erosion/Corrosion
  5. Perimolysis
  6. Abfraction
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7
Q

What is lack of tooth development, that is rare, and most cases occur in the presence of hereditary hypohidrotic ectodermal dysplasia?

A

Anodontia

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8
Q
  1. What is lack of one or more teeth?
  2. What are the three most commonly missing? **Absence of a deciduous tooth= likely to miss the associated permanent tooth
  3. What is lack of 6+ teeth?
A
  1. Hypodontia
  2. 3rd molars most commonly affected, then 2nd premolars and lateral incisors.
  3. Oligodontia
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9
Q
  1. What is supernumerary tooth/teeth?

2. What is the most common area?

A
  1. Hyperdontia

2. The most common site is the maxillary incisor region (mesiodens).

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10
Q

What is a single enlarged tooth or joined tooth in which the tooth count is normal when the anomalous tooth is counted as one? (Still have 32 teeth)

A

Gemination

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11
Q

What is a single enlarged tooth or joined tooth in which the tooth count reveals a missing tooth when the anomalous tooth is counted as one? (31 teeth or less)

A

Fusion

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12
Q

What is the union of two adjacent teeth by cementum alone without confluence of underlying dentin?

A

Concrescence

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13
Q
  1. What is a bend in the tooth root?

2. Where does this most commonly occur?

A
  1. Dilaceration

2. Mandibular 3rd molars > maxillary 2nd premolars > mandibular 2nd molars

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14
Q
  1. What is an enlargement of the body and pulp chamber of a multi-rooted tooth, with apical displacement of the pulpal floor?
  2. Can be isolated or syndromic, what are the three examples of this?
A
  1. Taurodontism

2. Kleinfelters Syndrome (XXY), Amelogenesis imperfecta, Tricho-dento-osseous syndrome

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15
Q

What is a syndrome which in addition to the dental findings, the predominant systemic changes are present variably and include kinky hair, osteosclerosis (base of skull and mastoid process), and brittle nails?

A

Tricho-dento-osseous syndrome

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16
Q
  1. What is a non-neoplastic deposition of excessive cementum that is continuous with the normal radicular cementum?
  2. This can occur by local and systemic factors, what are examples of each?
A
  1. Hypercementosis
  2. • Local factors
    o Occlusal trauma
    o Adjacent inflammation (pulpal, periapical, periodontal)
    • Systemic factors:
    o Paget’s Disease of bone is strongly associated with generalized hypercementosis.
17
Q

What is a lack of clavicle and supernumerary teeth?

A

Clidocranial Dysplasia

18
Q

What are teeth that erupt in the wrong order?

A

Transposition

19
Q

What is failure to fully erupt/reach the level of occlusion?

A

Ankylosis

20
Q
  1. What are developmental alterations in the structure of enamel in the absence of a systemic disorder?
  2. Examples are Hypoplastic, Hypocalcified, and Hypomaturation, what are these (Don’t need to know these for exams/quizzes, but good to know)? **Usually both deciduous and permanent dentition are diffusely involved.
A
  1. Amelogenesis Imperfecta
  2. o Hypoplastic: Elaboration of the enamel matrix ‘pitting’ (Inadequate deposition of enamel matrix. Thin, hard, rough enamel; yellow teeth, or no enamel)
    o Hypocalcified: Mineralization of the matrix (Matrix is laid down appropriately but no mineralization occurs. Enamel is soft and easily lost, starts as yellow-brown or orange and becomes brown to black with rapid calculus apposition)
    o Hypomaturation: Maturation of the enamel ‘snow capped’ (Enamel matrix is laid down appropriately and begins to mineralize. Opaque white-brown-yellow discoloration with soft Enamel and chips away from dentin.)
21
Q

What typically occurs on Premolar teeth, usually bilateral and more common in the mandibular arch, that is seen in association with shovel-shaped incisors and up to 100% prevalent in Native Americans and common in Asians?

A

Dens Evaginatus

22
Q

What is a “tooth within a tooth” with a deep surface invagination of the crown or root that is lined by enamel?

A

Dens-in-dente -OR- Dens Invaginatus

23
Q
  1. What is hereditary developmental disturbance of dentin in the absence of any systemic disorder? What is a way to see this?
  2. What do x-rays look like?
  3. This is a mutation in what gene?
  4. What if it occurs with osteogenesis imperfecta, what is it called? What is this a mutation of? What is a way to see this?
A
  1. Dentinogenesis Imperfecta, has Blue hue teeth color.
  2. Bulbous crowns with no root/canal.
  3. Dentin sialophosphoprotein gene (DSPP)
  4. Osteogenesis imperfecta with opalescent teeth (mutation of the COL1A1 or COL1A2), has a Blue Sclera color and Blue teeth.
24
Q
  1. What has two types (I and II) and has no correlation with systemic disease dentinogenesis imperfecta, but does affect dentin?
  2. What differentiates type I and type II?
A
  1. Dentin Dysplasia
  2. Type I: “Rootless teeth”, Autosomal dominant, Enamel and coronal dentin are NORMAL, Radicular dentin loses organization and is shortened dramatically, “stream flowing around boulders”
    Type II: Autosomal dominant, Root length is normal, Blue-to-amber-to-brown translucence, Via XRAY: Bulbous crowns/Cervical constriction/Thin roots/Early obliteration of the pulp/Altered pulpal anatomy/Thistle tube-shaped
    and Pulp stones
25
Q

What is a localized, non-hereditary developmental abnormality of teeth with extensive adverse effects on the formation of enamel, dentin, and pulp?

A

Regional Odontodysplasia “Ghost Teeth”

26
Q
  1. What is it called when you are born with teeth?

2. What is the syndrome where trauma occurs at the gingiva around Natal Teeth?

A
  1. Natal Teeth

2. Riga’ Fede Disease