Chapter 11 Flashcards

1
Q

Language?

A

-using syntax to communicate and convey meaning
Animals have complex communication system, but not language.
You need to use words and grammar syntax to have ability to create new meaning

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2
Q

Mental lexicon?
What does the mental lexicon store?

A

dictionary of words, their meanings, and grammar rules, in both hemisphere storing:
-semantic info (meaning of words)
-syntactic info (how words are combined to create new meaning)
-word forms: orthographic (visual: written word, what it looks like on paper) and phonological (how a word sounds like when spoken ex: aaron vs. erin) structure of words. We need mental lexicon for both speech and comprehension

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3
Q

semantic network mental model

A

The words in the mental lexicon are represented as conceptual nodes, which connect with each other. For example, the node that represents the word car will be close to and have a strong connection with the node that represents the word truck. Words that have strong associative or semantic relations are closer together in the network (e.g., car and truck) than are words that have no such relation (e.g., car and clouds).

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4
Q

How conceptual knowledge is represented in semantic network?

A

Some propose that words that co-occur in our language prime each other (e.g., cottage and cheese), and others suggest that concepts are represented by their semantic features or semantic properties. For example, the word dog has several semantic features, such as “is animate,” “has four legs,” and “barks,” and these features are represented in the conceptual network.

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5
Q

Semantic priming effect

A

People respond faster to an item presented if they saw an item before with similar meaning
(ex: seeing the word rose as a primer, then saying the target word: flower)

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6
Q

Semantic paraphasia

A

An error in speech in which one word is substituted for another word that is similar in meaning
- A person means to use one word, but they use another wrong word (say fork instead of spoon, when you meant spoon), the word is usually conceptually related in category

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7
Q

category-specific anomia?

A

person can describe the object within a category in detail, but has trouble to retrieve (recall) the name.
“I know this thing, but I can’t recall a name”

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8
Q

Physical vs. Functional Properties theory for category-specific anomia (Warrington)

A

theory states that it’s necessary to have the visual association with the word. For example, you associate the animal kangaroo with pouch (marsupial), the loss of which can be difficult to retrieve the word kangaroo (physical properties). But for the objects, such as comb, we have both: visual and sensory motor associations (functional properties). If you damage one area of the brain, the associations may still exist

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9
Q

Fundamental Categorical Organization theory for category-specific anomia (Caramazza)

A

His theory states that mental lexicon is thoroughly organized into categories, which correspond to specific areas of the brain. For example, people will have a trouble naming animals, when they get inferior temporal lobe damage, located anteriorly in the brain. If they will get a lesion further back: in posterior part of the brain, they will have difficulties naming tools. Whereas the damage to temporal pole will disrupt the ability to name people.

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10
Q

Complexity of Features theory for category-specific anomia (Tyler)

A

The theory is more focused on shared features of the word. It makes a distinction between the words at specific level (e.g., zebra or pencil), which requires additional features and the domain-general level (e.g., living or nonliving), which only needs a subset of features to name the picture. In addition, the living things are more difficult to recognize, because they share more features in comparison with nonliving things. As a result, if you have a damage to mental lexicon, you will still be able to activate the nonliving word (e.g., wood and lead for pencil). But you will not name a living word: zebra with damaged semantic network of mental lexicon, since you will have trouble to activate all association words, such as black and white stripes.

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11
Q

Top-down vs bottom-up processing (Language comprehension)?

A

Language comprehension model starts with input and ends up with conceptual representation (spoken or visual language)
-A mix of top-down (complex to simple) and bottom-up (simple to complex) processing.
Bottom-up - based on raw data: the idea that we perceive the word and then look for the word in our mental lexicon
Top-down - allow info in mental lexicon to influence the analysis of the incoming info: our knowledge of words and grammar can influence our visual and auditory perception

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12
Q

Segmentation Problem?

A

using knowledge in our mental lexicon to influence what we hear (our perception)
An issue of speech perception by sonograph
- how do you know that “do you mean” is 3 words and not one big word
Cap-tain – looks like 2 words;
testing – like 2 words on sonograph

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13
Q

Spreading activation

A

you mentally activate a word, when you think about it, which spreads to the connections, activating associative words. For example, you think about red and flower - you activate rose.

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14
Q

Language Comprehension, is it top-down or bottom-up processing?

A

Top-down influence, it affects auditory processing as well - you don’t know words of a doctor, but pharmacist can recognize them.
Top-down processing – perceives letters different, bottom-up perceive letters the same.

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15
Q

Brain areas and speech comprehension (Which part of brain?)

A

-some areas are very sensitive to phonological/acoustic properties (ex: high vs. low voices), but language sensitivity is very low (during early areas, red spot auditory cortex)
-high acoustic sensitivity in the primary auditory cortex (red spot); (sounds are treated the same as any sound)
-high language sensitivity in superior temporal sulcus ,
Speech comprehension – inferior left frontal lobe areas. Blue areas are the opposite of red

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16
Q

Brain areas and speech perception.
Where does the brain start treating language information differently?

A

The superior temporal sulcus - evidence that brain is responding differently to language sounds vs non-language sounds (sounds become treated like language); The Broca’s area (frontal lobe) is more sensitive to language rather than acoustics. Broca’s is involved in some aspects of comprehension, but it’s more about production of language.

17
Q

N400 ERP response?

A

negative deflection if semantic content is wrong (semantic violations)
(ex: He spread the warm bread with socks - semantically wrong)
-someone with wernicke’s aphasia may find it difficult to comprehend
N400 (400 ms) - language comprehension, reflecting about semantic processing; it can be a surprise, unexpected response.

18
Q

P600 ERP response?

A

a positive (P600) ERP shift following a syntactic violation in a sentence (grammatically incorrect)
Issue with grammar (syntax);
P600 it’s more about syntactic violations of language; you wouldn’t get P600 if you don’t know the grammar of language

19
Q

ERP’s and language deficits

A

N400 response still occurs in people w/ language deficits (comprehension) but it is reduced and delay
*suggests that language disorders are an issue of timing (delayed)

20
Q

Speech production? Self-monitoring?

A

Starts with an abstract concept/idea, then you need to use language to convey that idea
-conceptual idea–>mental lexicon —-> use mental lexicons to create sound waves into a sentence in your brain —-> conveying it through speech.
Foreign accent syndrome (speech problems);
Self-monitoring – what we say actually matches the idea that we want to express, we correct ourselves when we are saying incorrectly

21
Q

Mental lexicon model of speech production (steps?)

A

-Lemma: choosing the “word” you want to say (ex: identifying a verb you want to say, like “to run”)
-morphological encoding: finding the correct version of the lemma (ex: making “to run” into “running”)
-phonological word: what word should sound like, how to move your mouth, when to breath as you speak etc.

22
Q

How is the lexical, grammatical, and phonological model of speech production supported? Which phases?

A

ECoG recording – Broca’s area frontal lobe
ECoG recordings found that
1st phase: using less common words = larger response than common words
2nd phase: reading words on a screen = smaller response, but choosing correct morpheme version of word changes response = suggests grammatical change
3rd phase: words with more syllables= larger response

23
Q

How is the lexical, grammatical, and phonological model of speech production supported?

A

ECoG recording – Broca’s area frontal lobe

24
Q

Aphasias? Expressive aphasia?

A

Deficits in language ability (speech production or comprehension, or both);
expressive aphasia (Broca’s) – problem with expression: speech production

25
Q

Broca’s (Expressive) aphasia? Symptoms? Leborgne case? Which part of brain?

A

-non-fluent telegraphic speech (leaving out connecting words; ex: hands…tingling…stroke…reading)
-trouble making sounds/controlling muscles to make sound
-normal comprehension, but agrammatism (difficult comprehending more complicated sentences)
ex. Leborgne lost ability to speak, but he could make sounds; paralysis on right side; he was intellectually aware. Huge lesion in the left-inferior temporal lobe, localized in left-hemisphere.
We speak with the left hemisphere from French.

26
Q

Dysarthria? Aggramatism? Apraxia?

A

Dysarthria - motor problem, difficulty controlling the muscles involved in articulation (the formation of sounds speech);
Difficulty understanding complex grammar (aggramatism), they can think the girl hit the boy, while it’s vice versa.
Apraxia - a motor disorder caused by damage to the brain which causes difficulty with motor activity to perform tasks or movements

27
Q

Which part of brain lesion? (Broca’s aphasia)

A

Left inferior temporal lobe – Broca’s area in left hemisphere (cortical area, outside);
Broca’s aphasia produce not only Broca’s area
AND deeper damage to underlying structures (insular cortex damage)

28
Q

Wernicke’s (receptive) aphasia? What are symptoms?

A

person has fluent speech BUT makes many errors in speech and uses incorrect words (semantic paraphasia)
EX: I called my mother on the television”
-person is unaware of errors
***issue with self-monitoring and turning thoughts to words
No trouble with producing speech, but more comprehension;

29
Q

Which part of brain lesion for Wernicke’s (receptive) aphasia?

A

Wernicke’s area - language comprehension.
damage in superior temporal gyrus in left hemisphere – known as Wernicke’s area;

30
Q

Transcortical Sensory Aphasia

A
  • low comprehension, so that person can’t take what they are hearing and then comprehend it to a concept
    -can speak AND REPEAT easily
    **similar to Wernicke’s aphasia, but person can repeat easily
31
Q

Conduction aphasia?

A

-issues with self-monitoring
-have trouble repeating things back!!!
Fluent speech and comprehension intact, she has awareness of semantic mistakes, but it’s too late – video of woman who can’t say all numbers in order, while Wernicke’s don’t realize the mistakes.
** person only realizes they spoke incorrectly until they speak

32
Q

arcuate fasciculus

A

They are white matter tracts in the brain, which connect Wernicke’s area and Broca’s area, damage to this causes conduction aphasia

33
Q

Transcortical motor aphasia

A

-difficult to take concepts and convert into fluent speech
-similar to Broca’s aphasia but a person CAN repeat

34
Q

Language Network View

A

there are at least 4 networks involved in language in the left hemisphere
Dorsal pathway (up 2): involved in speech production and understanding complicated language
Ventral (bottom 2): involved in comprehension (semantic) and simple grammar

35
Q

Following a stroke, a patient has fluent speech, but it is full of semantic paraphasia and other mistakes. The patient doesn’t repeat what they hear and there is little evident of speech comprehension. What type of aphasia does this patient have?

A

Wernickes (receptive/fluent) aphasia

36
Q

Following a stroke, a patient seems to comprehend written and spoken questions, but they have difficulty expressing their ideas fluently. The only time they seem to be able to speak sentences w/o issue is when they are asked to repeat something back. What type of aphasia does this patient have?

A

Transcortical motor aphasia