Ch. 7: Somatosensation - Clinical Application Flashcards

1
Q

What are the Somatosensory Contributions to Function?

A
  • Contributes to smooth, coordinated movement.
  • Helps protect from injury.
  • Contributes to understanding (perception) of our environment
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2
Q

How do we test somatosensation?

A
  • Test conscious relay pathways
  • Quick Screenings - if not problems indicated
  • Complete Evaluations - if problems are indicated in screening or if refered to us for them.
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3
Q

What are the conscious relay pathways that we test?

What are their named neurons?

A
  • Discriminative touch
    • A-beta (exteroception), DC?ML
  • Conscious proprioception
    • Ia, Ib, II DC/ML
  • Fast pain - tell sharp stimuli from dull
    • A-delta, anterolateral column
  • Discriminative temperature - discriminate hot from cold
    • A-delta, anterolateral column

“Cortical” sensations” - intact operations of the first 4 - where cortex process sensory info to make meaning

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4
Q

What do we test in a Quick Screening?

A
  • Vibration (distal joints)
    • A-beta, DC/ML
  • Conscious proprioception (distal joints)
    • Ia, Ib, II DC/ML - position sense (ex: can they detect that big toe is flexed or extended with eyes closed)
  • Fast pain
    • A-delta, anterolateral column - sharp/dull
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5
Q

What do we evaluate in a Complete Evaluation?

A
  • Sensory Threshold
    • Myofilaments - lightest touch possible
  • Sensitivity
    • Two-point discrimination
  • “Higher” (“cortical”) sensations
    • “Stereognosis” - descirbe familiar objects w/o looking (ex: key)
  • Pattern of loss
    • Pain, temp, touch → pt education if stimulus not coming back.
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6
Q

What are the limits to formal sensory examination

A
  • Unconscious
  • Unresponsive

Cannot get good results

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7
Q

What is Anesthesia?

A

Lack of sensation

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8
Q

What is Analgesia?

A

Lack of pain sensation specifically

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9
Q

What is Hypesthesia?

A

Less than normal sensation

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10
Q

What is hypalgesia?

A

Less than normal amount of pain sensation

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11
Q

What is Paresthesia?

A

Abnormal sensation (painless, abnormal, tickling, tingling)

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12
Q

What is dysesthesia?

A

Absensce of direct stimuli

(unpleasant, abnormal, burning, shooting)

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13
Q

What is allodynia?

A

Type of Dysesthesia

  • Patient perceives pain from a stimulus that normally does not cause pain
  • Ex: how sensitive skin is after sun burn.
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14
Q

What is hyperalgesia?

A
  • Normally perceived as pain → produces response out of proportion.
  • Result of damage to nervous system.
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15
Q

What is Ataxia?

A

Lack of coordinated movement

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16
Q

What is the Order of LOSS of senstaion with compression?

A
  1. Conscious proprioception & discriminative touch
  2. Cold
  3. Fast pain
  4. Heat
  5. Slow pain
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17
Q

What is the Order of RETURN of senstaion after compression?

A

The reverse of loss:

  1. Slow pain
  2. Heat
  3. Fast pain
  4. Cold
  5. Conscious proprioception and discriminative touch
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18
Q

What is the definition of pain?

A

“Pain is an unpleasant sensory and emotional experience”

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19
Q

What locations of the brain may respond to pain?

A
  • Affective
  • Emotional
  • Behavioral

Limbic terminaltes in the autonomic areas

20
Q

What is Peripheral Sensitization?

A

Sensitize peripheral neuron = low threshold for activation, low amt of stimulus that produces an AP

21
Q

What are the possible functions of FAST pain?

What named neurons are used?

A

Withdraw/escape from pain.

(acute, A-delta, superficial)

22
Q

What are the possible functions of SLOW pain?

What named neurons are used?

A

Rest damaged tissue

(chronic, C, deep)

23
Q

FAST pain uses what pathway?

A

Spinothalamic pathway

24
Q

SLOW pain uses what pathway?

A

Divergent Pathways

25
Q

What is Referred Pain?

A

Convergence of somatic [body] and visceral [organs] axons in spinal cord

(ex: left arm & heart converge at same spot - heart never hurts, brain learns it is always arm pain. Heart attack = brain thinks arm pain)

26
Q

What is the Pain Matrix?

A

Brain structures that regulate pain. Can create pain in absence of stimulus.

27
Q

The LATERAL pain system uses what pathway?

A

Spinothalamic

(Conscious Relay Pathway)

28
Q

The MEDIAL pain system uses what pathway?

A

Spinolimbic

(Diverengent Pathways)

29
Q

The pain experience envolves what 3 aspects?

What pathways do each of them use?

A
  1. Sensory → Discriminative aspects
    • Spinothalamic
  2. Motivational →Affective aspects
    • Spinolimbic and Spinoreticular
  3. Cognitive →Evaluative aspects
    • Prefrontal lobes
30
Q

What are the 2 “Top-Down” responses to pain?

What do they do?

A
  1. Antinociception
    • pain relief
  2. Pronociception
    • increase or amplify - ability to “experience” pain
31
Q

What is the first place pain is recepted?

A

Dorsal Horn

32
Q

What is the Dorsal Horn’s NORMAL response to pain?

A

Action release, AP, normal reporting of pain

33
Q

What is Suppressed Nociception in the Dorsal Horn?

A

Diminished pain

(ex: “rub it where it hurts”)

34
Q

What is “Sensitized” processing in the Dorsal Horn?

A

Temporary, short term increase in strength of pathway

Excessive pain!

35
Q

What is “Reorganized” processing in the Dorsal Horn?

A

Long Term Potentiation (LTP) → excessive pain!

Reconstruction in synapses to make it easier to send signal

36
Q

What susbstance helps you preceive MORE pain?

A

Substance P

37
Q

How do you stop pain in the Periphery?

A

Remove stimulus/irritants

(ex: massage to remove edema)

38
Q

How do you stop pain in the Dorsal Horn of the Spinal Cord?

A

Counterirritant Theory - presynaptic inhibition of pain neurons

(“rub it where it hurts”)

Ex: TENS unit, massage, etc.

39
Q

How do you stop pain in the Brainstem Descending Systems?

A
  • Your own thoughts can turn down pain.
    • Periaqueductal gray = termination of spinomesencephalic → starter for all brainstem to spinal cord pathways
40
Q

How do you stop pain at the subcortical level?

A
  • Potential to slow pain messages body wide
  • Hypothalamus, pituitary gland, adrenal medulla (opiate-mediated…endorphins!) → lasts longer than pain meds (up to several hours)
41
Q

How do you stop pain at the cortical level?

A

Pathway from cortex to hormonal level -> can turn on body wide reduction system by thought. Can turn on brain stem decending pathway.

(Ex: placebos, distraction, perception)

42
Q

How could you summarize WHY the pain matrix is important to us as PT’s?

A
  • We can influence pain at multiple levels to decrease pain
    • Modalities in the periphery
    • Exercise for body wide
    • Mental for body wide
43
Q

What are examples of Pronociception at Level I, II & IV

A
  • Pain amplification
    • Level I: peripheral sensitization
      • pathological = pain w/o stimulus
    • Level II: LTP of pain pathway and other malfunctions
      • increase effectiveness of pathway = more pain
    • Level V: psychological stress can amplify pain
      • dispear, hopelessness
44
Q

What is the difference between Acute and Chronic Pain?

A
45
Q

What is Chronic Pain?

A

healing extend beyond normal tissue healing time

46
Q

Define the 2 types of Chronic Pain?

A
  1. Nociceptive
    • Continuing pain stimulus
    • Neurons functioning normally
  2. Neuropathic
    • NO continuing pain stimulus
    • Neurons NOT functioning normally
    • ***Hradest pain to deal wiht → cannot find cause