Ch. 3 Flashcards

1
Q

Arousal Theory

A

Holds that we can best understand behavior by understanding how the organism becomes activated
Arousal can be viewed on a continuum of behavioral activation

Low arousal = coma, sleep
High arousal = stress

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2
Q

Sympatho-Adrenomedullary System

SAM Axis

A

Increased Adrenaline

  • Neurotransmitters
  • faster
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3
Q

Hypothalamus-Pituitary-Adrenal Glands (HPA Axis)

A

Involuntary.

  • slower - cortisol
  • Hormones

when threat detected..

Hypo, pituitary, adrenal, cortisol

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4
Q

Fight or Flight response

A
  1. You detect a stressor (either in your environment or maybe even in your own thoughts)
  2. Distress signal sent to your hypothalamus (some say threat is sent by amygdala, prefrontal cortex, or hippocampus)
  3. Hypothalamus sends out signal to rest of body that all is not well
  4. Activates the sympathetic nervous system (part of the autonomic/involuntary nervous system; SAM axis – sympathetic-adrenal-medullary system) - FAST
    Activates the endocrine system (HPA axis – hypothalamus, pituitary gland, adrenal glands) - SLOWER
  5. Epinephrine & norepinephrine (also known as adrenaline/noradrenaline) and cortisol are released into the blood stream and mobilize the body for action
    Epinephrine & norepinephrine more fast acting; cortisol more for the long haul
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5
Q

General Adaptive Syndrome and its phases (Hans Selye)

A

The body’s resistance to stress can only last so long before exhaustion sets in.

Stress is a nonspecific response of the body to any demand made upon it
“complete freedom from stress is death” (Selye, 1973)
Systemic stress involves a challenge to the integrity of the physical body

The body reacts to invasions (bacteria, virus, heat, cold) in an effort to combat it

The body’s response to psychological stress is virtually identical to its systemic reaction

Phase 1 - Alarm Reaction
Phase 2 - Resistance
Phase 3 - Exhaustion (reserves depleted)

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6
Q

Differences in Arousal/Stress

Life-change and stress research

A

Level of arousal from any one situation may depend on
Life experiences

More recent life changes predict chance of illness and decreased performance
But also, life experiences with a certain type of stressor could either increase or decrease resistance

Individual differences
Neuroticism
Hardiness (commitment, control, challenge)

How one labels (attributes) the cause of arousal
Two-factor theory of emotion (Schachter & Singer, 1962)

People’s mindset toward stress

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7
Q

Coping with stress—ways to cope, individual differences in coping

A
- Social Support
      May be more important for men
- Explanatory style
       Pessimistic vs. Optimistic
- Humor/Positive Emotions
- Knowledge about the stress system
- Keep big picture in mind
- Meditation
- Slow, deep breathing
- Exercise
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8
Q

Placebo Response

A

The strategies for mitigating stress suggest that there are things that you can consciously do to change your body’s response

Indeed, having certain expectations affects how your body responds

Placebo often considered an inert substance that people nevertheless report makes them feel better
Placebo can also be an expectation or belief, not just a pill

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9
Q

Two-factor theory of emotion (Schachter & Singer, 1962)

A

you may be feeling a certain way - stress and aroused, if you had a different way to describe it, you might not identify it as stress.

sitting down, coffee, increased heart rate, it’s the coffee not other external things. Re-explain the situation.

  • Conducted a study to test how people label their arousal
  • Randomly assigned participants to four conditions

Epinephrine informed – told about the side effects of epinephrine
Epinephrine ignorant – not told about how they would feel
Epinephrine misinformed – told wrong symptoms
Control group – injected with a placebo and given no expectation

  • Expected epinephrine ignorant and misinformed group to use cues from their environment to label their arousal
  • Assigned participants to interact with a euphoric or angry confederate (two separate studies)

Results -

Measured how much the participant “caught the confederate’s mood” (i.e., was happy in euphoric group and angry in the angry group).
Euphoric (ordered from highest to lowest group euphoria)
Epinephrine misinformed, epinephrine ignorant, placebo, epinephrine informed)
Anger (ordered from highest to lowest group anger)
Epinephrine ignorant, placebo, epinephrine informed (no misinformed)
Both results show that participants who had no explanation of why their body felt aroused were more susceptible to environmental cues (the confederate)

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10
Q

Rethinking stress article – Crum, Salovey, & Achor, 2013

A

.

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11
Q

Adenosine and its role in sleep

A

Neurotransmitter released by brain metabolism during waking

As we think (work our brains), adenosine builds up
The longer you have been awake, the more adenosine you have in your system

And the more sleep pressure that builds
Caffeine blocks the receptor site for adenosine to maintain arousal

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12
Q

Functionality of sleep

A
Sleep is restorative
More REM sleep occurs after days full of worry, stress, or intense learning
Organizes our brains
Consolidates memories
Allows for better visual discrimination
Counteracts cortisol
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13
Q

Sleep Deprivation

A

Inhibits the learning of new things and the retention of tasks learned prior to deprivation

Inhibits decision-making, attention, span, and the speed at which we adapt to new information

Increases anxiety

Related to more car crashes than people with blood alcohol levels of .05

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14
Q

Chronic Sleep Deprivation

A
  • Dysregulates hormones related to appetite
  • Increases risk of heart attacks
  • Compromises our immune system
  • Lowers testosterone
  • Premature aging
  • Irritability, anxiety, depression, mental exhaustion
  • In a clinical sense, the effects of stress and sleep deprivation are indistinguishable
    We might be confusing the two
    Sleep is the restorative balancer of cortisol
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