ch 19 pulmonary edema Flashcards

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1
Q

what is cardiogenic pulmonary edema classified as

A

restrictive lung disease

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2
Q

what is cardiogenic pulmonary edema also called

A

hydrostatic pulmonary edema

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3
Q

what is it essentially caused by

A

left side heart failure or fluid overload

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4
Q

what is pulmonary edema

A

excessive amount of fluid in the lung tissue or alveoli due to an increase in pulmonary capillary pressure resulting from abnormal left heart function

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5
Q

what is the etiology of cardiogenic pulmonary edema

A

left heart failure (ex. coronary artery disease)
aortic stenosis
mitral valve stenosis
systemic hypertension
fluid overload
decrease in osmotic pressure (pressure in blood to keep blood in inter vascular space - protein - albumin level)

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6
Q

what does the etiology result in

A

backup of fluid from the heart into the pulmonary capillaries which becomes engorged

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7
Q

what happens when pulmonary capillaries become engorged

A

fluid leaks into interstitial space and then into the alveoli

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8
Q

what is the swan ganz catheter

A

it measures the back pressure from the pulmonary veins - done from the carotid vein or subclavian vein and into right atrium, tricuspid, right ventricle

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9
Q

is it possible to measure the pressure from the left heart

A

no

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10
Q

is it possible to measure from the right heart

A

yes

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11
Q

what carries the swan ganz

A

the right ventricle into the pulmonary artery

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12
Q

where does the swan ganz measure pressure and it should be no more than what

A

5-8 and only in front of the catheter is where it is measured

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13
Q

how do they measure the progress of the treatment of the pulmonary edema

A

swan ganz catheter

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14
Q

how can you treat pulmonary edema

A

ionotropic - make left side work harder (digoxin)

lasics - get the excess fluid out of the body like a diuretic

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15
Q

what is atrial fibrillation

A
atopic phoxi (spelling lol) 
350-600 / min
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16
Q

what is atrial fibrillation caused by

A

congestive heart of the left ventrical, academia, alkalemia, electrical imbalances

17
Q

vital signs on admission

A
increased RR 22 
pH 7.56 
co2 28
hco3 20
pao2 51
spo2 70%
respiratory alkalemia - caused by hypoxemia
18
Q

patient inspection

A
distended neck veins
frequent cough - pink frothy sputum
distended abdomen
pitting edema 
blue lips
19
Q

xray

A

faint opacities in lower lobes bilaterally

heart was enlarged (left ventrical)

20
Q

treatment

A

oxygen
intravenous digitalis - dobutamine makes heart beat faster
furosemide - laxis takes liquid out of body to make you pee

21
Q

digitalis

A
  • inotrophic
  • increases myocardial force of contraction
  • increases stroke volume
  • antiarrhythmic used to treat atrail flutter and fibrillation
22
Q

dobutamine

A

increase myocardial contraction and stroke volume without increasing systemic vascular resistance (doesn’t increase blood pressure)
beta one

23
Q

how does furosemide work

A

causes diuresis by inhibiting reabsorption of sodium, also causes loss of chlorine, and potassium

24
Q

what abg can furosemide cause

A

metabolic alkalosis

25
Q

auscultation

A

inspiratory crackles expiratory wheezes over lower lobes

26
Q

example of shunt in this disease

A

alveoli filled with fluid

27
Q

venous admixture

A

good lung mixing with bad lung leaded to lower pao2

28
Q

pulmonary mechanics what happens to lung volumes

A

all lung volumes get smaller

29
Q

what needs to be resupplied for lung volumes to go back to normal

A

surfactant

30
Q

forced vital capacity

A

flow would unaffected but forced vital capacity would be smaller (middle line in picture)

31
Q

lung compliance?

A

decreased compliance

32
Q

WOB ?

A

increased work of breath

33
Q

what is the gold standard treatment for cardiogenic pulmonary edema

A

cpap mask