Cervical spine pathologies Flashcards

1
Q

what is cervical spondylosis

A

degenerative disc disorder affecting interbody joint, lower cervical spine most affected (C5-6, C6-7, C4-5), age groups 45+, onset= insidious or traumatic. Disc space narrowing, anterior osteophyte formation, lipping & irregularity of vertebral bodies

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2
Q

cervical spondylosis- patho-anatomical changes

A

loss of disc height due to disc dehydration and degermation, vertebrae approximate, formation of marginal osteophytes, increased WB on facet joints, possible nerve root entrapment and spinal cord compression due to degenerative changes

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3
Q

cervical spondylosis- symptoms and signs

A

symptoms- bilateral/unilateral neck pain, referred pain into shoulder/arm/head, this can be somatic referred pain or radicular referred if degenerative irritate a spinal nerve, neck stiffness
signs- decreased cervical ROM (rotation and SF most affected), pain on PAIVM on affected level, altered posture, dermatomal changes if nerve root involved, degenerative changes on X-rays

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4
Q

cervical disc herniation

A

caused by degenerative weakening of annulus- nucleus propulsus goes through, affects individuals in 30’s, C6-7 and C5-6 most common affected. Uncommon- IVD in Cx supported posteriorly by PLL and laterally by uncovertebral joints, nucleus pulposus is Cx is largely fibrocartilaginous

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5
Q

cervical disc herniation- risk factors

A

age, smoking, lifting heavy objects, diving

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6
Q

cervical disc herniation- source of pain and disc prolapse

A

source= tear of outer annulus, inflammatory process, can be bilateral issue with all 4 limb (affect gait)
disc prolapse- centrally, posterolateral, bulge

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7
Q

cervical disc herniation- symptoms

A

S&S depended on size and location, acute and rapidly worsening neck pain (central or unilateral), referred pain to scapula, pain worse on ext and prolonged flex activities (cough and sneeze), antalgic posture- head held in flex

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8
Q

cervical disc herniation- different positions symptoms

A

posterolateral and spinal nerve will produce- radicular referred pain into arm and hand (lateral canal stenosis), paraesthesia and or anaesthesia into UL
if prolapse is posterior and central spinal cord may be involved= myelopathy, may get signs of cord compression (central canal stenosis)

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9
Q

Facet OA

A

degenerative disorder affecting synovial joints, most common in >65 y/o

pathology- synovitis, disintegration of articular cartilage, osteophyte formation, joint space narrowing

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10
Q

Facet OA- symptoms and signs

A

symptoms- local- often unilateral pain, somatic pain referral into shoulder/scap region depending on levels affected, stiff neck
signs- decreased ROM into facet closed pack position- ext and ipsilateral SF/ rotation
pain reproduced on PAIVM affected levels

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11
Q

Facet OA- position of affected levels

A

C2-C3= posterior to ear on left side, C3-C4= pain on neck on right side, C4-5= pain on neck lower than C3, C5-C6= pain over right shoulder, C6-7= pain over scapula

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12
Q

Facet OA- X ray changes

A

cartilage destruction of facet joints, loss of joint space, osteophyte formation around joint margin, IVD and vertebral bodies normal

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13
Q

Cervical radiculopathy

A

links to disc prolapse= nerve irritated, 9 cervical nerve roots, emerge above corresponding vertebrae but below IVD

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14
Q

Cervical radiculopathy- lateral canal stenosis

A

narrowing of I-V formation, causes nerve root compression and irritation, neurological changes

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15
Q

Cervical radiculopathy- nerve roots as source of pain

A

irritation of cervical nerve root in the IV foraminal usually in the medial half by: inflammation, posterolateral disc prolapse, degenerative changes of facet joints, most common affects C5/6/7

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16
Q

Cervical radiculopathy- signs and symptoms

A

acute severe arm pain in dermatomal distribution, altered sensation in dermatomal distribution, myotomal/reflex change, may have neck pain, arm pain worsened on movements or postures closing down IV foramina (e.g. ipsilateral rotation), significant night pain, antalgic posture, overhead arm positions may relieve

17
Q

Cervical myelopathy

A

compression of SC by- severe central degenerative changes (osteophytes) and large central disc prolapse
S and S- chronic neck pain, less severe than lateral canal stenosis, associated symptoms= mild gait disturbances and numb clumsy hand, may have other cord signs= bladder dysfunction/ gait disturbances, LL dysfunction

18
Q

cervical myelopathy- aggravated by

A

aggravated with movements or postures that decrease size of spinal canal (e.g. ext), usually medical emergency

19
Q

what is whiplash

A

whiplash is an acceleration-deccerlation mechanism- may result from rear end or side impact, may result in bony or soft tissue injuries
rear end collision mechanism: hyperextension (affects anterior cervical structures) occurs first followed by hyperflexion (limited by chin on chest)

20
Q

possible lesions in whiplash

A

muscle strain (SCM and scalenes), facet joint (capsule strain), ligament sprains/tears- ALL/PLL/IVL, IVD- prolapse and annular tears, SC and nerve root traction injuries, vertebral artery, concussion, Thoracic outlet syndrome

21
Q

Whiplash- clinical picture

A

whiplash is a multi-level/tissue/pathology disorder, be aware of potential for instability and cervical artery trauma, often involves BOS component (PTSD)

22
Q

Whiplash- duration and biological symptoms

A
duration= 6 weeks - 1 year
biological= pain, stiffness, headaches, nausea, dizziness, referred pain, paraesthesia, blurred vision, difficulties swallowing
23
Q

Whiplash association disorder (WAD) classification

A

WAD 1- neck pain only and no physical signs, WAD 2a- neck pain with alterations in movement and muscle recruitment and local mechanical hyperalgesia, WAD 2b- above plus psychological impairment, WAD 2c- above plus generalised hyperalgesia, WAD 3- above plus neuro sign, WAD 4- fracture/dislocation

24
Q

postural dysfunction

A

no tissue damage/ pathology, pain is a result of tissue overstress/ strain

25
Q

postural dysfunction S and S

A

symptoms= widespread neck pain radiating into shoulders and head, worsened by prolonged postures, often easier in morning and worse at end of day
signs- poor upper quadrant posture= trigger points, may have fall AROM and absence of joint signs, find postures that work better

26
Q

cervicogenic headache

A

down to irritation of nucelli in top of cervical spine (trans general nucleus- TGN) or cranial nerves. Onset- often pain related to trauma, postural strain and degenerative disease, precipitating factor- sustained neck postures or movements

27
Q

cervicogenic headache- S and S

A

dysfunction in upper Cx can present at- headaches, face/eye/TMJ/ear pain pain in suboccipital region
nausea, dizziness, SQ- headaches (5D’s) and 2 N’s

28
Q

5 D’s and 2 N’s

A

dizziness, drop attacks (faiting), dysarthria ( loss of speech), dysphagia (loss of ability to swallow), diplopia (blurred vision)
Nause, numbness and nystagmus

28
Q

Convergence theory

A

pathomechanics- referred pain from upper cervical spine structures

29
Q

somatic referred pain from upper Cx spine

A

front of head- C0/1= pain above eyebrows, C1/2- pain at eyebrows= C1/2, C2/3= pain at the eyes
back of head- C0/C1= pain is in head, C1/2= pain at occiput and upper neck, C2/3= pain in the head and into neck

30
Q

cervicogenic headaches- symptoms

A

classically unilateral, side consistency, associated with suboccipital or neck pain, pain starts in neck and spreads to head, variable severity, rarely has other symptoms, no pattern, aggravated by neck movements or sustained head or neck posture

31
Q

cervicogenic headaches- signs

A

forward head posture, positive upper cervical joint findings- decreased upper cervical AROM, decreased upper cervical accessory ROM, weakness in deep neck flexors, trigger points in muscles supplied by C1-C3- nerves