case 1 Flashcards
what does DRSABCs stand for
danger, responsiveness, send for help, airways, breathing, CPR
what is shock
shock is a state of circulatory failure that impairs tissue perfusion and may lead to tissue hypoxia
what are our 5 types of shock
cardiogenic, hypovolaemic, septic, anaphylactic, neuroplastic
what is cardiogenic shock
a type of distributive shock whgereby low blood pressure and poor perfusion is caused by the malfunctioning of the cardiac muscle
what is hypovolaemic shock
low blood pressure due to a large scale loss of blood
what is anaphylactic shock
this is when we get allergen activation of the mast cells, releasing histamine, which then results in widespread vasodilation of the blood vessels, thus extreme vascular leakage, and smooth muscle contraction so we get slowing of blood flow
what is septic shock
body becomes infected with widespread infection which results in widespread inflammation, vasodilation, microvascular thrombosis and endothelial dysfunction
what is neuroplastic shock
this is when we get damage to the CNS and damage to the Sympathetic vasomotor tone. so we see unregulated PNS activity so we get widespread vasodilation and bradycardia as the sympathetic innervation cannot increase the MABP.
why is the periosteum key in bone healing
this is when the osteogenic protenitor cells are contained. so the new bone growth occurs from here
potential adverse effects of morphine and how do we monitor
Drowsiness/CNS Depression – monitor the level of consciousness [LOC]
Respiratory Depression – Frequent monitoring of respiratory rate [>12b/min], rhythm,
depth
Nausea/Vomiting – Ask patient about symptoms and administer anti-emetics with the
morphine
Hypotension – Frequent blood pressure monitoring
Bradycardia – Frequent monitoring of heart rate and rhythm
how does morphine relieve pain in a person
Morphine binds to opioid receptors, specifically the μ, d, and k types for
analgesia, in the CNS (brain and spinal cord). Morphine’s receptor is a G-coupled
protein receptor that acts to inhibit adenylate cyclase, reducing concentrations of
the 2nd messenger cAMP. This in turn opens potassium channels which has the effect
of hyperpolarising neuronal cell membranes so they become unresponsive to action
potentials. Calcium entry into the same cells is also restricted so inhibiting
neurotransmitter release. Both mechanisms lead to a decrease in the transmission
of pain impulses.
define analgesia
inability to feel pain
analgesic is a drug that removes pain sensations
why is morphine usually IV delivered
fast, passes first pass metabolism, allows us to do small dose incriments as its fast acting. so we can give some, if it doesn’t work, give more
what do we do if the effects of the analgesic are too much
give an opiod antagonist, so something that fights it and binds to the drug so that they can no longer bind to their receptors
naloxone
what are treatment options for a compound fracture
Debride the wound: stabilise the fracture, remove any foreign material,
Debridement: Clean wounds by flushing with saline - doesn’t hurt as much as water