CASC Stations 4 Flashcards

1
Q

Types of indecent exposure?

A

Exhibitions - sudden powerful urge with little attempt to avoid capture and no further attempt to contact victim
Disinhibited - alcohol, drugs, stress, dementia, hypomania
Aggressive, impulsive and antisocial

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2
Q

Reoffending rate of indecent exposure

A

20-30%

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3
Q

Predictors of sexual reoffending

A

• Male Gender
• Low IQ
• Lower Age
• Previous criminal record/Previous history of sexual offence
• History of child hood sexual abuse, and other abuse
• Juvenile sex offences
• Deviant sexual practices like excessive masturbation, self
reported deviant sexual fantasias and urges.
• Involved in Paraphilia (Exhibitionism, Voyeurism)
• Previous history of poor engagement with services
• Personality disorder (esp. antisocial PD)
• Access to victims, Attitudes towards victim - low victim
empathy, justification of sexual crimes, and that they’re
entitled to express their high sexual drive
• Intoxication with drugs/ alcohol

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4
Q

Characteristics of high deviancy paedophiles

A
  1. They committed offences outside and inside the family
  2. They offended against both boys and girls
  3. They were twice as likely to have committed previous sexual
    offences
  4. They were more likely to have been abused as a child.
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5
Q

Paedophile assessment construct

A

Assess circumstances of the act/offence, Intent of
the act, Grooming activity
Obtain Patients’ view about the offence and arrest;
Denial, minimisation, justification, lack of remorse,
lack of guilt, low victim empathy, feelings of
‘entitlement’
Obtain relevant Personal history (traumatic
childhood, history of childhood abuse), Past
psychiatric history, Drug and alcohol history
Obtain relevant Psycho-sexual history (deviant
sexual practices, excessive masturbation and urges,
sexual fantasies, Paraphilia, h/o paedophile
activity, sexual preference - attracted to children,
explore sexual activity & relationship with adults
Previous Forensic history (sexual and non-sexual
offences), juvenile sexual offences, history of
cautioning, conviction, sentencing, previous history
of similar offences)
Assess Current social circumstances- friends and
support system
(Unemployment, homelessness, lack of stable

Relationships, abnormal personality traits-
impulsivity, lack of empathy)

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6
Q

What is fitness to stand trial?

A

Ability to conduct oneself appropriately in court

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7
Q

Sx of anorexia

A

• Weight loss > 15% and below expected BMI (Body mass
index) of 17.5 or less
• Body image distortion-Fear of fatness held as an intrusive
overvalued idea
• Avoidance of fattening foods, with behaviours aimed at
losing weight like vomiting, purging, over exercise, use of
appetite suppressants and/or diuretics.
• Amenorrhoea in women, a loss of sexual interest and
potency in men
• Pubertal delay if onset is early

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8
Q

Sx of bulimia

A

• Persistent preoccupation with food & eating and an irresistible
craving for food
• Binges-episodes of overeating
• Attempts to counteract the ‘fattening’ effects of foods by one or
more of the following like self-induced vomiting, alternating
periods of starvation, purgative abuse, over exercise and use of
appetite suppressants, diuretics
• Morbid fear of fatness with imposed ‘low weight threshold’

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9
Q

Physical health questions in eating disorders

A

a. Menstrual changes
When was your last period? Are you menstruating regularly?
b. Changes in libido
c. Symptoms of anemia: weakness, lethargy, constipation
Do you feel the cold badly?
Have you noticed any weakness in your muscles?
Have you fainted or had dizzy spells?

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10
Q

Predictors of poor prognosis in eating disorders

A
  1. Old age at illness onset
  2. Life time history of anorexia
  3. Poor impulse control
  4. Pre-morbid obesity
  5. Poor social adjustment
  6. Substance misuse
  7. Co-morbid affective disorder
  8. Low self esteem
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11
Q

Risk factors for poor outcome in anorexia

A

• Male sex
• Late onset
• Longer duration of disorder before presentation
• Chronic course of illness
• Excessive weight loss
• Longer duration of amenorrhoea
• Bulimic features (more bingeing behaviors, Vomiting and
purging as part of the clinical picture)
• Poor social adjustment
• Poor parental relation ships, Greater family hostility
• Psychiatric co-morbidity especially cluster C personality
disorders, depression, OCD, substance misuse and autistic
spectrum disorders
• Anxiety when eating with others

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12
Q

What does OP treatment of eating disorder involve

A

Outpatient management should involve a psychological treatment
with physical monitoring provided by a healthcare professional
competent to give it and to assess the physical risk of the illness to
the patient, and the monitoring should normally continue for at least
6 months

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13
Q

Goals of IP treatment in eating disorders

A

• Addressing physical and psychiatric complications
• Development of a healthy meal plan
• Addressing underlying conflicts, such as low self-esteem and
planning new coping strategies
• Enhancing communication skills§
• Hospitalise for weight restoration and monitoring as part of
the medical management. It is reasonable to aim for a weight
gain of between 0.5 and 1 kg each week, & weight restoration
takes between 8 and 12 weeks
• Rehydration and correction of serum electrolytes.
• Instigate nutritional rehabilitation and involve the dietician for
nutritional counselling: Aim for a target weight, refeeding
programme- a balanced diet of 2500 – 3000 k calories/day
provided as three or four meals a day with supplementary
snacks.
• Following weight restoration, the patient should be offered
outpatient psychological treatment, and typically this
outpatient treatment and physical monitoring following
inpatient weight restoration should continue for at least 12
months
• Offer support at meal times, monitor her food intake
• Eating pattern should be supervised by a nurse to provide
support, reassurance and to ensure that the patient does not
induce vomiting (or) take purgatives.
• Monitor for Suicidality, impulsive behaviour

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14
Q

Psychological treatment of eating disorder

A
Psychoeducation
CBT
IPT
Focal psychodynamic
Family interventions in CAMHS (NICE)
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15
Q

CBT in ED

A

The psychologist would attempt cognitive restructuring to
identify automatic negative thoughts and to challenge core beliefs.
CBT has been particularly used with the aim of modifying
abnormal cognitions about shape, weight and eating and the
behavioural component focuses on behavioural experiments
include self-monitoring of weight, goal setting, assertiveness
training and relaxation.

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16
Q

Outcome of Anorexia

A

Ø More than half of sufferers make a recovery.
Ø The average duration of illness is between 5 or 6 years
Ø Sometimes it may take a long time like 15-20 years for people
to make complete recovery
Ø 1 in 5 of severely ill people may also die

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17
Q

Treatment of bulimia

A

Diary of eating habits and developing self control
Dietary counselling and education
High dose fluoxetine can reduce urge to eat
Psychotherapy
Family interventions in CAMHS (NICE)
CBT
IPT

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18
Q

Outcome of bulimia

A

Ø More than half of sufferers recover and would cut their
bingeing and purging by atleast half, will help people to get
back some control of their life
Ø Recovery usually takes place slowly over a few months or in
cases over many years
Ø Both CBT and IPT work just as effectively over a year
Ø Research evidence suggests that Combining medications and
psychotherapy is found to be more effective than either
treatments on its own.

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19
Q

When is risk of refeeding highest

A

First 2 weeks

Usually within 4 days of refeeding

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20
Q

Characteristics of refeeding syndrome

A
  1. Oedema
  2. Excessive bloating
  3. Features of congestive cardiac failure
    Low phosphate
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21
Q

What happens in refeeding

A

Most effects result from a sudden shift from fat to carbohydrate
metabolism and a sudden increase in insulin levels after refeeding
which leads to increased cellular uptake of phosphate.
Refeeding increases the basal metabolic rate. Intracellular movement
of electrolytes occurs along with a fall in the serum electrolytes
including phosphate, potassium, magnesium, glucose, and thiamine.
Significant risks arising from refeeding syndrome include confusion,
coma, convulsions, and death. The shifting of electrolytes and fluid
balance increases cardiac workload and heart rate. This can
lead to acute heart failure. Oxygen consumption is also
increased which strains the respiratory system and can make
weaning from ventilation more difficult.

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22
Q

Treatment of refeeding

A

Refeeding syndrome can be fatal if not recognized and
treated properly. If potassium, phosphate or magnesium are low then
this should be corrected. Prescribing thiamine, vitamin B complex
(strong) and a multivitamin and mineral is recommended.
Biochemistry should be monitored regularly until it is stable.

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23
Q

Monitoring for refeeding

A

• Measure U&Es and correct abnormalities before re-feeding.
• Recheck U&Es every 3 days for the first 7 days and then weekly
during re-feeding period.
• Attempt to increase daily caloric intake slowly by 200- 300 kcal
every 3-5 days until sustained weight gain of 1- 2 pounds per
week is achieved.
• Monitor patient regularly for development of tachycardia or
oedema.

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24
Q

Management of PND

A
Psychoeducation to patient and family
Monitoring those at risk
Individual/marital counselling
CBT
Antidepressants
Prevention through enhancing coping and stress management techniques, relaxation training
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25
Risk factors for PND
1. Older Age 2. Single mother 3. Unplanned pregnancy 4. Personal history of depression 5. Family history of depression 6. Poor social support 7. Significant other psychosocial stressors
26
Risk factors for postpartum psychosis
1. Personal history of major psychiatric disorder 2. Previous postpartum psychosis (30% risk of developing psychosis in the subsequent pregnancies) 3. Family history of major psychiatric disorder 4. Single parenthood 5. Lack of adequate social support
27
Advice for breastfeeding if on antipsychotics
• Most psychotropic medication are not indicated during lactation period, Therefore discontinue and change it to bottle- feeding preferably, it gives the mother some rest especially during the acute phase of the illness. • If the mother still decided to breastfeed, then the benefits would have to be weighed against the risk of exposure in the infant. In which case I would use low dose lowest optimum dose of antipsychotics preferably Sulpiride or olanzapine (recommended by the Maudsley guidelines 2007) • I would tend to avoid drugs with long half life and the time feeds as to avoid peak plasma drug levels in the milk
28
Good prognostic factors for postpartum psychosis
* Acute onset * Affective illness * Good social support. * First episode * Well adjusted pre morbid functioning * Lack of social adversities.
29
Recurrence rate of postpartum depression
20-30%
30
How many women with postpartum depression will go on to have another, non-postpartum depressive episode
50%
31
Overview of family therapy
v Family Therapy see families as systems where every member’s behaviour impacts on other members and this in turn changes their behaviour which further impacts on other members. v The system involves behaviour (what is said and done) and how this is interpreted by each person and in turn how this influences their behaviour and so on. v Members of a family may not be aware of how the things they say and do are understood and experienced by others nor the beliefs and emotions that result. v The aim is to avoid blame but to discover why people feel and behave the way that they do and how this can be changed through understanding the system of interactions and developing strategies.
32
Structure of family therapy
v Ideally as many members of the family attend as are able but therapy can occur with just two members and even on an individual basis on occasion. v A therapist is in the room with the family and will ask questions and support discussion. v Everyone is given time and space to express their view and answer questions. v In a separate room there is a therapy team who observe the discussion and provide feedback or suggest other questions. v The family can meet all members of the team. v Therapy can take place weekly or less frequently and is usually recommended over a period of 3 months.
33
Goals of family therapy
v The goals are for members of the family to gain an understanding of how the others experience and think about their behaviour and their emotional responses. v In turn an understanding develops about how these perceptions contribute to certain behaviours and how those are in turn perceived by other members of the family.
34
What is high EE?
v High Expressed Emotion refers to a theory about the family environment in terms of how other family members interact with a patient and their attitude towards them. It describes a tendency to be overly involved or overly critical of their relative with mental disorder. Interactions will tend to be more hostile, intense, oppositional and negative towards the patient. These kinds of interaction, which are not unique to families with mental illness, can be understood as an environmental stressor. This stressor can increase the likelihood of a relapse. Examples could include speaking in a raised voice, irritability towards the patient, blaming them for their symptoms and expressing their experience of burden as a care-giver. Similarly over-involvement in the patient from feelings of guilt around their being unwell can have a detrimental effect. This might take the form of overprotectiveness or excessive self-sacrifice due to anxiety around the patient’s illness. This may also prevent patients from having the freedom to develop skills more independently.
35
Evidence of family therapy
Reduces rate of relapse of SCZ | Improves medication adherence in SCZ
36
What skills can family therapy teach?
``` Psychoeducation of SCZ Recognising precipitating factors and relapse signatures Structured problem solving Comm skills Stress management ```
37
How to assess appropriateness for family therapy
- Explore their experience as a care giver - Ask about interpersonal behaviours and dynamics related to challenges in care-giving (areas of disagreement or frustration with the patient or another care-giver are key examples). - Appraise whether there is a willingness to accept alternative explanations about others behaviour and that their personal experience may be different to what is expected. - You can suggest that this misunderstanding others’ viewpoints for each family member may lead to repeating problematic patterns of interaction. - Appraise whether they can see a benefit from increasing their understanding of how each family member experiences the others and interacts. - After explaining what Family Therapy is and the kinds of things that may be addressed, you can ask directly whether they think this is something they could engage in.
38
What is CBT
Cognitive therapy is a way of helping people to cope with stress and emotional problems. The idea behind it is quite simple – ‘the way we think about things affects how we feel emotionally’. When people are depressed, they often have negative thoughts about themselves, their future and the world in general. These thoughts come automatically into their minds. These negative thoughts or ‘cognitions’, undermine their self-confidence, and make them feel even more depressed leading to unhelpful behaviours. The therapist will work with you to identify the thinking and behavioural patterns that contribute to how you feel, and help you to make changes. ü CBT looks at ‘here and now’ issues rather than things from the past. It helps people to learn new methods of coping and solving problems, which they can use for the rest of their lives.
39
Structure CBT
50 minute session once a week for 8-12 weeks In the first few sessions, the client and the therapist decide which problems seem to be the most important. Clients/patients take an active part and carry out ‘homework’ tasks between sessions. They will often be asked to keep a diary of their thoughts, feelings and behaviours in the situations that they find particularly stressful. They then discuss these in detail in the sessions with the therapists, asking themselves whether or not their ways of thinking are - realistic. They can then learn to change these ways of thinking to use more helpful ones. CBT helps by changing your thinking and behaviour patterns and in fact, the last few sessions focus on ‘relapse prevention’. Hence, it is effective in reducing the chances of relapse.
40
Cognitive distortions
``` Catastrophising Labelling Dichotomous thinking Minimisation/magnification Selective abstraction Personalisation Overgeneralisation Arbitrary inference ```
41
What is selective abstraction?
Selectively picking failures and errors while ignoring positive features
42
What is personalisation
Assuming personal responsibility for all that | has gone bad
43
What is labelling
Here the distortion leads to a global, over | generalized negative view of oneself;
44
With is arbitrary inference
Predicting without sufficient evidence
45
Explaining psychodynamic therapy
ü Psychodynamic psychotherapy is one form of talking treatments and is based on listening and talking. It involves exploring past conflicts (journey of self exploration) in relation to your current problems in order to make change possible ü It aims to treat people with long histories of serious emotional difficulties. These are linked to personal development, often over many years, and sometimes right from early childhood. The aim in psychotherapy is to look more deeply into the emotions, conflicts and distress behind your mental health diagnoses. ü There is a small possibility that you may feel worse before you feel better, as the therapy requires an uncovering of painful feelings and memories that be experienced as a relief but it may also stir up uncomfortable feelings.
46
Structure of psychodynamic therapy
ü A brief course of psychotherapy would last about three months, and a course of group therapy or long term individual therapy can last for a couple of years. ü Your motivation and relationship with the therapist is extremely important. The initial consultations aim to help think about this, in collaboration with a therapist. If you are well motivated and set yourself realistic goals, psychotherapy is likely to be of benefit. However the therapist can only offer guidance and the ultimate responsibility of changing is with the patient. ü It is offered on a weekly basis- same day, same time and same place Each session normally last fifty minutes and group therapy meetings are for one and a half hours.
47
Explain systematic desensitisation
In this therapy, first the patient will be taught relaxation exercises to help them control the anxiety and panic. Then we make a list of hierarchy of - situations that you find difficult to face. We arrange them from the least difficult to the most difficult and you may find it easier to face situations if you move from the least to the most difficult, Then you start by facing the easiest situation, whilst managing to relax. When you feel comfortable with that situation, you then go onto the next one. You will have to practice this daily. e.g. like going out of the front door of your house, going out to your garden from your house, then going out to a nearby shop with a family member/friend and then going out to a supermarket with a family member/friend and so on. ü Practice the steps until it no longer causes anxiety. Once you feel confident with one step, move on to a more difficult step and repeat the practice. ü If there are difficulties in getting out of the house, then we can arrange for the therapist to come to your house to help you initially. ü Your family members and or your partner have an important role in the treatment and it will be very helpful if they can also be involved to improve confidence and support them.
48
Explain ERP
• This is a simple but highly effective technique • It is usually done in graded steps • The active participation of clients is necessary • The situation can be real or imagined (a real-life situation will be more effective) • It can be practised regularly with self-exposure tasks. • It is used particularly in treatment of phobias and OCD The treatment strategy involves exposing the individual to stimuli that trigger anxiety or discomfort, and then having the individual voluntarily refrain from performing his or her ritual or compulsion. For each ritual the individual will be required to list a range of situations that cause anxiety and The individual would then rate each of these situations according to the amount of anxiety or distress that would arise if he or she did not perform the particular ritual. These are then arranged in order according to those that generate the least anxiety or discomfort to those that generate the most anxiety or discomfort. The first task in the list would be an activity that is mildly discomforting but not too difficult, while the last task in the list would be the most difficult task that the individual can imagine. Before starting theses exercises, it is important to provide training for slow breathing exercises and relaxation. These exercises can be used prior to commencing each step of the graded exposure hierarchy to ensure that the individual is calm and relatively relaxed at the beginning of each graded exposure session.
49
CBT in OCD
The Cognitive component of CBT involves identify and modify maladaptive cognitions and seeks to reduce to suppress and avoid Obsessional thoughts. The Behavioural component involves Behavioural techniques such as exposure and response prevention which would be exposing the individual to those situations that triggers anxiety (or) discomfort and refrain the individual voluntarily from performing his (or) her ritual. The therapist works with the patient and helps the individual plan a graded programme of exposure tasks. These anxiety provoking situations are then arranged in order according to those that generate the most anxiety (or) discomfort (hierarchy of tasks) the patient is also provided training for relaxation and taught one step to the next until the person can mange the last step with minimal anxiety without getting anxious. The situation can be real (or) imagined (a real life situation will be more effective), usually done in graded steps and can be practiced regularly with self-exposure tasks.
50
Principles of IPT
• The focuses of treatment are the current interpersonal relationships and their relationship to the development of illness. • Emotional problems are best understood by studying the interpersonal context in which they arise. • It does not make any assumptions about the causation of the illness, but aims to use the connection between the onset of depressive symptoms and the current interpersonal problems as a focus of treatment • Resolving the interpersonal problem is seen as a route to recovery.
51
Areas of interpersonal areas related to illness development in IPT
The four interpersonal areas related to illness development include: a. Grief (loss of a loved object/relation) b. Interpersonal disputes e.g. Conflict with family members, colleague or a friend c. Change of role (e.g. graduation, new mother, retirement, job loss, medical ill health) d. Interpersonal deficits.(inadequate interpersonal relationships)
52
Structure of IPT
• It is time-limited • Treatment lasts for 12-16 hour long weekly sessions, offered by trained therapist • The therapist starts with a diagnostic phase, in which the disorder is identified and explained. Inventory of all close relationships is created in early part of therapy. • The therapist will now try to link the depressive symptoms with one of the four interpersonal areas and will pursue strategies specific to one of these problem areas. • The therapist is directive and takes an active & supportive stance. • The patient is encouraged to identify and carry through change in interpersonal relationships and to test the possibility of consequent improvement in their symptoms. • The final phase involves assessment of improvement and develops ways of identifying and countering depressive symptoms, should they recur in the future and plan for termination of therapy.
53
Explain IPT
Interpersonal Psychotherapy (IPT) is a form of structured psychotherapy • It is time-limited that is normally delivered over 12-16 weekly sessions. • IPT focuses primarily on relationship problems. It helps people recognise the problems they face with other people and to make changes in their relationships. • IPT looks at the ways in which a person’s current difficulties in relationships contribute to their psychological stress, and in turn looks at the ways psychological problems affect relationships. • When an individual can deal with relationship problems more effectively, their psychological symptoms often improve.
54
What happens in early phase of IPT
During the first few sessions, the therapist will be talking with your about your depression and current important relationships to see how they are linked You will work with your therapist to complete an Interpersonal Inventory, which is basically a review of your key relationships, looking at their strengths and problems and how other might be able to assist you in your recovery. This will help you to identify those relationships, which it would be most useful to focus on during therapy. You will agree a contract with your therapist, outlining the focus of the work for the remaining sessions.
55
What is the middle phase of IPT
During the middle sessions of treatment, you and your therapist will discuss your agreed main area of interpersonal difficulties and work on making positive changes. There are several tasks assigned during these sessions. This will include a) monitoring current relationship triggers for depression b) working on improving communication, and c) discussing your emotional reactions to your relationship problems. You might decide to invite someone who is important in your life to one of these sessions to help them to understand and support you in the work you are starting to do.
56
What happens in the final phase of IPT?
In the last few sessions, you and your therapist will discuss feelings about therapy ending and the progress you have made during the treatment. The therapist will spend some time with you planning ahead for any other problems you anticipate in the future and how to use the new skills and supports you have developed.
57
Response rate to clozapine
60%
58
Common SEs of clozapine
sedation, hypersalivation, constipation, | hypotension, tachycardia and weight gain
59
Prevalence of clozapine agranulocytosis
1%
60
When is risk of clozapine induced agranulocytosis highest
4-18 weeks
61
Clozapine use in pregnancy
Consider detailed USS in first trimester Risk of neonatal withdrawal syndrome Breast feeding not advised due to high conc in breast milk
62
Risk of metabolic syndrome in SCZ
Four fold risk compared to gen pop
63
Monitoring for metabolic syndrome in antipsychotic use
v Physical examination: Weigh patients, check blood pressure, waist circumference and track BMI at each visit v Take a history and record whether known risk factors are present or absent at baseline and monitor at regular intervals v Perform baseline laboratory tests including fasting glucose, fasting lipids, total cholesterol, Low density lipoproteins (LDL), high density lipoproteins (HDL), triglycerides, alanine aminotransferase (ALT) and gamma glutamyl transferase (GGT) v The choice of antipsychotic medication should be based on the cardio metabolic risk profile of each medication v Perform Physical examination and Repeat Laboratory tests at week 6, week 12, week 52. After 1 year if all the laboratory tests are within normal range, repeat tests annually
64
Diabetes monitoring in those on antipsychotics
OGTT or FPG, HbA1c if fasting not possible at baseline and every 12 months
65
Cholesterol monitoring in those on antipsychotics
Those on clozapine or olanzapine; fasting lipids every 3 months for a year then annually Other antipsychotics; fasting lipids at 3 months then annually
66
SEs of lithum
``` Thirst, polyuria Blurred vision Dry mouth Metallic taste Fine tremor of hands ```
67
Long term SE of lithium
``` Weight gain Kidneys Thyroid - hypothyroid Tremor Skin rash ```
68
Lithium toxicity sx
``` Severe tremor Vomiting Diarrhoea Blurred vision Slurred speech Lack of coordination Confusion Muscle twitching ```
69
Baseline investigations for lithium
FBC, U&Es, TFT, ECG
70
Monitoring requirements on lithium
Lithium plasma levels after 5-7 days then every week until therapeutic reached, then every 3 months Renal, thyroid and ECG every 6 months Lithium card to carry all the time
71
Prevention of lithium toxicity
Fluid balance Stop if toxicity suspected and contact GP Avoid overexercise or salt free diet Women - contraception
72
Duration of maintenance treatment of lithium for bipolar
2-3 years
73
Risk of relapse if lithium stopped in pregnancy
70% in 6 months
74
SEs of valproate
Nausea, weight gain, tremors, hair loss | Thrombocytopenia
75
SEs of typical antipsychotics
Sedation, low BP, dry mouth, constipation, blurred vision, urinary retention, EPSEs, weight gain
76
SEs of olanzapine
``` Drowsiness Weight gain Hypotension Impaired glucose tolerance Constipation ```
77
SEs of Risperidone
``` Weight gain Increased prolactin Headache Nausea EpSEs ```
78
SEs of Quetiapine
Weight gain Dry mouth Drowsiness Postural hypotesion
79
SEs of Amisulpride
Insomnia Nausea Constipation Increased prolactin
80
SEs of TCAs
``` Sediation Low BP Dry mouth Blurred vision Urinary retention Weight gain Arrhythmia ```
81
SEs of SSRIs
``` Nausea Abdominal pain Sweating, anxiety Insomnia Sexual dysfunction ```
82
What happens in ECT
The exact mechanism of how it works is not known. During ECT, a small amount of electric current is passed across your brain. This current produces a fit/seizure, which affects the entire brain including centres that control thinking, mood, appetite and sleep. Repeated treatments alter the chemical imbalance in the brain and bring them back to normal. This helps you begin to recover from your illness.
83
Explaining anaesthesia pre-ECT
An ECT treatment involves having an anaesthetic. The anaesthetist will ask you to hold out your hands so you can be given an anaesthetic injection. It will make you go to sleep and cause your muscles to relax completely. You will be given some oxygen to breathe as you go off to sleep. Once you are fast asleep, a small amount of electric current is passed across your head and this causes a mild fit/seizure in the brain. There are little movements of your body because of the relaxant injection that the anaesthetist gives.
84
Response to ECT
80% of depressed patients improve
85
Sessions of ECT
8-10 sessions given twice a week
86
Normal PR interval
0.12-0.2seconds | 3-5 small squares
87
Normal QRS
<0.12seconds/<3 small squares
88
In which leads is T wave inversion abnormal
Leads I, II and V4-6
89
Physiological causes of prolonged QTc
``` Female Children and elderly Severe exertion Anorexia IHD/LVH/MI/Long qt syndrome Low K+, Calcium or Mg2+ ```
90
Risk factors of adverse effects of prolonged QTc
Antipsychotic/antidepressants/Abx Cardiac FHx Symptoms - CP, SOB, LoC, dizziness Diabetes/HTN/cardiac hx/smoking
91
Causes of sexual dysfunction
1. Physical illness- diabetes, cardiovascular complications 2. Psychiatric illness- depression 3. Relationship difficulties 4. Substance misuse 5. Prescribed drugs such as SSRIs, venlafaxine etc
92
Rx of sexual dysfunction due to antidepressants
v Watchful waiting- to see if symptoms subside. Spontaneous remission- seen in 10% of cases and a partial remission in 11% v Reduction in dose of offending drug should be tried first (Dose alteration) v If no response, discontinuation of the antidepressant and possible switch to a different drug less likely to cause sexual dysfunction should be tried. (Sexual side effects are less common with Reboxetine- 5-10%, Mirtazapine- 25%, Bupropion). Bupropion is used as a first line drug in USA with minimal risk of sexual adverse effects and as an adjunct & antidote in patients with SSRI induced sexual dysfunction. (Not licensed in UK) v Other options- Addition of another agent to counteract the sexual side effects (‘Comination’ eg mirtazapine, bupropion, or ‘As required’ eg sildenafil, cyproheptadine, amanatadine, yohimbine etc. Other drugs of use- Selegiline transdermal patches, Sildenafil (Viagra) and Agomelatine. Sildenafil is more effective than placebo at improving erectile dysfunction in men and in improving sexual function in women taking SSRIs
93
Construct for sexual dysfunction history station
Explore current problems and obtain more history (Mode of onset, duration, progression, precipitants if any) Acknowledge the difficulty in talking about Sexual problems, Reassure-common and could be treated Agree to speak to his partner Elicit briefly Symptoms of depression (Low mood, anhedonia, Fatigueability, disturbed sleep etc) Obtain detailed sexual history (Lowered libido, Arousal difficulties, Reaching orgasm too soon or Failure, Erectile failure, inability to maintain erection etc) Identifying possible triggers for sexual problems (Physical, psychological- ? depression, Environmental factors, Factors related to the partner) Addressing concerns- 1. Longer term effect of medication on sex life 2. Risk of stopping meds- not advisable
94
Sleep hygiene approaches
a. Avoid late evening exercise; reduce caffeine (or) alcohol intake/smoking, excessive daytime sleep and napping, large late meals and thinking about problems before going to bed. b. Encourage bed time routines, regular exercise (not in the evening), routine of rising and retiring at the same time each day, use anxiety management (or) relaxation techniques and sleep environment should be quiet, familiar and comfortable. c. Other measure like relaxation techniques, anxiety management, medication, yoga, listening to relaxation cassettes.
95
Withdrawal sx of GHB
tachycardia, irritability, restlessness, diaphoresis initially followed by the later development of hallucinations, tremors, insomnia, confusion, nausea, hypertension after 3-6 hours. After 12-48 hours seizures, confusion, hypervigilance can occur. The life threatening complications are those of acute renal failure, rhabdomyolysis, excited delirium, catatonic stupor and seizure.
96
Treatment of GHB withdrawal
Benzos