Cardiotocograph Flashcards

1
Q

Aside from CTG, how else can you monitor baby?

A

pinard stethoscope

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2
Q

List two complications of fetal hypoxia?

A

perinatal death
hypoxic ischaemic encephalopathy
cerebral palsy

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3
Q

List three maternal factors associated with fetal hypoxia

A
hypotension
epidural or spinal
respiratory disease
cardiac disease
diabetes
smoking
infection
drugs alcohol
thyroid disease
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4
Q

List two placental factors associated with fetal hypoxia

A
placental abruption
cord accidents
infection
chorioamnionitis 
pre-eclampsia
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5
Q

List three fetal causes of hypoxia

A
instrumental delivery
cord prolapse
malpresentations
infection
anaemia
prematuryity
haemorrhage
congenital anomalies
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6
Q

What are the four features of fetal heart rate?

A

baseline rate
baseline variability
presence/absence of accelerations
presence/absence of decelerations

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7
Q

What are reassuring baseline rates for CTG?

A

110-160 bpm

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8
Q

What are abnormal/worrying baseline HR?

A

<100

>180

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9
Q

What is normal baseline variability?

A

5 to 25 bpm

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10
Q

What is abnormal baseline variability?

A

<5 bpm for >50 mins

>25 bpm for >25 mins

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11
Q

Are accelerations reassuring or not?

A

reassuring! the absence of them is abnormal.

15 bpm for 15 sec or more

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12
Q

When is variable deceleration concerning?

A

lasting more than 60 sec
failure to return to baseline
biphasic W shape

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13
Q

What is a normal/non concerning cause of variable deceleration?

A

cord compression

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14
Q

Which type of deceleration is most indicative of fetal hypoxia?

A

late deceleration

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15
Q

List two causes of decreased variability

A

sleep
fetal hypoxia
drugs

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16
Q

List one drug that can cause decreased varability

A

opiates

magnesium sulphate

17
Q

List one cause of fetal tachy?

A

fetal hypoxia

chorioamnionitis

18
Q

List two factors associated with high risk pregnancy?

A

meconium stained liquor
fever
intrauterine growth restriction

19
Q

Explain the compensating mechanisms for hypoxia

A

reduced myocardial workload + reduced movement to conserve energy. No accelerations. Release of catecholamines which increase HR, promote peripheral vasoconstriction (to divert blood to vital organs), and promote glycogenolysis to increase energy supply.

20
Q

What are the signs of decompensation on CTG?

A

loss of baseline variability (reduced oxygen to brain)

myocardia hypoxia and acidosis- unstable baseline

21
Q

What is a pneumonic to remember the patterns of CTG changes in response to hypoxia?

A

ABCDE
Accelerations disappear
Baseline FHR increases
Compensated stress (stable basline FHR and normal variability)
Decompensation (unstable baseline and changes in varaibility)
End stage (Step ladder pattern due to myocardial failure)

22
Q

Are early decelerations normal?

A

physiological- head compression, resolves when contraction ends

23
Q

What is shouldering?

A

umbilical cord compression- fetus not hypoxic, compensating well. Variable deceleration

24
Q

What are concerning features of variable decelerations?

A

> 60 sec
biphasic shape
no shouldering
reduced baseline variability

25
Q

What do late decelerations indicate?

A

utero-placental insufficiency

26
Q

List two causes of late decelerations?

A

maternal hypotension
pre-eclampsia
uterine hyperstimulation