cardiology uworld Flashcards
CHADVASC score > 2, what drug do you give to reduce systemic embolic risk?
moa?
why is it better than warfarin?
apixiban or dabiGATRan – long term
direct inhibits factor X (Xiban)
direct thrombin inhibitor (gator)
less risk of hemorrhagic bleeding like warfarin–
indirect causes of QT prolongation?
antipsychotics: haloperidol
atrial fibrillation management that ONLY causes QRS prolongation
Flecainide class 1C
CHA2DS2VASc scoring?
CHF
HTN
Age >75 gets 2, 64-75 gets 1
DM
Stroke or TIA gets. 2
Vascular Dx (PAD, past MI)
Sex Category : FEMALES
Most common comorbidity with A-fib?
effect on the heart?
chronic HTN
atrial remodeling –> atrial fibrosis & dilation
*will see atrial enlargement, concentric LV hypertrophy
*if paroxysmal you won’t see anything on EKG
rapid and irregular pulse with no p waves ?
management?
atrial fibrillation with RVR (>100bpm)
rate and rhythm control
no p-waves where does it originate ?
atrial fibrillation
pulmonary veins
atrial fibrillation management if greater than ___ >bpm
if greater than 150>bpm –> synchronized cardio version
atrial fibrillation guidelines for hemodynamically stable patients (<150bpm)
rate control: non-dihydropyridines(diltiazam or verapamil) or b-blockers (metoprolol)
for long term : check CHA2DS2VASc FOR anti-coagulation tx (usually need it)
Low BP + sinus bradycardia –> next best step?
Atropine
what causes atrial tachycardia with second degree av block
digoxin )can also cause lots of other arrhythmias
progressively lengthening of PR interval followed by a drop in QRS ?
next best step?
Mobitz type 1 weinkeback
no further intervention
*also seen in long distance athletes
what is this? and what’s next best step?
complete AV block, dissociation between P waves and QRS
leads to decreased cardiac output, (thus sxms fatigue, weakness)
next best step cardiac pacing followed by pacemaker
who do you see this in?
what’s usual sxms?
what’s next step?
mobitz type 1
P-R gets longer longer then drops.
AV node delayed conduction
asymptomatic
can see it in athletes, b-blockers, or structural heart changes
–> observation
vs mobitz 2 constant P-R then sudden drop
what is this d/t?
what is the best next step and why? (2)
sine wave d/t hyperkalemia
tx with calcium gluconate to stabilize cardiac membranes
and then with insulin and glucose to drive the K back into cells
young male syncope episode
T-wave inversion on EKG
d/t?
will likely see (2)?
dx?
tx?
cause of death?
HOCM
AD - sarcomere (myosin binding) protein
1) LVOT
2) ischemia induced ventricular tachycardia: O2 supply mismatch (increase in O2 demand d/t more muscle mass & decrease in O2 supply due to myocardial disarray with microvascular dysfunction)
dx: echocardiography
tx: b-blockers or non-di’s (verapamil/diltiazam) to get more LV blood volume
cause of death: ventricular arrhythmia
syncope vs seizure – tongue biting?
syncope: frontal tongue biting
seizure: lateral tongue biting
elderly patient
bradycardia or w/ a-fib
ekg: sinus bradycardia, sinus pauses (delayed p waves), sinoatrial nodal exit block ( dropped p waves)
what is it ?
what is it due to?
sick sinus syndrome
age related degeneration of cardiac conduction system w/ fibrosis of sinus node
cardiac conduction system degeneration
young patient with rapid heart beat and regular**
tx?
AVNRT: (paroxysmal supraventricular tachycardia)
TWO distinct conduction pathways in the atrioventricular node
accessory pathways avoiding AV node: WPW
p waves buried in the complex
narrow QRS
tx: adenosine – interrupts the AV nodal reentry circuit
any patient with NARROW QRS complex and tachycardia what to give them?
supraventricular tachycardia:
give ADENOSINE
hemodynamically NOT stable + tachycardia
management?
SYNCHRONIZED CARDIOVERSION
What is this?
next step?
Ventricular fibrillation
next step: DEFIBRILLATION**
only defibrillation if its in V-fib or pulseLESS v-tachy
Sudden cardiac death with pt hx of MI or EF <30% is?
VENTRICULAR ARRHYTHMIA
what is this ?
next best step?
QT prolongation –> torsades de pointes
next step: MAGNESIUM SULFATE
what is this?
next best step in Hemostable vs hemoUnstable?
monomorphic ventricular tachycardia
regular wide complex tachycardia
stable: amiodarone
unstable WITH pulse : synchronized cardio version
intermittent, prominent A waves with high JVP
canon A waves
d/t atrioventricular dissociation
WPW
hemo unstable
hemo stable
unstable: electrical cardio version
stable: ibutilide or procainamide
cocaine induced MI
next best step?
give benzodiazepines: for agitation anxiety
then aspiring for platelet aggregation
nitroglycerin and CCB for vasoconstrictive pain
nitrates:
____ preload
____ left ventricular end-diastolic volume
____ myocardial oxygen demand
lowers preload
lowers ventricular end diastolic volume
lowers myocardial oxygen demand by reducing wall stress
stable angina
first line therapy
B-blockers – decrease myocardial contractility
pt with atypical angina (suspecting stable ischemic heart disease) what is the initial best step?
Exercise EKG
pt <50yo, smoker, recurrent chest pain at rest or sleep, with spontaneous resolution
ekg: ST elevation
Coronary angio: negative
what is it ?
best preventative tx?
best abortive tx?
vasospastic angina
d/t: hyperactivity of intimal smooth muscle causing coronary artery vasospasm
tx: prev- Calcium Channel Blocker (diltiazam, verapamil)
abortive: sublingual nitroglycerin
acsvd ___ % >
start on :
7.5%
statin
leads ii, iii, aVF st elevation
_____ MI
involves the ___ ventricle
____ CONFIRMS R ventricular MI
____ RV preload
_____ LV preload
____ SVR
inferior wall MI
effects R ventricle
V4R confirms RVMI – right sided precordial EKG
increase in RV Preload
decrease in LV Preload
increase in SVR
pt with MI (ST elevation) with bibasilar crackles in lungs
next best step?
pulmonary edema with MI
emergency PCI
loop diuretic Furosemide (but not with hypotension)
medication management for MI