Cardiology Flashcards

Asystole

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1
Q

☆In the case of a patient with cardiac arrest where Basic Life Support (BLS) has been performed and the monitor reveals asystole, what are the following steps that typically taken as part of Advanced Cardiovascular Life Support (ACLS)?

A
  1. Continue High-Quality CPR: Ensure that chest compressions are of high quality, with a rate of 100-120 compressions per minute and a depth of at least 2 inches (5 cm) in adults, allowing full chest recoil between compressions.
  2. Ensure Adequate Oxygenation and Ventilation: Provide ventilation with 100% oxygen. Use a bag-mask device or an advanced airway if available. Ensure proper placement and secure the airway device.
  3. Epinephrine Administration:
    • Administer 1 mg of epinephrine intravenously (IV) or intraosseously (IO) as soon as possible.
    • Repeat every 3-5 minutes during the resuscitation efforts.
  4. Check for Reversible Causes (H’s and T’s): Identify and treat possible reversible causes of cardiac arrest, known as the H’s and T’s:
  • H’s: Hypovolemia, Hypoxia, Hydrogen ion (acidosis), Hyper-/hypokalemia, Hypothermia, Hypoglycemia
    • T’s: Tension pneumothorax, Tamponade (cardiac), Toxins, Thrombosis (pulmonary or coronary), Trauma
  1. Reassess Rhythm and Pulse:
    • After 2 minutes of CPR, reassess the rhythm and check for a pulse.
    • If asystole persists, continue CPR and administer epinephrine.
  2. Consider Advanced Airway and Capnography:
    • If not already done, consider placing an advanced airway (endotracheal tube or supraglottic airway).
    • Use waveform capnography to confirm and monitor correct placement of the airway device and effectiveness of CPR.
  3. Communication and Coordination: Ensure effective team communication and coordination throughout the resuscitation efforts. Assign specific roles to team members and rotate compressors every 2 minutes to avoid fatigue.
  4. Decisions Regarding Termination:
    • If there is no return of spontaneous circulation (ROSC) after an appropriate duration of resuscitation efforts and all reversible causes have been addressed, consider discussing the potential for termination of resuscitative efforts based on the clinical situation and local protocols.

Throughout the resuscitation, it is crucial to follow ACLS guidelines and adapt the approach based on the patient’s response and any identified reversible causes.

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2
Q

☆What are the steps to treat VT in patients who have been saved from cardiac arrest and then develop ventricular tachycardia (VT)?

Note: The treatment approach depends on whether the VT is stable or unstable:

A

For Unstable Ventricular Tachycardia (with signs of hemodynamic compromise such as hypotension, altered mental status, chest pain, or heart failure):

For Stable Ventricular Tachycardia:
- Antiarrhythmic Medications:
- Amiodarone: 150 mg IV over 10 minutes, followed by an infusion of 1 mg/min for the first 6 hours.

  • Lidocaine: 1-1.5 mg/kg IV bolus, followed by an infusion of 1-4 mg/min.
  • Procainamide: 20-50 mg/min IV until the arrhythmia is suppressed, hypotension ensues, the QRS duration increases by more than 50%, or a maximum dose of 17 mg/kg is given.
  • Observation and Monitoring:
    • Continuous ECG monitoring in an ICU or similar setting.
    • Frequent reassessment for any signs of hemodynamic instability.
  • Immediate Synchronized Cardioversion:
    • Deliver a synchronized shock starting at 100 Joules. Increase energy levels if necessary.
    • Sedate the patient if time and circumstances permit, but do not delay cardioversion for sedation.
  • Correct Underlying Causes:
    Identify and treat reversible causes of VT, such as electrolyte imbalances (e.g., hypokalemia, hypomagnesemia), ischemia, or drug toxicity.
  • Advanced Airway Management and Support: Ensure the airway is secure and provide supplemental oxygen to maintain adequate oxygenation.
  • Hemodynamic Support: Administer fluids or vasopressors (e.g., norepinephrine, dopamine) if the patient is hypotensive.

My answer:
Shock him by either 1 360 J monophasic or 120-200 J
Biphasic shock

Note ( Amiodarone is indicated for REFRACTORY VF at the 3rd cycle of BLS )

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3
Q

What is the optimal O2 saturation after successful ACLS ?

A

94% or above

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4
Q

How to treat hypotension in ptn just had cardiac arrest and you got him back ?

A

The systolic BP must be 90 or above.
If less then IV fluid bolus of 1-2 L of normal saline is given if BP is still low then epinephrine should be given ( 0.1-0.5 ) ugg/kg/min 0.1-0.5 ug/kg/min of norepinephrine or dopamine 5-10 ug/kg/min

Note ( Dobutamine is not part of the ttt )

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5
Q

How to treat hypotension in ptn just had cardiac arrest and you got him back ?

A

The systolic BP must be 90 or above.
If less then IV fluid bolus of 1-2 L of normal saline is given if BP is still low then epinephrine should be given ( 0.1-0.5 ) ugg/kg/min 0.1-0.5 ug/kg/min of norepinephrine or dopamine 5-10 ug/kg/min

Note ( Dobutamine is not part of the ttt )

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6
Q

How to treat a ptn in the ward after Having a surgery and is now having Asymptomatic bradycardia 40/min ? Then will become symptomatic?

A

In the beginning since the ptn is Asymptomatic just observation is enough but once we found the drop of BP for example in this case 70/35 then you must give Atropin 0.5mg IV push that can be repeated upto a limit of 3mg

عطينا اتروبين لان سبب هبوط الضغط هوا bradycardia وليس العكس

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7
Q

How to treat a ptn with VT but stable ( alert, BP is stable ect.) ?

A

Give 150 MG of Amiodarone over 10 minutes ( chemical cardioversion ) is the first line ttt in stable ptn

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8
Q

What does S1 represent? Ans S2? S3? S4

A

S1= closure of M & T valves

S2= closure of A&P valves

S3= rapid rush of blood from the atrium to the ventricle

S4= sudden slowing down of blood flow by the ventricle as the atrium contract

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9
Q

Explain physiological S2 splitting. when it happens?

A

It’s a physiological splitting . It happens when the ptn takes deep inspiration, which causes negative pressure in the chest, leading to more blood flow to the right side of the heart. Therefore, the pulmonary valve will close after the aortic valve

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10
Q

What’s paradoxic splitting and when do you see that?

A

It happens with LBBB. In this case, the pulmonary valve will close as usual during inspiration, but because of LBBB, the aortic valve is delayed

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11
Q

What are the right steps to hear abnormal heart sounds? And what does S3 , S4 suggest when heard ?

A

It’s better to have the ptn lies in the left lateral decubitus position

Because S3 and S4 are low pitched sounds, it’s better to be heard using the bill of the stethoscope

S4 comes after S1 and it suggests: Hypertrophic obstructive Cardiomypathies or LVH

S3 comes after S2 and it suggests: acute systolic heart failure, LVHF with dilatation (causing more blood filling to the LV)

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12
Q

What’s the average of atrial rate in Afib?

A

400 f wave /min

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13
Q

☆What’s the expected ventricular rate in patinet with atrial Fibrillation?

A

In patients with (AF), the expected ventricular rate can vary widely. It typically ranges from 100 to 175 beats per minute (bpm) if not controlled. The rate depends on factors such as the patient’s underlying heart condition, medications, and overall health. In well-managed cases, the ventricular rate can be brought down to a normal or near-normal range (60-100 bpm) with appropriate treatment.

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14
Q

What’s the expected rhythm and R-R interval in Afib?

A

The rhythm is totally irregular
R-R Interval is typically unequal

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15
Q

What do you expect the QRS to be in Afib?

A

Usually normal unless there’s preexisting intraventricual conduction delay

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16
Q

What’s the casuse of Afib?

A
  1. **High Blood Pressure
  2. **Heart Disease:
    Including heart valve disease, CAS, congenital disease, HF
  3. **Thyroid Problems: Both hyperthyroidism and hypothyroidism
  4. **Sleep Apnea
  5. **Excessive Alcohol Consumption
  6. Stimulants: Caffeine, nicotine, and certain medications
  7. **Chronic Conditions: DM, obesity
  8. **Infections: Severe infections, such as pneumonia, can stress the heart and lead to AFib.
  9. Family History: A genetic predisposition can play a role in the development of AFib.
  10. Age: The risk of developing AFib increases with age.

11.Other factors like stress, physical illness, and certain medications

17
Q

What’s the sign on echo that shows ptn is dehydrated

A

IVC collapsed

18
Q

Why do we need to do a portable chest xray for patient with STEMI?

A

To rule out aortic dissection as a secondary cause to the STEMI

19
Q

What are the typical & Atypical symptoms of MI?

A

The classic presentation is:
Retrosternal crushing chest pain which may radiate to the arm, shoulder, neck, or jaw, which may be associated with dyspnea, nausea, palpitations, and diaphoresis

Atypical: Abdominal pain, upper back pain, isolated shoulder pain, neck, jaw pain, or dyspnea alone.

Note: Patients with uncotrolled DM might not have symptoms

20
Q

What is the criteria for STEMI on ECG? 3 criterias

A
  1. ST Segment elevation>= 1 mm in any two limb leads
  2. ST Segment elevation >= 2 mm in any two contiguous chest leads
  3. New onset left bundle branch block (LBBB)If the patient has a prexisting LBBB, the diagnosis of STEMI becomes considerably more complex. In such a scenario, the Sgarbossa criteria may be used to determine if an acute infarction is present.