Cardiology Flashcards

1
Q

Which anti-hypertensive/AF med should be stopped in HF?

A

CCBs

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2
Q

At what point do both micro and macroscropic changes become visible in cardiac tissue post MI?

A

12-24 hours post occlusion

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3
Q

Which artery supplies the left atrium?

A

Left circumflex artery

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4
Q

Which artery supplies the left ventricle?

A

Left marginal artery - branch of circumflex

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5
Q

Which cardiac defect is associated with turner’s syndrome?

A

Co-arctation of the aorta

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6
Q

Main 3 problems associated with Kartagener’s syndrome

A

Dextrocardia, bronchiectasis and infertility - last 2 due to ineffective cilia

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7
Q

Which cardiac defects are associated with Marfan’s syndrome?

A

Aortic root dilatation (causes aortic regurg), mitral valve prolapse and mitral regurg. Risk aortic dissection

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8
Q

Which cardiac defects are associated with congenital rubella syndrome?

A

PDA, ASD and pulmonary stenosis

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9
Q

What pulmonary artery systolic pressure is indicative of pulmonary hypertension?

A

> 30mmHg

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10
Q

Presentation of dilated cardiomyopathy

A

Left bundle branch block and right axis deviation - can present weeks to months post partum

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11
Q

Presentation of HOCM

A

Exertional syncope/pre-syncope. ECG shows LVH or asymmetrical septal hypertrophy

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12
Q

Cause of wolf-parkinson white

A

Abnormal congenital accessory pathway (bundle of Kent) bypassing AVN with episodes of tachyarrythmia. Allows a re-entry circuit

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13
Q

ECG signs of WPW

A

SHortened PR interval, Delta wave (slow upstroke of initial portion of the QRS complex) and QRS prolongation. ST segment and T wave are often in the opposite direction to the QRS complex. Can be exacerbated by valsalva manouvre.

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14
Q

Treatment for WPW

A

Only definitively treat when high risk of sudden cardiac death - radiofrequency catheter ablation. If low risk can offer pharmacological management for the tachyarrythmias,

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15
Q

What is wenkebache phenomenom?

A

2nd degree mobitz type 1 heart block - PR progressively gets longer until it drops a beat

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16
Q

Features of Marfan’s

A

Pectus excavatum, ectopic lentis, pes planus, high arched palate, recurrent pneumothorax, aortic root dilation, mitral prolapse and positive wrist and thumb signs

17
Q

When would you hear VSD murmur?

A

Pan-systolic

18
Q

What type of pacemaker mode is used in patients with chronic AF causing bradycardia?

A

VVI mode

19
Q

What is pulsus paradoxus?

A

A decrease in SBP of >10mmHg upon inspiration - in pericarditis or pericardial effusion it is a sign of impending tamponade and circulatory collapse

20
Q

If patient develops bundle branch block post MI, which vessel is most likely affected?

A

LAD

21
Q

Treatment for symptomatic mobitz type 2 heart block?

A

Permanent pacing as can develop to complete heart block

22
Q

Likely cause of large JVP V-wave

A

Tricuspid regurgitation

23
Q

Features of tricuspid regurgitation

A

JVP v-wave, peripheral oedema, right ventiricular failure, Pan-systolic murmur, paradoxical splitting of second heart sound, pulsatile liver, tender hepatomegaly, ascites and right atrial hypertrophy

24
Q

What is dressler sydrome?

A

A complication of transmural MI, weeks after initial infarct. Causes mild fever and pleuritic chest pain (exacerbated by lying down/relieved by sitting forward). Friction rub may be heard.

25
Q

What does an opening snap in early diastole indicate?

A

Severe mitral stenosis

26
Q

Which organism is most likely to cause rheumatic fever?

A

Group a strep aka strep pyogenes

27
Q

What is kussmaul’s sign?

A

Distension od the jugular veins during inspiration whilst sitting up

28
Q

Machinery like murmur in bady which is loud systolic extending into diastole

A

PDA