Cardiology Flashcards

1
Q

Causes of Clubbing

A
  • Common
    • Cardiovascular
      • cyanotic congenital heart disease
      • Infective endocarditis
    • Respiratory
      • Lung ca
      • Chronic pulmonary suppuration(bronchiectasis, lung abscess, empyema)
      • Idiopathic pulmonary fibrosis
  • Uncommon
    • cystic fibrosis
    • asbestosis
    • mesothelioma
    • cirhhosis
    • IBD
    • Coeliac disease
    • idipathic or familial thyrotoxicosis
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2
Q

Loud S1

A
  • Mitral stenosis
  • reduced diastolic filling time (eg tachycardia)
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3
Q

Soft S1

A
  • Prolonged diastolic filling time - first-degree heart block
  • delayed onset of left ventricular systole - left bundle branch block
  • failure of the leaflets to coapt normally - mitral regurgitation
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4
Q

S2

1) Loud aortic component
2) Soft aortic component
3) Loud pulmonary component

A
  1. Systemic Hypertension and congenital aortic stenosis
  2. Aortic valve is calcified and leaflet movement is reduced, or in aortic regurgitation when the leaflets cannot coapt.
  3. Pulmonary hypertension
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5
Q

Increased normal splitting of the S2

A

Caused by any delay in right ventricular empyting

  • right bundle branch block (delayed right ventricular depolarisation)
  • pulmonary stenosis (delayed right ventricular ejection)
  • VSD( increased right ventricular volume load)
  • Mitral regurgitation (earlier aortic valve closure leading to more rapid left ventricular emptying)
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6
Q

Apex beats

A
  • Pressure loaded
    • hyperdynamic, systolic overloaded
    • forceful and sustained impluse that is not displaced
  • Volume loaded
    • hyperkinetic, diastolic overloaded
    • forceful but unsustained impluse that is displaced down and laterally
    • aortic or mitrac regurgitation
  • Dysskinetic = cardiac failure generally, and is palpable over a larger area than normal and moves in an incoordinated way.
  • Tapping apex beat = mitral stenosis (palpable first heart sound)
  • Double or triple apical impluse = hypertrophic cardiomyopathy
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7
Q

Killip Class

A
  • Class I - no evidence of heart failure
  • Class II - mild heart failure, crackles over the lower third or less of the lungs, systolic BP>90.
  • Class III - pulmonary oedema, crackles more than one third of the chest, systolic BP>90.
  • Class IV - cardiogenic shock, pulmonary oedema, crackles more than one third of the chest, BP<90.

Class III/IV is associated with a >5 fold mortality risk.

Class II is associated with a >3 fold risk compared to Class I.

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8
Q

Clinical Classification of Angina

A
  • Typical angina meets all 3 of the following characteristics:
    • characteristic retrosternal chest discomfort - typical quality and duration
    • provoked by exertion or emotion
    • relieved by rest or glyceryl trinitrate or both
  • Atypical angina meets 2 of the above characteristics
  • Non-cardiac chest pain meets 1 or none of the above charactersitcs
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9
Q

Causes of Orthopnoea

A

*Cardiac failure*

Uncommonly

  • massive ascites
  • pregnancy
  • bilateral diaphragmatic paralysis
  • large pleural effusion
  • severe pneumonia
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10
Q

Differential of ankle oedema

A

Heart failure

  • history of cardiac failure
  • other symptoms of heart failure
  • elevated JVP

Hypoproteinaemia

  • Normal JVP
  • Odema pits and refills in 2-3 seconds

DVT/Cellulitis

  • Unilateral
  • Skin erythema
  • Calf tenderness

Drug induced - takes a calcium channel blocker

Lymphoedema

  • not worse at the end of the day
  • not pitting when chronic

Lipodema

  • not pitting
  • spares foot
  • obese woman
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11
Q

Drugs and syncope

A

Associated with QT interval prologation and ventricular arrythmia

  • Anti-arrythmics: amiodarone, procainamide
  • Gastric motility promotr-cisapride
  • Antibiotics: clarithromycin and erythromycin
  • Antipsychotics: chlorpromazine and haloperidol

Associated with bradycardia

  • beta blockers
  • verapimil and diltiazem
  • digoxin

Associated with psotural hypotension

  • most antihypertensives but especially prazosin and calcium channel blockers
  • anti-parkinsonian drugs
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12
Q

Risk factors for coronary artery disease

A
  • previous ischaemic heart disease
  • hypercholesterolaemia
  • smoking
  • hypertension
  • family history of coronary artery disease
  • diabetes mellitus
  • chronic kidney disease
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13
Q

Causes of a regular bradycardia

A
  • Physiological - athletes, during sleep
  • Drugs - beta blockers, digoxin, amiodarone
  • Hypothyroidism
  • Hypothermia
  • Raised ICP
  • 3rd degree atrioventricular block or 2nd degree
  • myocardial infarction
  • vasovagal syncope
  • jaundice
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14
Q

Causes of irregular bradycardia

A

Irregularly irregular

  • atrial fibrillation (in combination with conduction sustem disease or av nodal block due to):
    • alcohol
    • post-thoracotomy
    • idiopathic
    • mitral valve disease or any cause of left ventricular enlargment
  • frequent ectopic beats

Regularly irregular

  • sinus arrythmia
  • Type 1 2nd degree av block
  • pulse deficit
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15
Q

Cause of a regular tachycardia

A
  • hyperdynamic circulation
    • exercise/emotion
    • fever (allow 15-20 beats per minute for every degree celsius above normal)
    • pregnancy
    • thyrotoxicosis
    • anaemia
    • a-v fistula
    • beri-beri
  • congestive cardiac failure
  • constrictive pericarditis
  • drugs - salbutamol
  • denervated heart )diabetic resting at 106-120)
  • hypovolaemic shock
  • supraventricular tachycardia (>150)
  • atrial flutter
  • ventricular tachycardia (>150)
  • sinus tachycardia
    • thyrotoxicosis
    • pulmoary embolism
    • myocarditis
    • myocardial ischaemia
    • fever, acute hypoxia or hypercapnia
  • multifocal atrial tachycardia
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16
Q

Causes of irregular tachycardia

A
  • Atrial fibrillation
    • myocardial ischaemia
    • mitral valve disease or left ventricular enlargement
    • thyrotoxicosis
    • hypertensive heart disease
    • sick sinus syndrome
    • pulmonary embolism
    • myocarditis
    • fever, acute hypoxia or hypercapnia
    • alcohol, post thoracotomy, idiopathic
  • multifocal atrial tachycardia
  • atrial flutter with variable block
17
Q

Causes of postural hypotension

A
  • hypovolaemia
  • hypopituitarism
  • addisons disease
  • neuropathy
  • drugs - vasodilators, TCA’s, diuretics, antipsychotics
  • idiopathic orthostatic hypotension
18
Q

Causes of an elevated central venous pressure

A
  • right ventricular failure
  • tricuspid stenosis or regurgitation
  • pericardial effusion or contrictive pericarditis
  • superior vena caval obstruction
  • fluid overload
  • hyperdynamic circulation
19
Q

JVP wave form changes

A

Dominant a wave

  • tricuspid stenosis
  • pulmonary stenosis
  • pulmonary hypertension

Cannon a waves

  • complete heart block
  • paroxysmal nodal tachycardia with retrograde atrial conduction
  • ventricular tachycardia with retrograde atrial conduction or atrioventricular dissociation

Dominant v wave

  • tricuspid regurgitation
  • x* descent
  • absent in atrial fibrillation
  • exaggerated in acute cardia tamponade and constrictive pericarditis

y descent

  • sharp in sever tricuspid regurgitation or constrictive pericarditis
  • slow in tricuspid stenosis and right atrial myxoma
20
Q
A