Cardio Flashcards
Mid-systolic murmurs:
aortic stenosis, pulmonic stenosis
Holo-systolic murmurs
mitral regurgitation, tricuspid regurgitation
Late-systolic murmurs
MVP (always showing up late)
Early-diastolic murmurs
aortic regurgitation, pulmonic regurgitation
Mid/late-diastolic murmurs
mitral stenosis, tricuspid stenosis
What maneuvers increase venous return?
Squatting, LR, lying down
What maneuvers decrease venous return?
Valsalva, standing
S1?
AV valve closure
S1 marks the?
beginning of systole
S1 is loudest at?
Apex
Which valve closes first in S1?
mitral, then tricuspid
S2?
Semilunar valve closure
S2 marks the?
end of systole
S2 loudest at?
Base
Which valve closes first in S2?
Aortic, then pulmonic
S3?
rapid passive ventricular filling
S4?
atrial contraction into ventricles
Elevated BP?
120/129/ <80
Stage 1 HTN?
130-139/ 80-89
Stage 2 HTN?
> 140/>90
HTN urgency?
> 180/120
Treatment for elevated BP?
lifestyle, recheck in 3-6 months
Treatment for stage 1 HTN?
10 year risk <10% –> lifestyle; >10% –> 1 med, check in one month
Treatment for stage 2 HTN?
2 meds, recheck in one month
Sodium restrictions for HTN?
<2.4 g/d
HTN tx: uncomplicated (non-AA)
thiazide diuretics, ACE, ARB
HTN tx: a. fib
BB, ND CCB
HTN tx: angina
BB, CCB
HTN tx: post-MI
BB, ACE
HTN tx: systolic HF
ACE, ARB, BB, diuretics
HTN tx: DM/CKD
ACE, ARB
HTN tx: systolic (isolated) HTN
diuretics +/- CCB
HTN tx: osteoporosis
thiazides
HTN tx: BPH
alpha blockers
HTN tx: AA (non-DM)
thiazides, CCB
HTN tx: gout
CCB, losartan (only arb that doesn’t cause hyperuricemia)
HTN urgency
> 180/>120 + no end organ damage
HTN urgency treatment
Decreased BP by 25% over 24-48 hours using oral agents –> goal <160/<100
HTN emergency
> 180/>120 + end organ damage
HTN emergency treatment
Decrease BP by no more than 25% within first hour, then by an additional 5-15%, over next 23 hours using IV agents
When should you not follow HTN emergency treatment protocol?
-Ischemic stroke: not lowered unless >185/110 in thrombo candidates or >220/120 in non-candidates AND in aortic dissection: rapidly reduce to SBP of 100-120 in 20 minutes
Inferior leads:
II, III, aVF
II, III, aVF leads?
inferior
lateral leads:
I, avL, V5, V6
I, avL, V5, V6?
lateral
septal leads:
V1, V2
V1, V2 leads?
septal
V3, V4 leads?
anterior
Anterior leads?
V3, V4
First degree AV block?
long PR interval (>0.20 sec)
First degree AV block treatment?
observe
Mobitz Type I (Wenckebach)
long, longer, drop
Mobitz Type I treatment
- Symptomatic: atropine, epi, +/- pacemaker
- Asymptomatic: observe +/- cardiac consult
Mobitz Type II
PR constant, random drops
Mobitz Type II treatment
atropine, temporary pacing; permanent pacemaker = definitive
Third degree AV block:
no relationship between P waves and QRS complexes
Third degree AV block treatmnetn
temp pacing –> permanent pacing
Pathologic Q wave definition:
> 0.04 sec, >2 mm deep, >25% depth of QRS complex
Pathologic Q waves may be normal in what leads?
III and aVR
STEMI EKG definition?
ST elevations 1+ mm in at least 2 anatomically contiguous leads + reciprocal changes in opposite leads
STEMI EKG progression?
hyperacute T waves –> ST elevations/depressions –> T wave inversions –> pathologic Q waves
Describe QT in hypocalcemia?
prolonged
Describe QT in hypercalcemia?
QT shortened
Sick sinus syndrome?
brady-tachy syndrome; sinus arrest with alternating paroxysms of atrial tachy and brady
Treatment of sick sinus syndrome?
Permanent pacemaker
MC chronic arrhythmia?
a. fib
Describe P waves in a.fib?
No discernible P waves
Paroxysmal A. fib?
self-terminating within 7 days
Persistent a. fib?
fails to self-terminate, lasts > 7 days
Permanent a. fib?
> 1 year; refractory to cardioversion or nerve tried
Lone a. fib?
paroxysmal, persistent, or permanent without evidence of heart disease
CHADSVASC
CHF or LVEF <40% (1) HTN (1) Age 75+ (2) DM (1) Stroke/TIA/thromboembolism (2) Vascular disease (1) Age 65-74 (1) Sex: Female (1)
CHADSVASC Score interpretation
2+: mod risk; chronic oral anticoags
1: low risk; clinical judgement
0: no anticoag risk
Treatment for unstable a. fib:
DC cardioversion
What type of drug is diltiazem?
ND-CCB
MCC of atrial flutter
HF
Atrial flutter tx: stable
vagal, CCB/BB
Atrial flutter tx: unstable
DC
Atrial flutter tx:
radiofrequency ablation
Long QT can be caused by?
macrolides, TCAs, and electrolyte abnormalities
Definitive treatment of long QT?
AICD
What type of PSVT is most common?
AVNRT
Describe AVNRT?
two pathways within the AV node (slow and fast)
Describe QRS of AVNRT?
Narrow w/ no discernible P waves
Describe AVRT?
1 pathway in the AV node and a second accessory pathway outside of the AV node
Types of AVRT?
orthodromic and antidromic
Orthodromic is antegrade via?
AV node
Antidromic is antegrade via?
accessory
orthodromic is retrograde via?
accessory pathway
Antidromic is retrograde via?
AV nod
Orthodromic is wide or narrow?
Narrow
Antidromic is wide or narrow?
wide
Tx of stable orthodromic AVRT?
vagal, adenosine, IV verapamil
Tx of unstable orthodromic AVRT?
DC
Tx of stable antidromic AVRT?
flecainidine, procainamide, amiodarone
What drugs must be avoided in WPW?
digoxin, verapamil, BB
Tx for multifocal atrial tachycardia?
CCB, BB
Describe EKG of premature ventricular complexes?
T wave opposite direction of QRS
Tx of PVCs?
no treatment needed; BB if symptomatic; if frequent, w/u for heart disease
Describe ventricular tachycardia?
3+ consecutive PVC at a rate of >100 bpm
Where does ventricular tachycardia originate?
Bundle of His
MCC of ventricular tachycardia
CAD with prior MI
Describe sustained v-tach?
> 30 seconds; almost always symptomatic; life threatening; can progress to v. fib
Describe non-sustained v-tach?
brief, limited, usually asymptomatic
Two types of ventricular tachycardia
monomorphic or polymorphic
Tx: sustained, stable v-tach?
amiodarone, procainamide, sotolol (does not respond to vagal or adenosine)
Tx: unstable v-tach?
DC, followed by amiodarone
Tx: no pulse v-tach?
defib + CPR
Name two non-D ccb?
diltiazem, verapamil
What rhythm do most ventricular fibrillations start with?
VT
MCC of ventricular fibrillation?
ischemic heart disease
Chronic treatment of f. fib?
amiodarone or ICD (unless within 48 hours of acute MI, then recurrence rate is low; no LT tx needed)
What lumen reduction becomes symptomatic?
70%
Worst RF for CAD?
DM
Most important modifiable RF for CAD?
cigarette
MC RF for CAD?
HTN
Angina classifications?
I: only with strenuous activities; no limitations
II: with more prolonged or rigorous activities; slight limitations
III: with usual activity; marked limitations
IV: at rest; often unable to carry out any physical activity
Most useful non-invasive test for CAD?
stress test