cardio 5 Flashcards

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1
Q

HCM management?

A

Avoid volume deplesion BB/CCB Surgery if symptom persist

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2
Q

Complication?

A

HF Stroke SCD

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3
Q

BB/NDCCB mechanism for management?

A

Decrease contraction and increase diastole period–decrease LVOT also, decrease anginal symptom NDCCB if symptom persist despite CCB

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4
Q

Common time of Dressler syndrome?

A

1-6 weeks post-MI.

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5
Q

Uremic pericarditis treatment?

A

Hemodialysis

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6
Q

Acute aortic dissection risk factor?

A

HTN(70 %), Marfan Bicuspid aortic valve Cocaine age >60

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7
Q

Acute aortic dissection sign and symptom?

A

Severe chest pain radiating to back SBP difference of >=20 in 2 arms

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8
Q

complication?

A

Stroke(C.artery) AAR(root dilation) Horner syndrome(carotid sympathetic plexus injury) MI: Ostia obstruction Pericardial effution(pericardium) Hemothorax (pleural cavity) Renal injury: renal arteries Abdominal pain: mesenteric ischemia LE paraplegia (spinal arteries)

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9
Q

Is atherosclerosis is the risk?

A

Yes but not be as a single cause is usually found in a patient with thoracic and abdominal aneurythm

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10
Q

ECG future of Hypertrophic cardiomyopathy?

A

Tall long R wave in aVL and deep S wave in V3 Defect in depolarization in the anterior lead (I,aVL, V4,V5, and V6)

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11
Q

Pulmonary embolism ECG feature?

A

S wave in V1 Q wave in V3 T wave abnormality in V3

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12
Q

How many factors d/t mobtize type 1 with 2

A

6

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13
Q

Level of the block?

A

I-AV node II–below AV node(e.g Purkinje)

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14
Q

ECG feature?

A

I–(progressive PR prolongation with dropped QRS wave follow P wave at the end (grouped beat) and narrow QRS

II-PR interval remains the same but some unconducted p wave(dropped QRS with narrow(but wider than I) or wide QRS.

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15
Q

Exercise /atropin?

A

I–improve II–worsen

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16
Q

Carotid massage?

A

I–worsens type 1 II–paradoxically improve ll

17
Q

Risk of progression to complete HB?

A

I–low risk ll–high risk–indication for pacemaker

18
Q

When do we say PR is prolonged and QRS is wider?

A

PR>20OMS QRS>120MS

19
Q

Heart block risk?

A

athletes underling heart disease drug(digoxin,BB and CCB) old age

20
Q

A common cause of MR in westerns?

A

MVP

21
Q

MVP mechanism of pathophysiology?

A

myxomatous degeneration of valve leaflet and cordea tendinea.