Cardio Flashcards

1
Q

Neonatal Murmurs are common when?

A
  • Common in 1st days of life, don’t necessarily mean problem
  • If murmur present at birth, consider a valvular problem
  • The common benign transitional murmurs (PDA) aren’t audible until mins=hrs after birth
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Transitional neonatal murmurs:

A

o not audible until hrs after birth
o infant pink, not in resp. distress, pulses are palpable and symmetrical
o soft, heard at left upper midsternal border
o loudest during 1st 24 hrs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

When to evaluate for coarctation of aorta?

A

o murmur lasts longer than 24hrs,
o legs have difference of 15mmhg blood pressure from UE
o pulses in LE are hard to palpate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

4 innocent murmurs

A

Stills
Pulmonic Flow Murmur
Venous Hum
Carotid

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Stills Murmur

A

low frequency, systolic, grade less than 3
o Heard best along left sternal boarder
o Normal S2, no click,

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Pulmonic Flow Murmur

A

less than or equal to grade 3
o Heard best at upper left sternal border
o Normal S2, no click
o No diastolic murmur

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Venous Hum Murmur

A

high frequency, grade 3 or less
o Best heard sitting/standing at base of neck (infraclavicular or supraclavicular )
o Normal S2, systolic and or diastolic
o Stopped by compression of the jugular vein, change in head position, or assumption of supine position

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Carotid Murmur

A

grade 3, heard over carotid

o no click, no aortic stenosis, no diastolic murmur, normal S2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

If the right ventricle is enlarged how would the silhouette on the anteroposterior film?

A

apex of the heart tipped upward

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

If the left ventricle is enlarged how would the silhouette on the anteroposterior film?

A

apex of the heart tipped downward

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

If the right atrium is enlarged how would the silhouette on the anteroposterior film?

A

prominence right atrial border of the heart

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

If the left atrium is enlarged how would the silhouette on the anteroposterior film?

A

double shadow behind cardiac silhouette increase in subcarinal angle

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Atrial Septal Defect

A

fixed, widely split S2, right ventricular heave
• grade 3 systolic ejection murmur at pulmonary area
• large shunts cause diastolic flow murmur at lower left sternal border
• EKG→ rsR’ in V1
• Often asx

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Ventricular Septal Defect

A

holosystolic murmur at lower left sternal border w/RV heave
• Presentation & cause depend on size
• Clinical features→ FTT, tachypnea, diaphoresis when eating
• L→R shunt w/normal pulm vascular resistance
• Large defects can cause eisenmenger syndrome

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Atrioventricular Septal Defect

A
  • Often cant heat in neonates
  • Loud S2
  • Common w/downs syndrome
  • EKG left axis deviation
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Patent (Persistent) Ductus Arteriosus

A
  • Continuous machinery type murmur
  • Bounding peripheral pulses if large ductus
  • Presentation & course depend on size of the ductus & pulmonary vascular resistance
  • Clinical features→ FTT, tachypnea, diaphoresis w/feeds
  • L→R shunt w/normal pulm vascular resistance
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

5 Right sided obstructive lesions

A

Pulmonary Valve stenosis
Peripheral (Branch) Pulmonary Artery Stenosis
Subvalvular Pulmonary Stenosis:
Supravalvular Pulmnonary Stenosis:
Ebstein Malformation of the Tricuspid Valve

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Pulmonary Valve stenosis

A
  • No sx when milder/moderate
  • Cyanosis & often r sided HF in ductal dependent lesions
  • RV lift w/systolic ejection click→ heard at 3rd left intercostal space
  • S2 widely split w/soft to inaudible P2, systolic ejection murmur at pulmonary area
  • Dilated pulmonary artery on chest radiograph
19
Q

Peripheral (Branch) Pulmonary Artery Stenosis:

A
  • Systolic murmurs
  • can be heard over both lung fields anteriorly and posteriorly, radiating to axilla
  • Mild no pathologic pulm branch stenosis produces mumur in infancy that resolves by 6mo
20
Q

Ebstein Malformation of the Tricuspid Valve

A
  • “atrialized” portion of the RV is thin-walled, does not contribute to RV output
  • Portion of ventricle below displaced tricuspid is smaller in volume & represents the functioning RV
21
Q

3 Left Sided Obstructive leisons

A

Coarctation of the Aorta
Aortic Stenosis
mitral valve prolapse

22
Q

Coarctation of the Aorta

A
  • Absent diminished femoral pulses
  • Upper/lower extremity systolic bp gradient of >20mmHg
  • Blowing systolic murmur→ heard in back or left axilla
23
Q

Aortic Stenosis

A
  • Harsh, systolic, ejection murmur @ upper right sternal border w/radiation to neck thrill in carotid arteries
  • Systolic click at apex
  • CXR→ see dilation of ascending aorta
24
Q

Mitral Valve Prolapse

A
  • Midsystolic click
  • Late systolic whooping/honking
  • Sx→ chest pain, palps, dizziness
25
3 diseases of the aorta
Bicuspid Aortic Valve Marfan & Loeys-Dietz Syndromes Turners syndrome
26
Bicuspid Aortic Valve
* Increased risk of aortic dilation & dissection | * Often get valvular stenosis adhesions and more prone to dissections
27
Marfan & Loeys-Dietz Syndromes
* Cardiac manifestations→ aortic root dilation & MVP * At risk for aortic dilation & dissection * Often restricted from contact sports * B.blockers & ACE used to lower BP and slow rate of aortic dilation
28
Turners Syndrome
* Risk of aortic dissection | * Risk factors: HTN, aortic dilation, bicuspid aortic valve, coarctation of aorta
29
Tetralogy of Fallot:
* Hypoxia spells during infancy * 25% of pts have a right sided aortic arch seen on CXR * Systolic ejection murmur heard at upper left sternal border
30
Pulmonary Atresia w/Ventricular Septal Defect
* Sx depend on degree of pulmonary blood flow | * Pulmonary blood flow via PDA and/or aortopulmonary collaterals
31
Pulmonary Atresia w/Intact Ventricular Septum
* Cyanosis at birth * Pulm blood flow is always ductal dependent, rarely w/aortopulmonary collateral arteries present * RV dependent coronary arteries sometimes present
32
Tricuspid Atresia
* Cyanosis at birth | * EKG= left axis deviation, right atrial enlargement, LVH
33
Hypoplastic Left Heart Syndrome:
* Mild cyanosis at birth * Minimal ausculatory findings * Ductal closure= rapid onset of shock
34
Transposition of the Great Arteries
* Cyanotic newborn w/out respiratory distress | * MC in males
35
Total anomalous pulmonary venous return
* Abnormal pulm venous connection leading to cyanosis * Occurs with or w/out murmur, may have accentuated P2 * Right atrial enlargement & RVH
36
Truncus Arteriosus
* Early HF with or w/out cyanosis | * Systolic ejection click
37
Sinus Arrhythmia
* Normal variation of the heart rate * Rate varies w/respiratory cycle→ P-QRS-T intervals stay stable * Purely changes between the R’s * To test it→ speed up their HR, if it goes away that’s good, if it gets worse→ need to work it up
38
Atrial Premature Beats
much more common than premature ventricular beats • Triggered by ectopic focus in atrium • Particularly common during fetal and newborn periods • If they are isolated, they are benign and require no tx
39
Ventricular Premature beat
PVC, ventricular ectopy • Common (occur in 1-2% pf ppl) • Usually benign in otherwise normal pt • As HR increases, benign ventricular premature beats usually disappear • If exercise causes increase/coupling of contractions→ underlying dz, work up • Multifocal premature beats are always abnormal
40
Are Arrhythmias Common during 1st few days of life?
PAC, PVC | Yes they are benign & often resolve within the first week
41
Bradyarrhythmias in infants
often associated w/congenital heart block→ esp if pts mother has lupus, it can harm the child’s cardiac system o Causes: • If structural normal hear→ think lupus (of mother) • Structural cardiac abnormalities o Tx: pacing if CO is inadequate
42
Tachyarrhythmias in infants
o Wide complex→ doesn’t follow conducting pathway o Narrow complex (MC)→ SVT, can indicate structural dz, myocarditis, left atrial enlargement, aberrant conduction pathway
43
Treatment for Tachyarrhythmias in infants
o Tx: • Ice to face • IV adenosine • LT therapy w/digoxin or propranolol (unless they have WPW) • Cardioversion only if pt is hemodynamically unstable