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2021 > Cardio > Flashcards

Cardio Flashcards

1
Q

Common causes of AS

A
  • Bicuspid aortic valve (most common cause in the young)
  • Degenerative calcification (most common cause in the elderly)
  • RHD
  • Congenital
  • IE
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2
Q

DDx of ESM

A
  • AS

- HOCM

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3
Q

Signs of severity of AS (11)

A
  • Low volume pulse
  • Slow-rising pulse
  • Narrow pulse pressure
  • Heaving apex
  • Systolic thrill
  • Reversed splitting of S2
  • Soft or absent aortic component of S2
  • S4
  • Late systolic peaking of a long murmur
  • Signs of pulmonary HTN
  • Signs of pulmonary congestion (or cardiac failure)
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4
Q

Complications of AS

A
  • LVF
  • Sudden death
  • Pul HTN
  • Arrhythmias (AF, VT)
  • Heart block (calcification of the conduction system)
  • IE
  • Systemic embolic complications (disintegration of aortic valve apparatus)
  • Haemolytic anaemia
  • IDA (Heyde’s syndrome)
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5
Q

ECG changes in AS

A
  • LVH
  • LV strain pattern
  • Left atrial hypertrophy (bifid p waves in lead II)
  • Left atrial dilatation (inverted of biphasic p waves in V1-V2)
  • LAD
  • Conduction abnormalities (LBBB, 1st degree Hb)
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6
Q

CXR findings in AS

A
  • Post-stenotic dilatation of proximal ascending aorta (marked in bicuspid AV)
  • Rib notching (sign of coarctation of the aorta, frequently seen with bicuspid AV)
  • Calcification of the AV
  • Cardiomegaly (late stages)
  • Pulmonary congestion
  • Prominent pulmonary arteries (pul HTN)
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7
Q

Causes of MS

A
  • Rheumatic fever (most common)
  • Degenerative

Rare:

  • Congenital MS
  • RA
  • SLE
  • Carcinoid syndrome
  • Fabry’s disease
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8
Q

DDx of mid-diastolic rumbling murmur

A
  • L) atrial mass
  • L) atrial thrombus
  • Cor triatriatum
  • Severe MR (increased forward flow across the MV)
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9
Q

MS signs of severity (6)

A
  • Early opening snap (lost with calcified leaflets)
  • Increasing length of murmur
  • Signs of pulmonary hypertension
  • Signs of pulmonary congestion
  • Graham-Steel murmur (pulmonary regurgitation)
  • Low pulse pressure
  • Apical thrill

Other key findings:

  • AF (chronicity)
  • Malar flush
  • Tapping apex beat
  • RV heave, loud P2
  • Loud S1
  • Diastolic rumble at apex
  • Concomitant MR/AS/AR
  • Soft S1/absent OS (MS/MR)
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10
Q

Complications of MS

A
  • LA enlargement
  • AF
  • LA thrombus for
  • Pulmonary oedema
  • R) HF
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11
Q

ECG changes in MS

A
  • AF
  • L) atrial hypertrophy (bifid p waves in lead II) if in SR
  • L) atrial dilatation (inverted or biphasic P waves in V1-V2) if in SR
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12
Q

CXR findings in MS

A
  • Double right heart border (left atrial enlargement)
  • Splaying of the carina (dilated LA)
  • Pulmonary congestion
  • Prominent pulmonary arteries (pul HTN)
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13
Q

Causes of Chronic MR (13)

A
  • Rheumatic fever
  • MVP
  • IE
  • LV dilatation (functional MR) - dilated annulus from DCM or pap muscle restriction from infarct/ischemia
  • SAM (HCM, concentric LVH)
  • Marfan’s syndrome
  • Ehlers Danlos Syndrome
  • RA
  • SLE (Libman-Sachs Endocarditis)
  • MAC
  • Papillary muscle dysfunction (ischemia or degenerative diseases of the chordae)
  • Cardiomyopathies (restrictive, hypertrophic, dilated)
  • Osteogenesis imperfecta
  • Pseudoxanthoma elasticum
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14
Q

Causes of Acute MR (3)

A
  • IE
  • Rupture of chordae tendinae (IE, acute rheumatic fever, ischemia)
  • Trauma
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15
Q

Signs of severity of MR (9)

A
  • Soft S1
  • S3
  • S4 (if in SR)
  • Displaced apex beat (sign of LV enlargement), volume loaded
  • Precordial thrill
  • Mid-diastolic flow murmur
  • Widely split S2
  • Signs of pulmonary HTN
  • Signs of pulmonary congestion
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16
Q

S3 (lub de dub)

A
  • turbulent flow in the ventricles - best heard at apex/LSE
  • Due to rapid ventricular filling due to the increased blood volume in the LA due to the regurgitant volume in the previous cardiac cycle.
  • normal in young patients (<40YO)
  • older patients - HF - LV chordae are stiff and weak so reach their limit quicker than normal.
  • MR, HF
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17
Q

DDx of pansystolic murmur

A
  • MR (apex)
  • TR (LSE)
  • VSD (LSE)
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18
Q

ECG in MR

A
  • AF
  • LA hypertrophy (bifid P waves in lead II) if in SR
  • LA dilatation (inverted or biphasic P waves in V1-V2) if in SR
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19
Q

CXR in MR

A
  • Double right heart border (LA enlargement)
  • LA appendage
  • Splaying of the carina (dilated of LA)
  • Cardiomegaly
  • Pulmonary congestion
  • Prominent pulmonary arteries (pulmonary HTN)
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20
Q

Causes of chronic AR

A
  • Bicuspid aortic valve
  • HTN
  • Rheumatic fever
  • Aortitis (syphilis, Takayasu’s arteritis, Ank Spond, Reiter’s syndrome, Psoriatic arthropathy)
  • RA
  • SLE
  • CTD (Marfan’s syndrome, EDS, pseudoxanthoma elasticum, osteogenesis imperfecta)
  • Perimembranous VSD
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21
Q

Causes of acute AR

A
  • Aortic dissection
  • IE
  • Ruptured sinus of Valsalva aneurysm
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22
Q

Signs of severity of AR

A
  • Wide pulse pressure
  • Long duration of the decrescendo diastolic murmur
  • S3
  • Soft S2
  • Austin Flint murmur
  • Signs of Pulmonary HTN
  • Signs of LVF
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23
Q

Austin Flint Murmur

A
  • Low frequency mid diastolic murmur heard at the apex caused by:
    1) the aortic regurgitant jet impinging on the anterior mitral valve leaflet leading to functional MS
    2) the LV diastolic pressure rising more rapidly than the LA pressure.
  • mimics MS - MS can be differentiated by the presence of an opening snap and loud S1. However, the S1 may be loud in AR (reflecting a hyperdynamic circulation), but it is not palpable (as it would be in MS, which would give a tapping apex beat).
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24
Q

Corrigan’s sign

A
  • visible carotid pulsations in the neck
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25
Q

Quinke’s sign

A
  • capillary pulsations in the fingernails
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26
Q

De Musset’s sign

A
  • head nodding with each heart beat
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27
Q

Muller’s sign

A
  • systolic pulsations of the uvula
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28
Q

Causes of collapsing/bounding pulse

A
  • Anaemia
  • Fever
  • Pregnancy
  • Thyrotoxicosis
  • PDA
  • AVF
  • Severe bradycardia
  • Severe MR
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29
Q

ECG of AR

A
  • Usually no specific findings

- LVH +/- strain pattern (esp if co-existant aortic stenosis)

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30
Q

CXR of AR

A
  • Valvular calcification
  • Cardiomegaly
  • Pulmonary congestion
  • Prominent pulmonary arteries (pulmonary HTN)
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31
Q

Anacrotic pulse

A
  • slow volume, slow uptake - AS
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32
Q

Plateua pulse

A
  • slow uptake - AS
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33
Q

Bisferien’s pulse

A
  • mixed AR and AS - collapsing and slow uptake
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34
Q

Collapsing (Corrigan’s) pulse

A
  • AR, PDA, Hyperdynamic circulation
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35
Q

Small volume pulse

A
  • AS, tamponade
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36
Q

Alternans pulse

A
  • Alternating strong and weak beats - LVF
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37
Q

Pressure loaded apex beat

38
Q

Volume loaded apex beat

39
Q

Dyskinetic apex beat

A
  • large area of impulse and not co-ordinated - LV failure eg., anterior infarct
40
Q

Double impulse apex beat

A
  • characteristic of HOCM
41
Q

Tapping apex beat

A
  • palpable S1 - early MS/TS
42
Q

Causes of beat no palpable

A
  • normal
  • Emphysema
  • Obesity
  • Tamponade
  • Dextrocardia
43
Q

Right Ventricular Hypertrophy Criteria

A

Right axis deviation of +110° or more.
Dominant R wave in V1 (> 7mm tall or R/S ratio > 1).
Dominant S wave in V5 or V6 (> 7mm deep or R/S ratio < 1).
QRS duration < 120ms (i.e. changes not due to RBBB).

Right ventricular strain pattern with ST depression and T-wave inversion in V1-4.

44
Q

RVH Causes

A

Pulmonary hypertension
Mitral stenosis
Pulmonary embolism
Chronic lung disease (cor pulmonale)
Congenital heart disease (e.g. Tetralogy of Fallot, pulmonary stenosis)
Arrhythmogenic right ventricular cardiomyopathy

45
Q

RAD Criteria

A

QRS is POSITIVE (dominant R wave) in Lead II, Lead III and aVF
QRS is NEGATIVE (dominant S wave) in Lead I

46
Q

RAD Causes

A 
Left posterior fascicular block
Lateral myocardial infarction
Right ventricular hypertrophy
Acute lung disease (e.g. Pulmonary Embolus)
Chronic lung disease (e.g. COPD)
Ventricular ectopy
Hyperkalaemia
Sodium-channel blocker toxicity
WPW syndrome
Normal in children or thin adults with a horizontally positioned heart
47
Q

LAD Criteria

A

QRS is POSITIVE (dominant R wave) in Lead I

QRS is NEGATIVE (dominant S wave) in leads II, III and aVF

48
Q

LAD Causes

A 
Left anterior fascicular block
Left bundle branch block
Left ventricular hypertrophy
Inferior MI
Ventricular ectopy
Paced rhythm
Wolff-Parkinson White syndrome
49
Q

LVH Criteria

A

Sokolov-Lyon criteria: S wave depth in V1 + tallest R wave height in V5-V6 > 35 mm.

Limb Leads

R wave in lead I + S wave in lead III > 25 mm
R wave in aVL > 11 mm
R wave in aVF > 20 mm
S wave in aVR > 14 mm

Precordial Leads

R wave in V4, V5 or V6 > 26 mm
R wave in V5 or V6 plus S wave in V1 > 35 mm
Largest R wave plus largest S wave in precordial leads > 45 mm

Non Voltage Criteria:
Increased R wave peak time > 50 ms in leads V5 or V6
ST segment depression and T wave inversion in the left-sided leads: AKA the left ventricular ‘strain’ pattern

50
Q

LVH Pathophys

A

The left ventricle hypertrophies in response to pressure overload secondary to conditions such as aortic stenosis and hypertension.

This results in increased R wave amplitude in the left-sided ECG leads (I, aVL and V4-6) and increased S wave depth in the right-sided leads (III, aVR, V1-3).

The thickened LV wall leads to prolonged depolarisation (increased R wave peak time) and delayed repolarisation (ST and T-wave abnormalities) in the lateral leads.

51
Q

LVH Causes

A 
Hypertension (most common cause)
Aortic stenosis
Aortic regurgitation
Mitral regurgitation
Coarctation of the aorta
Hypertrophic cardiomyopathy
52
Q

Malar Flush Causes

A
  • MS
  • Hypothyroidism
  • Cold weather
  • Carcinoid syndrome
  • SLE
  • Systemic Sclerosis
  • Irradiation
  • Polycythaemia

Malar flush signifies a low cardiac output state with pulmonary HTN, often seen with patients with severe MS.

53
Q

Common cardiac cases:

A
  • MR (27%)
  • AS (18%)
  • AR (15%)
  • HOCM (14%)
  • Mixed mitral and Aortic Valve disease (13%)
  • Pul HTN +/- TR (13%)
  • Congenital Heart Disease (12%)
  • VSD (11%)
  • Mixed Aortic Valve Disease (10%)
  • Prosthetic valve (7%)
  • MVP (5%)
  • MS (5%)
  • Mixed MV disease (4%)
  • PR (4%)
54
Q

Pansystolic mumur ddx (3)

55
Q

Midsystolic murmur ddx (3)

A
  • AS
  • PS
  • HOCM
56
Q

Early systolic murmur ddx (2)

A
  • VSD (small or large and pul HTN)

- TR

57
Q

Late systolic murmur ddx (2)

A
  • MVP

- Papillary muscle dysfunction

58
Q

Early diastolic murmur ddx (2)

59
Q

Mid diastolic murmur ddx (4)

A
  • MS
  • TS
  • Austin flint murmur of AR, Carey Combs of acute rheumatic fever
60
Q

Pre-systolic murmur ddx (3)

A
  • MS
  • TS
  • Atrial myxoma
61
Q

Continuous murmur ddx (2)

62
Q

S4 (before S1) - Le lub dub

A
  • high atrial pressure reflecting a non-compliant ventricle, e.g., AS, HTN, LVH
  • always abnormal
  • rare to hear
  • stiff and hypertrophic LV
  • turbulent flow from atria that is contracting against a non-compliant ventricle
63
Q

10 questions of murmurs

A

1) Is it systolic or diastolic?
2) Description of murmur:
- if systolic: pan, ejection (harsh, musical), mid systolic
- if diastolic: early decrescendo, mid-diastolic/rumbling (?pre systolic accentuation if in SR)
3) Where is the murmur loudest & where does it radiate?
4) How loud is it? How long is it? Where is the peak?
5) How does the murmur change with respiration? Are any additional manoeuvres required? Valsalva, hand grip
6) How many murmurs? (listen in aortic area/LSE then out the axilla
7) Does S1, S2 or additional HS fit with out diagnosis/severity?
8) Do other findings fit or is there another lesion?
9) Are there additional OS, clicks?
10) How will you grade the severity clinically?

64
Q

Loudness of murmurs (6)

A

1-2: soft
3-4: moderate
5: loud
6: very loud - can be heard even with stethoscope off the chest wall

65
Q

MS - mid diastolic rumble, loud S1

A
  • mid-diastolic murmur - sometimes louder just before systole.
  • tapping apex beat
  • opening snap better at base (like widely split S2)
  • murmur best heard at apex with patient rolled on left side
  • often VERY LOCALISED and DOES NOT RADIATE.
  • accentuated by handgrip in mitral area
66
Q

LSE - listen for:

A
  • TR
  • PR/PS (also in pulmonary areas
  • AR (early diastolic decrescendo)
  • HOCM (no radiation to neck, pulse - jerky, +/- MR)
  • VSD

*Must listen in inspiration/expiration

67
Q

Gallavardin Phenomenon

A
  • dissociation between the noisy and musical components of the systolic murmur heard in aortic stenosis.
  • the harsh noisy component is best heard at the aortic area radiating to the neck due to the high velocity jet in the ascending aorta.
  • the musical high frequency component is best heard at the cardiac apex - can be confused with MR.

***However, the apical murmur of the Gallavardin phenomenon is ejection systolic and does not usually radiate to the L) axilla.

68
Q

ASD

A
  • soft ESM

- Fixed splitting of S2

69
Q

ESM of AS at aortic area

A
  • if quiet but hear an ESM at LSE, think of HOCM.
70
Q

Once cardiac auscultation completed:

A
  • sit patient up:
  • percuss and listen to lung bases
  • look for sacral oedema

lie patient down:

  • feel for liver edge - ? is it pulsatile
  • spleen - ? IE

Ankles - gently
Pedal pulses!

71
Q

Causes of TR

A 
Usually secondary:
- Functional (RV dilatation), LV failure
- Pulmonary HTN
- PPM leads
Primary tricuspid pathology
- Endocarditis, carcinoid, trauma
- Congenital: ebstein, AV canal, repaired TGA
72
Q

Key findings of TR

A
  • JVP: prominent v waves
  • RV heave
  • Pansystolic murmur at LLSB loudest on inspiration (not always audible)
  • Pulmonary HTN (loud P2, RV heave, fixed split S2)
73
Q

TR signs of severity

A
  • Pulsatile liver
  • RV failure
  • Cirrhosis (ascites)
74
Q

Pul HTN causes (5)

A

Group 1 - primary PAH:

  • scleroderma - hands/face
  • ank spond - back
  • idiopathic

Group 2 - LV failure
Group 3 - Lung disease - ILD/COPD –> resp exam
Group 4 - CTEPH
Group 5 - mixed/other: myeloprofilerative disorders, sickle cell, thalassaemia, sarcoid, thyrotoxicosis

75
Q

HCM

A
  • ESM max at Erb’s point/Aortic region
  • Increased with Valsalva (decrease pre-load)
  • MR murmur due to SAM
  • Pressure loaded apex beat

ECG:

  • LVH by voltage
  • Prominent septal S/Q waves
  • Non-specific ant/inf TWI
  • LA enlargement
76
Q

VSD

A
  • Harsh PSM +/- thrill at LLSB
  • S3/S4
  • Pulmonary HTN
77
Q

ASD

A
  • Fixed, split S2 @ RUSB
  • DDx: S3, opening snap
  • Pul HTN, RV failure, RV heave, TR
  • Tricuspid flow murmur (mid diastolic rumble at LLSE)
  • Echo: RV volume overload, TOE/CMR/gated cardiac CT to confirm

Indications for closure:

  • unexplained stroke
  • Qp:Qs > 2:1
  • RV dilation/failure
  • Caution with pul HTN (>5 wood units)
78
Q

Austin Flint Murmur

A

Diastolic (MS) associated with severe AR - due to AR jet impingement on anterior mitral valve leaflet

79
Q

Graham Steele Murmur

A

Early diastolic murmur of PR, heard at Erb’s point

80
Q

What to think of when you can’t hear anything:

A
  • MS - listen again at the apex with bell, in L) lateral with handgrip
  • Pul HTN - Loud P2, RV heave, RV failure, associated conditions
  • ASD - fixed, split S2
  • HCM - pressure loaded apex beat, ESM at aortic area with Valsalva
  • Dextrocardia - check other side
81
Q

Parvus et tardus

A

Small volume, slow rising carotid pulse

82
Q

Kussmaul’s sign

A

JVP rise during inspiration (should fall with decreased intrathoracic pressure).

Signifies impaired RV filling (tamponade, RV failure, constriction)

83
Q

Displaced Apex Beat suggests?

A

Dilated Cardiomyopathy

84
Q

Pressure loaded apex beat

A

Slow ejection against increased afterload – ‘forceful and sustained’
Well localised, non-displaced

Causes: AS, HTN, coarctation

85
Q

Volume loaded apex beat

A

Rapid ejection of increased SV (increased preload) – ‘Forceful but unsustained’
Diffuse impulse felt over a larger area

Causes: DCM, AR, MR, shunts

86
Q

Iron targets in HF

A
  • Ferritin >100, or between 100-299
    AND
  • T Sat >20%

*in order to alleviate heart failure symptoms, increase exercise capacity and improve QOL

87
Q

MR Echocardiography (5)

A
  • Thickened leaflets (rheumatic)
  • Prolapsing leaflets
  • LA size
  • LV size and function
  • Doppler detection of the regurgitant jet in the LA; colour mapping of jet size and detection of reversal of flow in the pulmonary veins
  • Calcification of the mitral annulus - common in elderly
88
Q

MR indications for surgery:

A
  • severe, symptomatic MR
  • asymptomatic severe MR + dilated LV (LVESD > 40mm), reduced LVEF (<60%) or pul HTN (PASP >50mmHg)
89
Q

Cardiac Rehab (4)

A
  • Safe exercise
  • LOW
  • Diet
  • Smoking habits
90
Q

Lifestyle modification CVD (4)

A
  • LOW - BMI target 25 - Waist circum <94cm men, <80cm women
  • Exercise - 30 mins moderate intensity (brisk walk) at least 5 days/week
  • ETOH - 2x std drinks/day
  • Salt restriction - decrease snacks

2021 (34 decks)

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