Cardiac cycle and heart sounds Flashcards
what causes a murmur
sound produced when the flow of blood through a valve is turbulent or disrupted
not always indicative of a problem
Heart sounds: S1
lub (closure of A-V valves)
Heart sounds: S2
dub (closure of semilunar valves)
Heart sounds: S3
Ken-tuckee (from blood filling the ventricle)
Heart sounds: S4
Tenness-ee (atrial filling from high pressure SVC/IVC and pulmonary venous return; occurs before S1)
Regurgitation
turbulent flow sound when blood is moving in the wrong direction
Stenosis
turbulent flow when blood is moving through a stiff, damaged valve
auscultation location
APTM (All Physicians Take Money)
Aortic - R 2nd ICS
Pulmonary - L 2nd ICS
Tricuspid - L 4th ICS
Mitral - L 5th ICS mid clavicular line
what makes murmurs louder
inspiration - right (T&P)
expiration - left (M&A)
increased preload (decreased makes it softer
HOCM (hypertrophic obstructive cardiac myopathy)
thickened ventricular septum
exception to general rules of preload and afterload:
increased preload improves aortic murmur by pushing septum away from the aortic outflow track allowing blood to be ejected more easily
MVP (Mitral Valve Prolapse)
“see the MVP to be the MVP and everything will CLICK”
exception to general rules of preload and afterload:
increase in preload improves the “mid-systolic click” heard with MVP by allowing prolapsed leaflets to return to their normal orientation
Afterload effect on murmurs
Afterload: Pressure the ventricles have to generate to move blood forward into the arterial system (PVR)
increased - louder
decreased - softer
Aortic stenosis
Crescendo-decrescendo murmur
radiates UP to the carotids
Mitral regurgitation
“Rheu-mitral” (associated with rheumatoid fever”)
radiates to the axilla
best heard at apex
holosystolic murmur (plateau-shaped loud, blowing murmur)
Tricuspid regurgitation
“want to TRI some drugs?”
holosystolic
history of IVDA (intravenous drug abuse)