Cardiac Cycle Flashcards

1
Q

Describe what happens at each part of a typical ECG wave

A
P, atrial depolarization
Q, septal depolarization => atria
R, ventricular depolarization=> apex
S, ventricular depolarization => atria
T, ventricular repolarisation => endocardiogram
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2
Q

Name the valves and describe how they are affected by changes in pressure

A

RA => tricuspid => RV
RV => pulmonary => pulmonary arteries
Pulmonary veins => LA => mitral/bicuspid => LV
LV => aortic semilunar => aorta

Healthy valves have v little resistance so small pressure resistance so small pressure gradient across is enough for them to open

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3
Q

How to interpret a 2D ECG

A

Black areas = heart chambers
Top of cone, probe
Blue, away from probe
Red, towards probe (color doppler)

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4
Q

How do the valves operate under changing pressures

A

Opening valves due to pressure differences
AV, anchored to ventricle walls by papillary muscles, chord tendinae to prevent inverting
Bulge back into atria in ventricular systole, causes increase in atrial pressure and pulse of blood into jugular vein

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5
Q

How are the anchoring structures damaged and what happens as a result

A

After MI, AV valve everts into atrium

Backflow, can be seen as mitral regurgitation in ECG

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6
Q

Describe the pressure and volume changes in the heart

A

Fractional increase in LA pressure > LV pressure => mitral valve opens
LA contracts but not very much
LV contracts, isovolumic contraction (increase in pressure, no change in volume)
LV pressure increases, aortic valves still closed
Aortic valve opens when V pressure > aortic pressure
V ejection
End systolic pressure falls
LV pressure falls below aortic pressure, shuts => dicrotic notch => isovolumic relaxation
LV pressure falls below atrial pressure => mitral opens, V fills

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7
Q

Describe the steps in the pressure volume loop

A
Mitral opens, empty ventricles
Period of filling
Mitral closes
Isovolumic contraction
Aortic opens
Period of ejection
Aortic closes
Isovolumic relaxes
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8
Q

What information can you get from the pressure volume loop

A

Volume differences => ventricular volume/stroke volume
Diastolic function
Systolic function

Total area, stroke/work

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9
Q

Describe the peaks and troughs found in jugular venous pressure

A

A, atrial contraction (systole)
C, carotid pulse (carotid parallel to jugular, crushed jugular => P falls)
X, Atrial relaxation
V, R atrial filling in ventricular systole, closed tricuspid bulging, increasing RA pressure (pushes blood => venous system)
Y, Passive atrial emptying

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10
Q

What are the main characteristics of the jugular venous pulse

A

Biphasic, low pressure

Easily affected by other factors,

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11
Q

Describe the peaks and troughs found in the peripheral arterial pressures

A

Peak after ventricular systole

Smaller peak, wave of pressure bounces off peripheral vessels

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12
Q

What are the main characteristics found in the peripheral arterial pressure waves

What is it influenced by?

A

Monophonic and high pressure

Shape of peak and descending phase influenced by

  • Reflected waves
  • Resonance
  • Compliance
  • Interference
  • Damping

Shape and magnitude of arterial pulse wave varies along arterial tree

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13
Q

How do hydrostatic influences affect the jugular vein

What is the pressure found here

A

Will collapse at 5cm above RA due to hydrostatic influences, blood can still flow here

4-8mmHg

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14
Q

How does arterial pressure change

A

Changes as you move away from heart
Generally high
Increased height, increased BP as gravity moves everything down

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15
Q

How to judge if CVP is normal

A

Support patient at 45
JVP is height of collapse of internal jugular above manubriosternal angle

Collapse point = 3cm above MBS angle
If above 3cm, something is not right

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16
Q

Why use the internal jugular and not the external one

A
Anatomically closer than external to RA
Drains directly => SVC
Valveless, pulse can be seen
Vasoconstriction, 2ndary to hypotension can make external small, not visible
External is superficial and kink prone
17
Q

How does tricuspid stenosis affect the JVP wave

A

Atrial wave enhanced (RA pressure increases in atrial contraction due to increased tricuspid resistance)

Ventricular wave diminished (V pressure wave not transmitted well through stenotic tricuspid so less blood pushed out to periphery)

18
Q

How does tricuspid regurgitation affect the JVP wave

A

Atrial wave diminished

Ventricular wave enhanced (V contraction ejects blood through incompetent tricuspid, increased RA and J pressure as blood pushed into atria)

19
Q

How does the arterial pressure pulse change as you get further from the heart
Why

A

Greater time delay
Dicrotic notch gets looser

Changes due to dampening, reflection, interference, resonance
Affected by vasoconstriction, compliance, aging, hypertension, drugs

Reflected waves interfere w forward compression wave => altered pulse wave

20
Q

Describe how reflected waves in phase and out of phase affect pressure

A

Summative => P increased

Out of phase => P falls

21
Q

What are the normal heart sounds

A

S1, initiation of V systole, AV closer, low frequency

S2, closure of semilunar valves, high freq, shorter

22
Q

What are the secondary heart sounds

A

S3, Opening of AV valves, rapid refilling

S4, Atrial systole, rarely heard except when EDP raised

23
Q

What happens when EDP is raised

A

S3, S4 heard too

Gallop rhythm

24
Q

What are heart murmurs caused by

A

Turbulence in blood
Not always pathological, occurs in young and exercise

Caused by

  • Valve stenosis
  • Valve regurgitation

Heard between valve sounds

25
Q

Types of diastolic murmur

A

Mitral stenosis
-Diastolic murmur as ventricle fills

Aortic incompetence
-Early diastolic murmur with softening and prolongation of 2ndary sound

26
Q

Types of systolic murmur

A

Aortic stenosis
-Systolic murmur (high pressure)

Mitral incompetence
-Pansystolic (lush)