Cardiac 2 Flashcards
primary hypertension
dt genetics and lifestyle. (multifactorial and poorly understood)
secondary hypertension
dt pathological process e.g. alcohol, OCP, sympathomimetics, corticosteroids, cocaine
*if pt has malignant/moderate-severe hypertensive retinopathy and under 30yrs…
look for secondary cause!
signs and sxs of HTN
usu asymptomatic!
may have dizziness, facial flushing, HA, fatigue, epistaxis, nervousness
may have 4th heart sound
How to dx HTN
2 BP readings on 3 days….average
Malignant HTN
now called mod-severe hypertensive retinopathy. HTN w retinal hemorrhages, exudates, papilledema
(usu diastolic >120mmHg)
hypertensive urgency
severe HTN defined by a diastolic BO >120 mmHg in asx pts
Tests for HTN
UA, resting ECG, homocysteine, CMP, fractionated lipids
Risks factors for CAD and atherosclerosis
obesity, dyslipidemia, HTN, insulin resistance, prothrombic states, inflammation, smoking, elevated CRP
Signs and sxs of CAD
can be asymptomatic for decades.
sxs depend on where plaque is! angina, TIA, IC…unstable angina, stroke, limb pain, sudden death
IC
intermittent claudication. angina in legs
*if it is brought on by exercise and relieved by rest…
think CAD!
angina pectoris etiology
dt ischemia. leading cause of death in industrialized countries (dt atherosclerosis)
Signs and sxs of angina pectoris
transient precordial pain (brought on by exertion, relieved w rest); substernal heaviness. Lasts 2-5 min
Worse with: cold air, after meal, anxiety, morning
elevated HR and BP
*patterns consistent for an individual
(may also have SOB, belching, nausea, indigestion, dizziness)
variant angina
due to coronary a spasm. occurs at rest.
see ST elevation on EKG, happens at same time each day
silent ischemia
CAD without symptoms…DM!
chest pain DDX
GI dz, pulmonary dz, pericarditis, psychological, costochondral separation, costochondritis, dyspnea, aortic dissection, MVP, radiculopathy
unstable angina
NOT destroying heart cells. no change in cardiac enzymes. transient ECG changes
NSTEMI
Non-ST segment elevation MI. myocardial necrosis without acute ST elevation of Q waves. increased cardiac enzymes
STEMI
ST segment elevation MI. more dramatic symptom picture