cardiac Flashcards

1
Q

what are the two types of angina?

A

stable angina and unstable angina

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2
Q

what is stable angina

A

it is heart pain that resolves with rest

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3
Q

what is angina

A

chest pain upon exertion caused by heart ischemia (lack of blood supply)

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4
Q

what is unstable angina?

A

Chest pain that is not caused by exertion and does not go away with rest or medication.

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5
Q

why is angina dangerous?

A

It is a sign that someone could be going into cardiac arrest, or having a MI.

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6
Q

how do you treat angina?

A

With nitrates, GTN spray, and rest.

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7
Q

what are risk factors for an MI (heart attack)

A

non modifiable factors:
- older adult age
- family genetics/ history
- male sex

modifiable risk factors:
- hypercholesteremia
- hypertension
- obesity
- physical inactivity
- tobacco use
- hyperlipidemia
- hyperglycemia

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8
Q

what are signs and symptoms of an MI?

A
  • pallor/ pale peripheries
  • chest pain or discomfort
  • SOB
  • anxiety like feeling
  • pain/ discomfort in shoulder, neck, arm and jaw
  • sweating (diaphoresis)
  • nausea/ vomiting
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9
Q

what is an MI?

A

it occurs when there is a sustained myocardial ischemia due to blockage of a coronary artery. Most commonly occurs when a thrombus forms after an atherosclerotic plaque in a coronary artery ruptures. this leads to irriversible hypoxia damage to the effected section of the heart wall, resulting in necrotic tissue

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10
Q

what is myocardial stunning, hibermation and remodelling

A
  • myocardial stunning: temporary loss of contraction
  • myocardial hibernation: stop contracting to preserve life
  • myocardial remodelling: permanent loss of contraction
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11
Q

what is a STEMI MI

A
  • S-T elevation
  • full thickness nectosis
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12
Q

what is a NSTEMI MI

A
  • no S-T elevation
  • partial thickness necrosis
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13
Q

what tests are done for an MI

A
  • chest X-ray
  • ECG
  • angiogram
  • blood tests (full blood count, cardiac enzymes, u’s & e’s, thyroid function and blood glucose)
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14
Q

what are cardiac enzyme markers

A

Due to the breakdown of the myocardial cell membrane (sarcolemma), proteins leak out of the myocytes and become detectible in blood. Elevated cardiac enzymes are significant markers for cardiac injury/MI. Troponin T is a contractile protein that is only released from myocytes when necrosis has occurred, and thus it is highly sensitive, and specific, to cardiac muscle cell damage. Troponin levels may not be detectable for up to six hours after the onset of myocardial cell injury. It is usually measured at presentation and again 10-12 hours after the onset of symptoms or presentation

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15
Q

what is management of an MI?

A
  • decrease heart workload
  • increase oxygen delivery to the heart
  • re-perfuse the heart wall
  • dietary and lifestyle changes

MONA drugs
M- morphine to relieve chest pain
O- oxygen to ensure 02 stats are increased
N- nitroglycerin to reduce chest pain, vasodilation and decreases MI oxygen consumption and cardian workload
A- aspirin to slow platelet aggregation, reduce the risk of further occlusion

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16
Q

what is an angiogram and angioplasty for management of an MI

A

angiogram:
performed by injecting a contrast agent into the bloodstream, which makes the blood vessels surrounding the heart visible on an x-ray.

angioplasty:
This is a technique used to widen a narrowed or obstructed artery. In the procedure, a collapsed metal tube (stent) surrounding a collapsed balloon is passed into the vascular system using a guide wire (catheter). The balloon is positioned in the narrowed area and then inflated. This squashes the deposits causing the narrowing and expands the stent, thus opening the blood vessel. The balloon is then deflated and removed, while the stent remains to keep the artery open, thus allowing the return of normal blood flow.

17
Q

what is an ECG

A

A test that checks for electrical activity in the heart.

18
Q

what are the segments of an ECG

A

P-Q-R-S-T

19
Q

what does ST elevation mean?

A

Usually indicates someone is having a MI.

19
Q

where are the electrodes placed on a patient?

A

V1- 4th intercostal space to the right of the sternum
v2- 4th intercostal space to the left of the sternum
v3- midway between v2 and v4
v4- 5th intercostal space at the midclavicular line
v5- anterior axillary line at the same level as v4
v6- midaxillary line at the same level as v4 and v5
RL- anywhere above the right ankle and below the torso
RA- anywhere between the right shoulder and the wrist
LL- anywhere above the left ankle and below the torso
LA- anywhere between the left shoulder and the wrist

19
Q

what are the 4 different types of arrythmias (alterations of cardiac conduction)

A

atrial fibrillation: chaotic signals in the atria cause them to quiver, leading to an irregular, and often rapid, heart rate. Atrial fibrillation is the most common type of arrhythmia, particularly in the elderly

ventricle tachycardia: abnormal and rapid discharge of electrical signals in the ventricles. This manifests as a very high HR (~150-200 bpm) which decreases filling time (diastole) and therefore decreases CO and subsequently BP. This may manifest with palpitations, shortness of breath, chest pain, dizziness and loss of consciousness. If lasting for more than a few seconds, this arrhythmia becomes life threatening as CO will rapidly drop, eventually leading to cardiac arrest

ventricle fibrillation: ventricles quiver instead of contracting. There will be a loss of consciousness and breathing stops

asystole: cessation of electrical signals (also known as a ‘flatline’). Without electrical activity in the heart, there is no mechanical activity (so no pumping). This is the terminal rhythm of cardiac arrest. This is not a shockable rhythm, thus defibrillation will not work. Instead, high quality CPR must be instigated, usually with the use of adrenaline (epinephrine) to first try and establish some electrical activity.