burns Flashcards

1
Q

immediate management

A

-ABCs
-Begin CPR if needed
-Establish adequate airway (RSI or cricothyrotomy)
-Consider early ET intubation when:
-Severe burns to lower face & neck
-Full-thickness chest wall burns, especially if circumferential -> eschar formation -> restriction
-Burn size >60%, including face

-Support ventilation & oxygenation
-Oxygen 2-12 L/min via NC or face mask
-With smoke inhalation, give 100% O2 -> give 100% to all of these pts…we dont know who has smoke inhalation injuries
-Monitor pulse oximetry and CO level

-EKG
-NGT
-tetanus prophylaxis
-remove any jewelry!!!!

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2
Q

injuries associated with burns

A

-Fractures: 45-64%
-Complex soft tissue injuries: 36-52 %
-TBI: 17-26%
-Thoracic and abdominal injuries: 4-24 %
-mortality- 3-55%

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3
Q

burn center referral criteria

A

comorbidities
-do it early

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4
Q

criteria for ICU admission

A

-too unstable to be transferred
-Mechanical ventilation (inhalation injury, massive fluid resuscitation)
-Require cardiac or other hemodynamic monitoring - fluid shifts
-Have risk factors for multisystem organ failure

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5
Q

when does a pt need IV access

A

-Deep burns covering >15% of BSA
-Clinical findings suggesting shock or volume depletion
-Use 2 large-bore peripheral IV lines, preferably in nonburned skin
-Central line only if peripheral access is impossible

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6
Q

obtain labs

A

-ABG analysis
-Carboxyhemoglobin level
-CBC, lytes, urine myoglobin
-CXR and ECG
-cyanide
-tox screen

-musculoskeletal / internal injury -> CT scan

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7
Q

factors that affect rate of IV transfusion

A

-flow rate=
-length of tubing
-viscosity (blood > colloids > crystalloid)
-radius- bore
-pressure- pressure adding systems for IVs

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8
Q

nutrition

A

-all have NG tube inserted
-decompress to prevent -> ileus from severe burns -> no GI motility
-have very high caloric requirements too
-sudden hypotension -> may be gastric distention -> kink on IVC in T8

-deep burns >20% of BSA -> develop ileus -> vomit -> give NG tube
-prevents aspiration

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9
Q

fluid resuscitation!!!!!!

A

-loss of large volumes of intravascular fluid, electrolytes and proteins
-Fluid loss maximal during first 6-8 hrs
-!Fluid resuscitation: crystalloid (LR or NS) on day #1
-normal saline is avoided bc of hyperchloremic acidosis it creates

-Fluid resuscitation formulas
-Parkland (Baxter): 4ml/kg per % of BSA burned
-when did the pt get the burns?
-70kg pt, 30% burn, 6:00AM-> get to hospital at 8:00AM -> 30(70)= 2100(4) = 8400cc of fluids
-since two hours have already gone by -> do 8400/2 = 4200/6 = 700cc per hour for first 8 hours
-4200/6 = 260cc for the next 16 hrs

-Modified Brooke:
-Adult: 2ml/kg per % of BSA burned
-Child: 3ml/kg per % of BSA burned

-½ of calculated volume given in first 8 hours; the remainder is given over the next 16 hours

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10
Q

eval for any other possible injuries

A

-Especially the patient burned in MVA, explosion, or after jumping from a burning building
-Search for fractures or injuries to the hands, c-spine, chest and abdomen

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11
Q

indwelling catheter

A

-Insert indwelling urinary catheter to monitor UO
-MOST IMPORTANT MONITORING DEVICE IN A BURN PT
-IV infusion rates adjusted to maintain UO of 0.5-1.0 mL/kg/h (adults) & 1.0 mL/kg/h (peds <10kg)
-UA and urine myoglobin

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12
Q

further evaluation

A

-Obtain hx: injury mechanism, possible presence of toxic combustibles, burn take place in open or enclosed space, PMH, tetanus status, allergies
-person passed out from smoke inhalation -> palms can be burned (usually people clench the hands) -> suspect inhalation injury

-Determine severity of injury (table on next slide)
-Crucial for deciding on hospital admission or transfer to burn center
-Guides initial fluid resuscitation & establishes prognosis

-In General:
-Minor burns – usually managed as outpatients
-Moderate uncomplicated burns – hospital admission
-Major burns – transfer to burn center

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13
Q

determinants of burn severity

A

BURN SIZE
BURN DEPTH
BURN SITE
PRESENCE OF CIRCUMFERENTIAL BURNS- act like a tourniquet -> compartment syndrome, prevent breathing
INHALATION INJURY
ELECTRICAL INJURY
AGE OF PATIENT
ASSOCIATED INJURIES
MAJOR UNDERLYING MEDICAL PROBLEMS

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14
Q

summary of american burn association burn severity categorizations

A

MAJOR BURN INJURY
-SECOND-DEGREE BURN >20% BSA IN ADULTS
-SECOND-DEGREE BURN >10% BSA IN CHILDREN
-THIRD-DEGREE BURN >5% BSA
SIGNIFICANT BURNS INVOLVING HANDS, FACE, EYES, EARS, FEET, PERINEUM, MAJOR JOINTS
-PATIENTS WITH: INHALATION INJURY, ELECTRICAL INJURY, BURN INJURY COMPLICATED BY OTHER MAJOR TRAUMA
-POOR-RISK PATIENTS WITH BURNS

MODERATE UNCOMPLICATED BURN INJURY
-SECOND-DEGREE BURN OF 10-20% BSA IN ADULTS
-SECOND-DEGREE BURN OF 5-10% BSA IN CHILDREN
-THIRD-DEGREE BURN OF 2-5% BSA

MINOR BURN INJURY
-SECOND-DEGREE BURN <10% BSA IN ADULTS
-SECOND-DEGREE BURN <5% BSA IN CHILDREN
-THIRD-DEGREE BURN <2% BSA

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15
Q

classification of burns by depth of injury

A

-morphine
-fentanyl
-benzos
-for 2nd degree

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16
Q

burn size

A

-Accurate measurement of burned area in % of body surface area (BSA)
-Can be estimated by using age-adjusted chart or “rule of 9s” for adults or “rule of 5s for infants and children
-Scattered small burns can be estimated by comparing them with the size of PTS HAND -> 1% of BSA
-Extent of all burns should be recorded on a drawing (front and back views) on the pt’s chart

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17
Q

rule of nines

A

-head and neck- 9
-arm- 9 each
-torso front- 18
-torso back- 18
-leg- 18 each
-genitalia and perineum- 1

18
Q

burn depth

A

-First-degree –
-only epidermal layer
-red, painful, dry, and tender without blisters
-usually heals in about 7 days w/o scarring
-sunburn

-Second-degree –
-extends into dermis
-further classified into superficial or deep partial-thickness
-Superficial partial-thickness – blisters and very painful; exposed dermis is red and moist, with intact capillary refill; healing occurs in 2-3 weeks with little to no scarring
-Deep partial-thickness – white to yellow in color; pressure applied to skin is felt but 2-point discrimination is often diminished; capillary refill and pain sensation are absent; healing occurs in 3 weeks - 3 months and scarring is common

-Third-degree –
-aka full-thickness burns
-charred, pale, leathery, and painless
-do not heal spontaneously because all dermal elements are destroyed
-surgical repair and skin grafting are needed

-Fourth-degree – extend through SC fat, muscle and bone
-amputation or extensive reconstruction is required

19
Q

antibiotics

A

-always assume worst burn if you cant distinguish
-clean wounds, escharotomy, remove clothing
-anti-infection agents on the skin- bacitracin etc.
-IV antibiotics shouldnt be used unless active infection

20
Q

blisters

A

-do not pop them
-if they do pop do it in a sterile manner -> do not deroof -> acts as a physiologic bandage
-infection risk
-2nd degree

21
Q

major burns

A

-Burns areas considered as major injuries:
-Hands and feet – deep burns can result in scarring leaving the pt with permanent disability
-Face – sever scarring can result in profound physical and emotional impact; often associated with inhalation injuries and compromised airway
-Eyes – corneal scarring and eyelid dysfunction may ultimately lead to blindness
-massive periorbitial edema -> examine eyes before they swell short
-Ears – deep burns predispose to the development of pressure deformity and infection
-Perineum – more susceptible to infection

22
Q

circumferential burns

A

-major burn
-Neck - may cause lymphatic & venous obstruction that leads to laryngeal edema & airway obstruction
-Extremities – compartment syndrome
-Chest – respiratory failure

23
Q

inhalation injury

A

-Burned sustained in confined space
-singed nasal hairs
-facial burns
-soot in nose or mouth
-hoarseness
-carbonaceous sputum
-wheezing
-CO level >10%

24
Q

electrical injury

A

-Damage may be extensive, even if outward signs are minimal
-arrhythmias - EF drops
-renal failure may occur
-transient -> pts can recover on their own well

25
Q

risks for major burn

A

-Age of pt
-Mortality rates increase with very young (<5 yo) or very old (>55 yo) patients

-Associated injuries
-Burns occurring with other injuries such as fractures or internal injuries due to MVAs, falls or explosions

-Major underlying medical problems
-Morbidity & mortality increased in patients with hx of MI, angina, significant pulmonary disease, DM, renal failure, alcohol or substance abuse

26
Q

pain control

A

-Minor burns, especially of the extremities, can be relieved by immersing in cool water
-Oral, subcutaneous, or intramuscular narcotics may be used for outpatient therapy
-IV Morphine, Meperidine or Hydromorphone should be used to control pain in patients with moderate or major burns
-benzos
-fentanyl
-Be careful with the patient with compromised respiration!!!
-cool them down -> but not too much -> hypothermia -> coagulation

27
Q

tx and wound care (minor burns)

A

-Treated on an outpatient basis
-Clean with mild soap & water or a dilute antiseptic solution
-Large blisters >2 cm or involving mobile joints should be drained or debrided
-Small blisters on nonmobile areas should be left intact

-Topical antibiotics –
-1% silver sulfadiazine cream (Silvadene) -> not for face or sulfa allergies
-bacitracin, neomycin, polymixin B
-Analgesics - NSAIDs -> careful if they require surgery

28
Q

tx and wound care (moderate and major burns)

A

-Gently remove clothing, dirt, & other debris adhering to burn
-irrigation with sterile saline may help
-Don’t scrub wounds or use harsh detergents
-Little or no debridement of moderate or major burns should be performed in the ER -> sterile burn unit
-Redundant skin from ruptured blisters of minor superficial partial thickness burns may be removed -> dont want to remove viable skin
-Avoid ointments & complex dressings on pts being transferred -> use simple nonadherent dressing -> vasaline and pregmented gauze
-Pain control- don’t use systemic antibiotics

29
Q

pts transferred to burn center

A

-When pt with serious burn is first evaluated -> closest burn center should be contacted immediately -> they give recommendations and plans -> transfer
-With quick transfer, early escharotomy may be performed and can prevent complications of circumferential burns
-Fluid resuscitation and all other supportive measures should be continued during transport
-Keep patient warm during transfer- they will lose heat

30
Q

escharotomy

A

-surgical incision(s) used to treat full-thickness circumferential burns
-Indications for emergent escharotomy:
-Vascular compromise of extremities or digits -> ischemia bowel
-Suspected abdominal compartment syndrome- urine output decrease
-measure bladder pressure -> >40 dx, >20 suspect
-Impending or established respiratory failure
-decreases pressure -> increase perfusion -> gapping is seen

-Contraindictaions:
-Pts with irreversible gangrene of the extremity or digit
-too much of a bleed risk

-Complications
-Excessive blood loss - blood = tissue
-Inadvertent fasciotomy into underlying viable tissus

31
Q

smoke inhalation

A

-Mechanisms of injury (3 categories):
-1. Thermal injury of the airways
-2. Chemical injury of airways & lung parenchyma
-3. Systemic chemical poisoning
-Many pts have a combination of above

32
Q

smoke inhalation: thermal injury

A

-Pts who have been trapped! in a fire in a confined space inhale superheated gases & may also have direct flame injury of the !face & neck!
-Exposure to water or steam in heated gas mixture may produce thermal damage in upper and lower airways
-Clinical Findings:
-Should be suspected in any pt in a fire within a confined space
-Pts with obvious face or neck burns
-Nasal soot!, soot-tinged sputum, burned nasal & facial hairs
-Dyspnea, stridor, drooling or dysphonia
-Dx confirmed by laryngoscopy or bronchoscopy!

33
Q

smoke inhalation: chemical injury

A

-Caused by noxious products of combustion of flammable materials; directly toxic to airways & lung parenchyma
-ex. burning of plastics
-materials include: Acrolein, Hydrochloric acid, Toluene diisocyanate, Nitrogen dioxide

-Clinical findings:
-Very difficult to dx in ER
-Direct laryngoscopy or flexible fiberoptic bronchoscopy may reveal mucosal friability & airway edema
-CXR is usually normal initially -> ARDS may develop hours after exposure
-CO created due to incomplete combustion

-above the vocal cords edema
-bc of affective exchange that occurs there
-edematous vocal cords
-may need criccoidthyrotomy or trach for ventilation

34
Q

sequelae of smoke inhalation: systemic chemical poisoning

A

-MC complication of smoke inhalation is CO poisoning
-CO binds to Hgb with an affinity that is 260x greater than O2, forms carboxyhemoglobin
-even small amounts of carboxyhemoglobin drastically alters
-Bound CO is not easily displaced from Hgb -> shifts oxyhemoglobin dissociation curve to left -> more difficult for Hgb to release O2 to tissues
-Net result is tissue hypoxia & lactic acidosis
-cyanide poisoning
-CO also binds to the cytochromes, interfering with intracellular energy production
-In high concentrations, CO is also bound to myoglobin & rhabdomyolysis, myoglobinuria, and acute renal failure may occur

-Clinical findings:
-Should be expected in every fire victim
-Cherry-red skin color is not a frequent or reliable findings
-ABGs and Pulse oximetry is not reliable
-New Carboxyhemoglobin oximetry is available
-Confirmed by measuring serum carboxyhemoglobin level
-MI or arrhythmias may occur!

-Carboxyhemoglobin levels:
-Non-smokers- about 1%
-Smokers- 4-6%
-Levels >10% indicate significant exposure)
-Pts may be asymptomatic with levels 10-15%
-Levels > 50-60% have a high incidence of coma & seizures
-Levels > 70% are usually fatal

35
Q

smoke inhalation: ED clinical workup, tx, and disposition

A

-ABCs apply to ALL pts
-Facial & neck burns treat as previously discussed
-Low threshold for early intubation (RSI)
-While waiting for ABG & carboxyhemobglobin levels
-give 100% oxygen via a tight-fitting reservoir mask or ET tube
-Obtain ECG, continuous cardiac monitoring, CXR, & UA
-Inhaled and parenteral bronchodilators to pts with clinical evidence of bronchospasm
-Supportive care & admit pt for at least 24 hrs
-hyperbaric chamber
-acetylcysteine or heparin -> aerosolized and put in lungs -> bloody casts in the lung -> break it up -> allow ventilation

36
Q

cyanide toxicity

A

-burning of furniture or plastics

-Initiate treatment in:
-Severe burn victims with……
-Unexplained lactic acidosis or……
-low or declining end tidal volume CO2level

-If parameters are unavailable treat (you dont have CO2)….
-Any pt with depressed LOC
-Cardiac arrest
-Cardiac decompensation.

-In severe burn victims, use hydroxocobalamin

37
Q

in inhalation injury…primary attention to:

A

-supplemental O2
-airway protection strategies
-100% O2

38
Q

chemical burns

A

-Usually result from skin exposure to strong acids or alkalis (more dangerous bc lasts longer)
-Full development is slower than thermal burns
-initial size is usually underestimated during initial evaluation -> grows
-Clinical features depend on type of agent, concentration, volume, & duration of exposure
-Most acids produce coagulation necrosis due to desiccating action on superficial tissue proteins; superficial and full-thickness burns may result

-Hydrofluoric (HF) acid rapidly penetrates intact skin and can cause progressive pain & deep tissue destruction
-onset of pain may be delayed up to 12 hrs postexposure -> pt c/o pain with the absence of any presentation of burn!
-As progresses, skin may turn -> blue-gray appearance with surrounding erythema

-Alkalis cause liquefaction necrosis, which allows hydroxyl ions to penetrate into deep tissues
-Soft, gelatinous, friable, brownish eschars are produced
-Airbags deploy by ignition of a solid propellant (sodium azide) creating an exothermic reaction and certain corrosive byproducts (sodium hydroxide, nitric oxide, and ammonia) -> alkaline type burn
-Airbag! deployment may cause a combination of friction, thermal, and chemical burns

39
Q

chemical eye burns

A

-true ocular emergencies causing redness, pain, and tearing
-rinse rinse rinse
-call ophthalmologist
-injected all the way up to the limbus
-Acid ocular burns quickly precipitate proteins in the superficial eye structures -> “ground glass” appearance
-Alkali ocular burns cause deeper penetration secondary to liquefaction necrosis -> severe chemosis, blanched conjunctiva, & opacified cornea obscuring the view of the iris and lens can occur
-dilute acid quickly
-alkaline burns need significant dilution over a period of time

40
Q

chemical burns ED care and disposition

A

-First priority - terminate burning process; remove garments & copious skin irrigation
-Remove dry chemical particles manually before irrigation
-Elemental metals (Na+, lithium, Ca+, and Mg+) can ignite spontaneously when exposed to air
-water shouldn’t be used to extinguish burning particles because of the explosive exothermic reaction that results

-oxalic, hydrofluoric acid- hypocalcemia, hypomagnesium, hyperkalemia, arrythmia, death
-tannic, formic, chromic- hepatic necrosis nephrotoxicity,
-phenol, cresol- hemoglobin, massive hemolysis, multiple organ failure

-These metal particles should be covered with mineral oil or extinguished with a class D! fire extinguisher
-Systemic toxicity can result from topical exposure of certain chemical agents

41
Q

burn tx

A

-In ocular exposure to alkalis or acids, eye irrigation with 2 L NS via a Morgan lens (IV) for a min of 1 hr is needed
-With alkali or acid exposure, return of the pH to neutral -> endpoint for irrigation
-Visual acuity, check & pH testing should follow -> not precede irrigation
-HF acid burns often require calcium gluconate to bind fluoride and neutralize its toxic effects; applied topically, subcutaneous, intradermal injection
-Treat systemic toxicity similar to thermal burns (pain control, IVF, tetanus)

42
Q

long term complications of burn

A

-Depression
-PTSD
-Contractures
-Sensory Loss
-**Skin Cancer is NOT a risk

-BSA + age = if > 100 -> minimal chance of survivorship -> palliative care
-BSA% + age + 17 (for multiple organ failure) = >140-150 -> negligible chance of survivorship
-