brain and behaviour Flashcards

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1
Q

theory 1: stages of memory

A

registration: input from our senses into the memory system
encoding: processing and combining of received information
storage: holding of that input in the memory system
retrieval: recovering stored information from the memory system

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2
Q

theory II: duration

A

Conceptual divisions in memory systems • Sensory • Working or short term memory • Long-term memory

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3
Q

a model of memory

A

look at slides

  • sensory registers
  • working memory
  • long term memory
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4
Q

theory III: types

A

see diagram
long term memory
-declarative (episodic, semantic)
-non-declarative (procedural, priming, conditioning, non-associative learning)

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5
Q

define episodic memeory

A

nvolves the medial temporal lobes including the hippocampus, entorhinal cortex, mammilary bodies, and parahippocampal cortex

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6
Q

memory systems:

  • sematic
  • procedural
  • working
A

Memory systems: Semantic - Knowledge Procedural – how to do things Working – short term

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7
Q

memory disorders

A

• Total amnesia is rare, especially isolated amnesia with otherwise preserved cognition • Numerous neurological conditions can affect memory with varying lesion sites • Varying aspects of memory - e.g. episodic, semantic, anterograde, retrograde etc. – are affected in different ways by different disorders • Implicit memory or learning often intact in memory disorders

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8
Q

Theory V: modality

A
  • Left hemisphere: Mainly concerned with verbal information processing
  • Right hemisphere: Mainly concerned with non-verbal information
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9
Q

HOW DO WE REMEMBER? SERIAL POSITION EFFECT

A

-we remember the first and last words on the list due to the primary effect and the recency effect

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10
Q

The probability of recalling a word is related to

A

Order in the list Personal salience of words Number of words Chunking or other encoding strategy Delay time Distractio

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11
Q

how does the way we remember have clinical implications?

A

Give important information at beginning and end of consultation •Emphasise and repeat important information •Make salient to the person •Chunk information into meaningful categories •Avoid overloading with information

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12
Q

define phoneme:

A

Phoneme: the smallest unit of speech sound in a language that can signal a difference in meaning

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13
Q

define morphemes:

A

Morphemes: the smallest units of meaning in a language – Typically consist of one syllable – Morphemes are combined into words

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14
Q

syntax

A

• Rules and principles which govern the way in which morphemes and words can be combined to communicate meaning in a particular language • Theory of ‘universal grammar’ widely accepted: that under normal conditions human beings will develop language with particular properties (e.g. distinguishing nouns from verbs) • Pinker (1984) - children of speakers of pidgin languages which lack basic grammatical structures develop languages which are fully grammatical • An innate property of the brain that results in this

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15
Q

critical period in language acquisition

A

draw graph

down after 5ish

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16
Q

language acquisition

A

Exposure to other people using language required • Between ages 5 to puberty language acquisition becomes more difficult, based on: – Feral children – Brain injury at different ages – Second language acquisition

17
Q

Broca’s (expressive) Aphasia

A

Characteristics: • Non-fluent speech • Impaired repetition • Poor ability to produce syntactically correct sentences • Intact comprehension

18
Q

Wernicke’s (receptive) Aphasia

A

• Problems in comprehending speech (input or reception of language) • Fluent meaningless speech • Paraphasias – errors in producing specific words • Semantic paraphasias – substituting words similar in meaning (“barn” –“house”) • Phonemic paraphasias – substituting words similar in sound (“house” –“mouse”) • Neologisms – non words (“galump”) • Poor repetition • Impairment in writing

19
Q

Conditions Associated with Aphasia

A

Lesions to the dominant hemisphere can be caused by: – Stroke – Traumatic brain injury – Cerebral tumour – Progressive neurodegenerative conditions

20
Q

dysexecutive syndrome

A

Dysexecutive syndrome involves the disruption of executive function and is closely related to frontal lobe damage

21
Q

executive functioning

A

Executive functioning skills are the mental processes that enable us to plan, focus attention, remember instructions, and juggle multiple tasks successfully. • Dysexecutive syndrome encompasses cognitive, emotional, and behavioural symptoms • Dysexecutive syndrome can result from many causes, including head trauma, tumours, degenerative diseases, and cerebrovascular disease, as well as in several psychiatric conditions

22
Q

The Dysexecutive Syndrome: Behavioural & Emotional Aspects

A
  • Lack of drive • Apathetic • Poor initiation of tasks • Emotional bluntness
  • Theory of mind difficulties • Reduced empathy
  • Hyperactivity • Impulsive • Disinhibited • Perseverative • Emotional dysregulation
  • Socially inappropriate • Rude, crass, prone to swearing
23
Q

the dysexecutive syndrome

-cognitive aspects

A

• Attentional and working memory difficulties • Poor planning & organisation • Difficulty coping with novel situations and unstructured tasks • Difficulty switching from task to task • Difficulty keeping track of multiple tasks • Difficulty with complex/abstract thinkin

24
Q

Deficits associated with specific regions of the frontal lobes

A

– Orbito-frontal • Impulsivity, disinhibition

– Medial • Loss of spontaneity, initiation (akinetic mutism)

– Lateral • Inability to formulate and carry out plans