BOOK QUESTIONS Flashcards
what does the dental pulp form as a defensive response?
Tertiary dentin
what cell in an antigen recognition cell in the dental pulp
odontoblasts
what is the most prominent antigen presenting cell in the dental pulp?
dendritic cell
what type of collagen most prominent in the dental pulp?
type I
What is NOT a type of pulp stone:
free, attached, embedded, or floating?
FLOATING
what are free pulp stones?
surrounded by pulp
what are attached stones?
continuous with the dentin
what are embedded pulp stones?
surrounded entirely by DENTIN and mostly of the TERTIARY type
Describe a pulp cap?
capping of exposed vital pulp tissue by placing a layer of MTA
this will stimulate reparative dentin and maintain pulp vitality
what is the effect of blood flow to the pulp when anesthtics with vasoconstriction are sued during restorative procedures?
blood flow is reduced to less than HALF of its normal rate.
what is dentin blushing?
vascular injury (hemorrhage) of pulp tissue often during crown preps. Thought to be due to the frictional heat
why are deeper carious lesions more injurious to the dental pulp?
increased dentin permeability in deeper areas, and greater cellular injury to the odontoblasts.
what depth of dentin shield is often sufficent to sheild pulp
at least 1 mm
why does a blast of compressed air directed at freshly exposed dentin create a sensation of pain?
causes a rapid outward movement of fluid in patent dentinal tubules and this stimulates nociceptors in the dentin pulp
what is the primary reason for the placement of a liner between biocompatible restorative materials and the dentin?
eliminate microleakage
what is the most significant determinant of the success of vital pulp therapy?
Pulp status before the procedure
what do you place at each visit with step wise evacuation of caries?
glas ionomer base
what is apexogenesis?
continued physiologic root formation… is a vital pulp therapy procedure performed to encourage continued physiologic development and formation of the root end.
What is Apexification?
induction of a calcific barrier across and open apex
you remove the necrotic pulp, depbride the canal, and place an antimicrobial medicament.
why is there greater dentin permeability near the pulp?
higher density of the dentinal tubules AND larger diameter
bacterial invasion of the dentinal tubules occurs more rapidly in which teeth?
non vital teeth
what is anachoresis?
microorganism transport from blood vessels into damaged tissue
PEOPLE are trying to research this topic to see if traumatized teeth with intact crowns are affected this way…
T/F
Root canals can become infected through anachoresis?
false
…research says the MAIN pathway of pulpal infection is from dentinal exposure due to enamel cracks
Which is NOT a category of Intraradiuclar infections?
tertiary?
Intraradiuclar are primary, secondary, or persistant
most common microorganisms in PRIMARY endo infections are?
Gram negative bacteria!
what microorganisms is commonly present in large percentages of root canal treated teeth that present with persistent apical periodontitis, indicaitve of failed treatment?
Enterococous Faecalis
It’s faculative anerobic gram pos coccus that has been frequently found in root canal treated teeth.
more restrictive group compared to primary infection
describe gram positive bacteria and endo
they have a higher occurence in post instrumentation samples, MORE resitant to antimicrobial treatmetns, adapt ot harsh environmental conditions.
GRAM NEG bacteria are usually eliminated by ENDO treatment!
t/f
A direct pulp exposure of a carious lesion is necessary to have a pulpal response and inflammation?
FALSE!
cracks in the enamel can attract inflammatory cells to the pulp
what is necessary for pulp and periradicular pathosis to develop?
presence of bacteria
T/F
transient changes, such as aspiration of the odontoblasts into the dentinal tubules, are usually reversible in healthy pulps?
TRUE
what is NOT a hard tissue change from pulpal inflammation?
NOT thickening of periodontal ligamnet
but yes to calcification of pulp tissue space, resoprtion of opulp tissue spaces, formation of pulp stones
T/F
Acute apical abscess is always assoicated with necrotic pulp?
true
what is the risk of endo and bisphosphanate therapy
greater risk of osteonecrosis if IV bisphosphonate therapy. dont’ damage soft tissue if endo treatment
is selective anesthesia of individual teeth useful in the mandible?
no, marginally more effective in the maxilla
A narrow vertical probing defect associated with a tooth exhibiting pulp necrosis but no or mild periodontal disease is most likely a:
draining sinus tract