Blackouts Flashcards

1
Q

How can you differentiate between a fall an a collapse?

A

Ask the patient if they remember falling and if they remember hitting the ground. If yes, it is a fall. If no, it is a collapse.

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2
Q

What features suggest syncope?

A

Triggering factor, distinct prodrome, ‘convulsion’ after LoC and less than 15 seconds, short duration, quick recovery, ongoing lethargy but no confusion.

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3
Q

What features suggest seizures?

A

Aura, automatisms, convulsions at onset of LoC, longer duration, post-ictal phase, tongue biting, incontinence.

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4
Q

What are the four big causes of blackouts and hoe do they typically present?

A
  1. Syncope - light headedness, nausea, tunnel vision
  2. Seizure - fit, tongue/urine, post-ictal
  3. Stroke/TIA - unilateral weakness/sensory loss, speech, face
  4. Cardiogenic - palpitations
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5
Q

What is the pathophysiology of vasovagal syncope?

A

Reflex bradycardia and peripheral vasodilation provoked by emotion, pain, or standing too long.

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6
Q

What symptoms typically precede vasovagal syncope?

A

Nausea, pallor, sweating, narrowing of visual fields.

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7
Q

How long does vasovagal syncope last and how quickly do people recover?

A
  1. <2 minutes

2. Rapid recovery

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8
Q

What is this a presentation of?

Vasovagal syncope symptoms but brought on by exercise (cardiac), or during/after urination (mostly in men, at night).

A

Situational syncope - cough/effort/micturition

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9
Q

What is this describing?
Hypersensitive baroreceptors cause excessive reflex bradycardia +/- vasodilation on minimal stimulation (e.g. head turning/shaving).

A

Carotid sinus syncope

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10
Q

What is this a presentation of?
Attacks when asleep/lying down, aura, identifiable triggers, altered breathing, cyanosis, tonic-clonic movements, incontinence of urine, tongue-biting, prolonged post-ictal drowsiness, confusion, amnesia.

A

Epilepsy

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11
Q

What is this a presentation of?

Unilateral weakness, sensory loss, slurred speech, facial asymmetry.

A

Stroke/TIA

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12
Q

What is this a presentation of?
Patient falls to the ground often with no warning except for palpitations, injuries common, pale, slow/absent pulse, recovery in seconds.

A

Stokes-Adams attack

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13
Q

What is the cause of a Stokes-Adams attack?

A

Transient arrhythmias cause reduction in CO and LoC.

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14
Q

What is this a presentation of?

Tremor, hunger, perspiration, rare in non-diabetics.

A

Hypoglycaemia

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15
Q

What are the causes of hypoglycaemia?

A

Poorly controlled diabetes, alcohol, liver failure, adrenal insufficiency, insulinoma.

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16
Q

What is this a presentation of?
Unsteadiness or LoC on standing from lying in those with inadequate vasomotor reflexes, elderly, autonomic neuropathy, antihypertensive medications, overdiuresis.

A

Orthostatic hypotension

17
Q

What is this a presentation of?

Hyperventilation, tremor, sweating, tachycardia, paraesthesia, light-headedness, no LoC.

A

Anxiety attack

18
Q

What is this a presentation of?

Sudden fall to the ground without LoC, mostly benign and due to leg weakness.

A

Drop attack

19
Q

What is this a presentation of?

Gradual onset, prolonged, abrupt termination, closed eyes, resistance to eye opening, dyssynchronous blackout.

A

Factitious blackout (Munchausen’s)

20
Q

What is it important to ask someone who has just had a blackout?

A
  1. Lose awareness? Injury themselves? Stiff/floppy? Movements?
  2. Incontinence? Complexion change? Tongue biting? Other symptoms?
  3. How long? When did it start? More/less frequent? Anyone in family get them? History of birth injury/CNS infections?
21
Q

What should you ask about before a blackout?

A
  1. Any warning? Aura/pre-syncope?
  2. In what circumstances do attacks occur?
  3. Can the patient prevent them?
22
Q

What should you ask about after a blackout?

A
  1. How much does the patient remember?
  2. Muscle ache?
  3. Confusion/sleepiness?
  4. Injuries sustained?
23
Q

What are the typical initial syncope investigations?

A
  1. Cardiac assessment - ECG
  2. Metabolic assessment - calcium, U&Es, magnesium, glucose
  3. ABG, low PaO2 suggests hyperventilation