Biology - 2.2 Transportation Animals Flashcards
Atrial Systole
Both atria contract pushing blood to ventricles
Cuspid valves open
Ventricles in diastole
Vena cava and pulmonary vein sealed off
Semi lunar valves are closed
Ventricular Systole
Atria in diastole
Ventricles contract pushing blood into arteries
Cuspid valves shut
Semi lunar valves open allowing blood to leave
Diastole
Both atria and ventricles relaxed
Blood flows into heart via vena cava and pulmonary vein
Semi lunar valves shut
Heart refills with blood
A on pressure diagram
Atrioventricular valves closing, Ventricular pressure increases to more than atrial pressure
Ventricles contracting
B on pressure diagram
Semilunar valves are opened
Aortic pressure increases but slightly lower than ventricular
C on pressure diagram
Semilunar valves closing
Ventricular pressure much lower than aorta pressure
D on pressure diagram
Atrioventricular valves open
Aortic pressure slightly more than ventricular pressure
Lub
Closure of cuspid valves - ventricular systole
Dub
Closure of semilunar valves - atrial systole
Events at QRS complex
Atrioventricular node transmits electrical impulses through bundles of HIS and Purkinje fibres
Ventricles depolarise
Ventricular systole
Events at P wave
Sinoatrial node generates electrical impulse
Impulse causes atria to depolarise
Atria contracts (atrial systole)
Events at T wave
Sinoatrial node generates electrical impulse
Impulse causes ventricles to repolarise
Ventricular diastole takes place
How would distance between P waves would differ if a person takes exercise?
Lengths of P wave would be shorter
Less time between ventricular and atrial systole
Effect of first degree heart block on functioning heart
Slower heart beat
How many molecules of 02 in oxyhemoglobin?
4
Why is flat top advantageous in association curve?
If partial pressure is reduced most haemoglobin would still be highly saturated with oxygen
Why is steepness of association curve advantageous?
A small increase in oxygen tension leads to a large increase in saturation
Myoglobin
Can bind to one molecule of o2, delays anaerobic respiration, takes low pressure for myoglobin to let go
Bohr Shift
Shift to the right
Higher CO2 production
More shift = more oxygen is released to be used in aerobic respiration
What happens when respiration rates are high?
More CO2 produced by cells
More CO2 diffuses into red blood cells
More carbonic acid formed
More H+ ions formed = more disassociation at higher partial pressure
Chloride Shift
- Co2 diffuses into red blood cell
- Co2+H2O+carbonic anhydrase = carbonic acid
- Carbonic acid dissociated into H+ and HCO3- ions
- CL- ions facilitated diffuse into RBC to maintain electrochemical neutrality
- H+ binds to oxhaemoglobin reducing its affinity to oxygen
- O2 released and diffuses into plasma
Structure of artery
Tough collagen outer coat to prevent overstretching
Small lumen to prevent friction
Thick layer of smooth muscle that recoils to maintain blood pressure
Structure of vein
Larger lumen as blood is under lower pressure
Semilunar valves to prevent back flow of blood