Benign skin lesions (dermatofibroma, seborrhoeic keratoses, naevi) Flashcards

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1
Q

What are the three types of naevi?

Which one is flat?

A
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2
Q

Which size moles are at greater risk of malignant change?

A

larger ones

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3
Q

Is it reasonable to excise benign moles prone to mechanical irritation?

A

Yes

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4
Q

What is this?

A

Seborrhoeic wart/keratosis aka basal cell papillomas

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5
Q

Describe the development of seborrhoeic warts.

A

Light brown raised lesions –> gradually grow and darken –> surface gets rougher.

They can vary in shape and size, from one centimeter to several in diameter.

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6
Q

What is this?

A

Dermatofibroma

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7
Q

When can seborrhoeic warts resemble squamous cell carcinomas?

A

When they get inflamed and crusty

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8
Q

What is the management of seborrhoeic keratosis?

A

Curettage
OR liquid nitrogen therapy

Should be done if there is diagnostic uncertainty or probelms e.g. itchy

They are relatively easy to remove.

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9
Q

Define dermatofibroma.

A

AKA cutaneous fibrous histocytoma

Common benign fibrous nodule usually found on the skin of the lower legs,

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10
Q

What causes dermatofibroma?

A
  • May be a reactive process or a true neoplasm e.g. after insect bites, injections, rose thorn injuries
  • More common in those with altered immunity e.g. HIV, immunosuppression, autoimmune conditions
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11
Q

What are the clinical features of dermatofibroma?

A
  • Solitary firm papule or nodule
  • Usually on lower legs
  • 0.5-1.5cm in diameter
  • Tethered to the skin surface and mobile over subcutaneous tissue
  • “Dimple sign”
  • Colour ma be pink to light brown in light skin, dark brown to black in dark skin

NB: usually asymptomatic

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12
Q

What are the complications of dermatofibroma?

A

May be traumatised by razor
Multiple eruptions may occur especially in immunosuppression

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13
Q

How is a dermatofibroma diagnosed?

A

Clincial diagnosis
Dermoscopy - central white area surrounded by a faint pigment network

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14
Q

What is the management of dermatofibroma?

A

Usually reassurance is enough and other treatments often cause recurrence and are rarely successful e.g. cryotherapy, shave biopsy, laser.

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15
Q

Define seborrhoeic keratoses.

A

A harmless warty spot that appears during adult life as a common sign of skin ageing. Some people have hundreds of them.

AKA basal cell papilloma, senile/brown/wisdom wart, barnacle.

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16
Q

What are the features shown here?

A
  • Flat or raised papule or plaque
  • 1 mm to several cm in diameter
  • Skin coloured, yellow, grey, light brown, dark brown, black or mixed colours
  • Smooth, waxy or warty surface
  • Solitary or grouped in certain areas, such as within the scalp, under the breasts, over the spine or in the groin

Stuck on the surface like barnacles. Do not occur on palms or soles.

17
Q

Why is seborrhoeic keratoses a misleading term?

A

The name is misleading, because they are not limited to a seborrhoeic distribution (scalp, mid-face, chest, upper back).

Nor are they formed from sebaceous glands, as is the case with sebaceous hyperplasia

Nor are they associated with sebum — which is greasy.

18
Q

What are the risk factors for seborrhoeic keratoses?

A
  • > 60yrs - 90% of adults have one or more, starting in 30-40s.
  • Sunburn or dermatitis
  • Skin friction
  • Solar lentigo may give rise to them
19
Q

What are the complications of seborrhoeic keratoses?

A

Skin cancers may be difficult to tell apart from SKs

Very rarely eruption of SKs may indicate a gastric adenocarcinoma - Leser-Trelat sign.

NB: SKs are not premalignant tumours.

20
Q

How do you diagnose seborrhoeic keratoses?

A

Clinical diagnosis - stuck on appearance with other similar lesions

21
Q

What is the management of seborrhoeic keratoses?

A

Not harmful but may be removed if desired as may be unsightly, itchy or catches on clothing.

  • Cryotherapy
  • Curretage +/- electrocautery
  • Ablative laser
  • Shave biopsy
  • Focal chemical peel with trichloracetic acid
22
Q

What is the prognosis with seborrhoeic keratoses?

A

Persist - sometimes remit spontaneously or via lichenoid keratosis mechanism.