Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

CVS > Arrhythmias > Flashcards

Arrhythmias Flashcards

1
Q

How to determine the rate of an ECG?

A

count the large boxes between each R wave and divide 300 by that number
300, 150, 100, 75, 60

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

is the rhythm regular or irregular for tachycardia and fibrillation ?

A

tachycardia - regular

fibrillation - irregular

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What presentation may someone have who has an arrhythmia/supraventricular tachycardia?

A
  • asymptomatic
  • palpitations, dyspnoea, chest pain, fatigue
  • embolism
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

investigations would you do?

A

ECG - 12 leads
Heart monitoring for 24 hours
Blood test (especially thyroid function)
Echocardiogram

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

First-Degree Heart Block

A

In first-degree heart block, the heart’s electrical signals are slowed as they move from the atria to the ventricles (the heart’s upper and lower chambers, respectively) through the AV node.
This results in a longer, flatter line between the P and the R waves on the ECG (electrocardiogram).
First-degree heart block usually doesn’t cause any symptoms or require treatment.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Second-degree Heart Block

A

In this type of heart block, electrical signals between the atria and ventricles are slowed to a large degree. Some signals don’t reach the ventricles. On an ECG, the pattern of QRS waves doesn’t follow each P wave as it normally would.
There is increasing delays in the time it takes the AV node to send the pulse to the ventricle. This will eventually lead to a heartbeat being skipped.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What are the two types of second degree heart block called?

A

Mobitz type I and Mobitz type II

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Mobitz type I

A

In this type (also known as Wenckebach’s block), the electrical signals are delayed more and more with each heartbeat, until the heart skips a beat.
On the ECG, the delay is shown as a line (called the PR interval) between the P and QRS waves. The line gets longer and longer until the QRS waves don’t follow the next P wave.
This is the less serious type. It occasionally causes mild dizziness and doesn’t usually require treatment.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Mobitz type II

A

Some of the electrical signals don’t reach the ventricles. However, the pattern is less regular than it is in Mobitz type I.
Some signals move between the atria and ventricles normally, while others are blocked.
This is the more serious type. It can cause light-headedness, dizziness and fainting and usually requires treatment.
On an ECG, the QRS wave follows the P wave at a normal speed. Sometimes, though, the QRS wave is missing (when a signal is blocked).
Less common than type I, but it’s usually more severe. Some people who have type II need medical devices called pacemakers to maintain their heart rates.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Third-Degree Heart Block

A

None of the electrical signals reach the ventricles through the AV node. This type also is called complete heart block or complete AV block.
When complete heart block occurs, special areas in the ventricles may create electrical signals to cause the ventricles to contract. This natural backup system is slower than the normal heart rate and isn’t coordinated with the contraction of the atria. On an ECG, the normal pattern is disrupted. The Pwaves occur at a faster rate, and it isn’t coordinated with the QRS waves.
This type of heart block can be a medical emergency, although in many cases it’s mild and doesn’t require treatment.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is Supraventricular tachycardia (SVT)

A

abnormally fast heart rate of over 100 heartbeats a minute.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What happens during SVT?

A

the heart rate is not being controlled by the sinus atrial node. another part of the heart is over ridding this and causing the heart to beat faster

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Describe Wolff-Parkinson-White Syndrome

A

There is an extra electrical pathway between your heart’s upper chambers (atria) and lower chambers (ventricles) which causes a rapid heartbeat (tachycardia).
Treatment for Wolff-Parkinson-White syndrome can stop or prevent episodes of fast heartbeats. A catheter-based procedure, known as ablation, can permanently correct the heart rhythm problems.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Indications for temporary pace maker

A
  • intermittent or sustained symptomatic bradycardia,
  • syncope
  • prophylactic when patient at high risk for development of severe bradycardia eg 2nd or 3rd degree AV block, post anterior MI, even when asymptomatic
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Indications for permanent pace maker

A
  • symptomatic or profound 2nd/3rd degree AV block
  • Mobitz type II 2nd/3rd degree AV block even if asymptomatic
  • AV block associated with neuromuscular diseases
  • after (or in preparation for) AV-node ablation
  • alternating RBBB/LBBB
  • syncope
  • sinus node disease associated with symptoms
  • carotid sinus hypersensitivity/malignant vasovagal syncope
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

In the Vaughan Williams Classification,

how does 1A work?

A

Fast Moderate Na blockage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

In the Vaughan Williams Classification,

how does 1B work?

A

Intermediate Weak Na blockage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

In the Vaughan Williams Classification,

how does 1C work?

A

Slow Strong Na blockage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

In the Vaughan Williams Classification,

how does 2 work?

A

B Adrenergic receptor antagonism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

In the Vaughan Williams Classification,

how does 3 work?

A

K channel blockage

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

In the Vaughan Williams Classification,

how does 4 work?

A

Ca channel blocker

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

In the Vaughan Williams Classification,

Name a 1A drug

A

Disopyramide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

In the Vaughan Williams Classification,

Name a 1B drug

A

Phenytoin, Lidocaine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

In the Vaughan Williams Classification,

Name a 1C drug

A

Flecainide

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

In the Vaughan Williams Classification,

Name a 2 drug

A

Bisoprolol, atenolol

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

In the Vaughan Williams Classification,

Name a 3 drug

A

Amiodarone

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

In the Vaughan Williams Classification,

Name a 4 drug

A

Verapamil, diltiazem

28
Q

In the Vaughan Williams Classification,

What type of problem is 1A drug used for?

A 
¥	atrial fibrillation, 
¥	premature atrial contractions, 
¥	premature ventricular contractions, 
¥	ventricular tachycardia, 
¥	Wolff-Parkinson-White syndrome
29
Q

In the Vaughan Williams Classification,

What type of problem is 1B drug used for?

A

Ventricular tachyarrhythmias

30
Q

In the Vaughan Williams Classification,

What type of problem is 1C drug used for?

A

For serious problems
¥ Severe ventricular dysrhythmias
¥ May be used in atrial fibrillation/flutter

31
Q

In the Vaughan Williams Classification,

What type of problem is 2 drug used for?

A

¥ General myocardial depressants for both supraventricular and ventricular dysrhythmias

32
Q

In the Vaughan Williams Classification,

What type of problem is 3 drug used for?

A

¥ dysrhythmias that are difficult to treat
¥ Life-threatening ventricular tachycardia or fibrillation, atrial fibrillation or flutter—resistant to other drugs
¥ Sustained ventricular tachycardia

33
Q

In the Vaughan Williams Classification,

What type of problem is 4 drug used for?

A

¥ paroxysmal supraventricular tachycardia

¥ rate control for atrial fibrillation and flutter

34
Q

In the Vaughan Williams Classification,

Name two drugs in the other classification

A

digoxin

adenosine

35
Q

What is the pathophysiology of digoxin?

A
  • Inhibits the sodium-potassium ATPase pump so there is less ATP to maintain the resting membrane potential
  • increases the conc of Na in the cell as it cannot be pumped out of the cell
  • increases the time of depolarisation
  • phase 4 and 0 is increased
  • this leads to a decrease in heart rate
36
Q

What conditions is digoxin used for?

A

¥ CHF

¥ Atrial dysrhythmias (AF)

37
Q

What are the toxicity signs of digoxin ?

A 
¥	Nausea and vomiting
¥	Xanthopsia
¥	Bradycardia - low HR
¥	Tachycardia - high HR 
¥	Arrhythmias: VT and VF (tachycardia and fibrillation)
38
Q

What increases the risk of toxicity from digoxin?

A

low potassium levels in the blood

39
Q

How would you treat toxicity from digoxin?

A 
1. stop medication
if severe:
2. give digibind 
- digoxin immune antibody 
- binds to digoxin and is exerted in urine
40
Q

What does adenosine do pathophysiologily?

A

¥ Slows conduction through the AV node to decrease the HR

41
Q

What is adenosine used to treat?

A

¥ Used to convert paroxysmal supraventricular tachycardia to sinus rhythm

42
Q

how does Amiodarone (class three) work pathophysiology?

A

¥ Delayed repolarization of the heart by potassium channel blockage

43
Q

What is amiodarone used to treat?

A

¥ VT

¥ occasionally in supraventricular tachycardia (SVT)

44
Q

What are the side effects of amiodarone?

A 
¥	Thyroid (hypo or hyperthyroidism)
¥	Pulmonary fibrosis
¥	Slate – grey pigmentation
¥	Corneal deposits
¥	LFT abnormalities
45
Q

Does adenosine have a long or short half life?

A

very short

46
Q

What is the purpose of treating a patient who has AF with an anticoagulant?

A

Patient with AF has atria that cannot pump blood effectively around the body so thrombosis formation risk is high
Therefore they are at a greater risk of embolism. (Stroke, ischaemic limb disease, PE)
Giving them an anticoagulant will decrease this risk

47
Q

What two types of thrombosis can a patient get?

A

arterial

venous

48
Q

What are the characteristics of an arterial thrombosis ?

A

Adherence of platelets to arterial walls
White in colour
Often associated with MI, stroke and ischaemia

49
Q

What are the characteristics of a venous thrombosis?

A

Develops in areas of stagnated blood flow - where blood flow is slower (e.g. deep vein thrombosis- damaged valves in the venous circulation, left atrium - blood is not being pumped out of the atrium effectively)
Red in colour
Associated with Congestive Heart Failure, Cancer, Surgery.
Made up of fibrin

50
Q

Generally, what is warfarin used for?

A

to thin the blood

51
Q

What structure does warfarin look similar to ?

52
Q

What is the clearance time for warfarin?

53
Q

How long does it take for warfarin to begin having an effect?

A

8-12 hours

54
Q

How does warfarin work pathophysiology ?

A

it is a vitamin K antagonist

55
Q

Can warfarin cross the placenta?

56
Q

How is a warfarin overdose treated?

A

Vitamin K infusion

57
Q

What are three side effects of warfarin?

A

intraocular haemorrhage - bleeding in the eye
bleeding
chondrodysplasia - cartilage not forming properly

58
Q

What is the scoring system used to determine the risk of bleeding with a patient on warfarin?

59
Q

What does CHADS stand for?

A 
congestive heart failure 
hypertension 
age >75 
diabetes 
stroke (or TIA - transient ischaemic attack)
60
Q

Name 5 drugs that increase the activity of warfarin and describe briefly how they work

A

Aspirin, Sulphonamides - decrease binding to albumin
Cimetidine, Disulfiram - inhibit degradation
Oral antibiotics - decrease synthesis of clotting factor

61
Q

Name 5 drugs that decrease the activity of warfarin and describe briefly how they work

A
  • Induce metabolising enzymes
    Barbiturates, phenytoin
  • Promote clotting factor synthesis
    Vitamin k
  • Reduce absorption
    Cholestyramine, colestipol
62
Q

What should patients be educated on when taking warfarin?

A

alcohol intake
diet - certain foods they shouldn’t eat (broccoli)
avoid injuring them selfs

63
Q

What does INR stand for?

A

international normalised ratio

64
Q

What is the normal INR and what is the normal therapeutic INR?

A 
normal = 1 
therapeutic = 2.5-4
65
Q

Name three new drugs they act in a similar to warfarin

A
  • Dabigatran
  • Apixaban
  • Rivaroxaban
66
Q

Do these new drugs reduce the risk of stroke and non CVS embolism more than warfarin?

CVS (20 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2025 Bold Learning Solutions. Terms and Conditions