Applied anatomy of the heart Flashcards

1
Q

Where is referred cardiac pain usually felt?

A

T1-4 medial arm
Neck
Jaw
Also to epigastrium T5-T9

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2
Q

Why might somatic pain and visceral (heart) pain get confused?

A

Visceral afferents travel in the same pathway as sensory nerve fibres in the skin
Brain cannot differentiate and therefore somatic and visceral pain are confused

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3
Q

What % of the septum do the left and right coronary arteries supply?

A
Left = anterior ⅔ of coronary septum 
Right = posterior ⅓
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4
Q

Which leads give a view of the septum of the heart (LAD)?

A

V1

V2

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5
Q

Which leads give an anterior view of the heart (LAD)?

A

V3

V4

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6
Q

Which leads give a lateral view of the heart (Left circumflex or diagonal branch of LAD)?

A

V5
V6
aVL
I

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7
Q

Which leads give an inferior view of the heart (RCA or left circumflex)?

A

aVF
II
III

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8
Q

What type of cardiac hypertrophy is caused by pressure overload? What can cause pressure overload?

A

Concentric hypertrophy

Caused by hypertension and aortic stenosis, strength training

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9
Q

What type of cardiac hypertrophy is caused by volume overload? What can cause volume overload?

A

Eccentric hypertrophy
Caused by athletes, pregnancy + valve disease
Can lead to aortic or mitral regurgitation

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10
Q

Describe the effects of concentric hypertrophy on the structure and function of the heart.

A

Increase in myocyte cell width
Thick walls and small lumens
Compliance is reduced –> Can lead to eccentric as reduced compliance can cause volume overload
Ventricular filling is compromised

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11
Q

Describe the effects of eccentric hypertrophy on the structure and function of the heart.

A

Increase in myocyte cell length
Chamber dilatation
Elevated oxygen demand and lower mechanical efficacy

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12
Q

Describe how the heart remodels after injury and how this can lead to systolic and diastolic dysfunction.

A
  • New sarcomeres
  • Reduced capillary:myocyte ratio (dye to insufficient apoptosis)
    Result =
  • Increased fibrous tissue ‘stiffen’
  • Myocytes undergo apoptosis
Systolic = reduced ejection fraction 
Diastolic = increased resistance
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13
Q

What are the states of the AV and semilunar valves during S1 (systole) and S2 (diastole)?

A
S1 = AV valves close; semilunar valves open 
S2 = AV valves open; semilunar valves close
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14
Q

Mitral regurgitation:

  • In what situations does it occur?
  • What are the structural changes to the heart?
  • What is heard on auscultation?
A
  • Valve prolapse or rheumatic heart disease
  • Left atrial enlargement and left ventricular eccentric hypertrophy
  • Auscultation = Systolic murmur; between S1 and S2 –> heard at the apex
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15
Q

Aortic stenosis:

  • What are the structural changes to the heart?
  • What is heard on auscultation?
A
  • LV concentric hypertrophy –> less compliance and reduced coronary blood flow
  • Systolic murmur (during mid-systole) –> heard at right second intercostal space
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16
Q

Mitral valve stenosis:

  • What are the structural changes to the heart?
  • What is heard on auscultation?
A
  • LA generates higher pressure (pulmonary congestion)
  • Enlargement of LA - can lead to AF and loss of atrial kick
  • LV small and underfilled
  • Diastolic murmur (between S2 and S1)
17
Q

Aortic regurgitation:

  • What are the structural changes to the heart?
  • What is heard on auscultation?
A
  • Backflow of blood into the LV
  • Volume overload = eccentric hypertrophy
  • LV dilatation and congestive heart failure
  • Diastolic murmur (S2 + S1, loud to quiet)
18
Q

What is the most common abnormal heart rhythm? How is it diagnosed?

A

Atrial fibrillation
Diagnosed via ECG - absent P waves
- reduced cardiac output
- thrombi and syncope