ANS Meds Flashcards

1
Q

Uses of adrenergic medications

A

Hemodynamic compromise
Bronchospasms
Asthma
Nasal and sinus congestion

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2
Q

Adrenergic contraindications

A
Narrow angle glaucoma
Heart disease
CAD
Arrhythmias
CHF
Peripheral vascular disease
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3
Q

Andrenergic interactions

A
Drugs that stimulate the CNS
MAOI
Tricyclic antidepressants
Alcohol
Theophylline
Atropine
Antihistamines
Alpha or beta blockers
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4
Q

Adrenergic adverse reaction

A
Nervousness/tremor/anxiety
Dysrhythmias
Hypertension
Cardiac ischemia
Elevated glucose levels
Pupil dilation
Tissue necrosis
Headache, insomnia
GI and bladder involvement
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5
Q

Adrenergic target which receptors

A

Alpha 1 & 2

Beta 1 & 2

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6
Q

Adrenergic antagonist target which receptors

A

Alpha
Beta 1
Beta 2

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7
Q

Adrenergic antagonist uses

A
Treat cardiac conditions
Pupil constriction
Increase peripheral blood flow
BPH
Migraine headache
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8
Q

Atenolol classification

A

Adrenergic antagonist

Treats hypertension, AMI, arrhythmia

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9
Q

Inderal classification

A

Adrenergic antagonist

Treats migraines

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10
Q

Adrenergic antagonist contraindications

A
Ischemic heart disease
Hypotension
Asthma (extreme)
Diabetes (caution)
Some dysrhythmias
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11
Q

Adrenergic antagonist interactions

A

Any adrenergic agonist or antagonist
Insulin or oral antidiabetic
Alcohol

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12
Q

Adrenergic antagonist adverse reaction

A
Orthostatic hypotension
Bradycardia
Bronchoconstriction
Hypoglycemia
Fatigue/weakness
GI upset (increased motility)
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13
Q

Adrenergic antagonist Nx implications/Teaching

A
Do not discontinue abruptly
Monitor for orthostatic hypotension
Avoid alcohol, sedatives
Watch BP, P, RR
Evenly dose around the clock
Report GI or sexual adverse effects
Report breathing difficulties
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14
Q

Cholinergic receptors

A

Muscarinic

Nicotinic

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15
Q

Direct cholinergic uses

A

Urinary retention

GI tract symptoms

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16
Q

Cholinergic contraindications

A

Bradycardia
Hypotension
Urinary tract or intestinal obstruction
Asthma

17
Q

Antidote for cholinergic overdose

A

Atropine

18
Q

Cholinergic adverse effects

A
Sedation and drowsiness
Hypotension with tachycardia
Excessive salivation
Diarrhea
Cramping
Abdominal pain
Life threatening pulmonary effects
19
Q

Cholinergic Nx Implications/teaching

A

Give 30 min before meal
Have atropine at bedside for OD
Watch for respiratory complications

20
Q

Anticholinergic uses

A
Dry up secretions
Decrease motility GI and GU
Dilation of pupils
Parkinson disease symptom control
Treat bronchospasm
Treat cholinergic crisis
21
Q

Anticholinergic contraindications

A

Narrow angle glaucoma
Myasthenia gravis
Caution with coronary artery disease

22
Q

Anticholinergic drug interactions

A

Antihistamines
Tricyclic antidepressants
Antacids

23
Q

Anticholinergic adverse effects

A
Dryness
Decreases GI and GU motility
Hypertension
Drowsiness, nervousness
Blurred vision
Palpitations
Confusion
24
Q

Anticholinergic Nx Implications/Teaching

A

Give antacids one hour between anticholinergics
Provide oral care
Watch for sedative reaction
Watch for urinary retention and constipation

25
Q
dopamine
albuterol (Proventil)
pseudoephedrine (Sudafed)
phenylephrine (Neo-Synephrine)
norepinephrine (Levophed)
A

Adrenergic

26
Q
bethanechol (Urecholine)
edrophonium chloride (Tensilon)
A

Cholinergic

27
Q
Sympathetic effects on
Heart
Lungs
Pupil reaction
Glucose
GI activity
Sphincter
Blood flow
A

Heart: rate increase, pump increase, automaticity, BP
Lungs: rate increase, depth increase, SpO2 increase, bronchodilation, decrease secretions
Pupil reaction: dilate, increase pressure in back of eye
Glucose: Breakdown of glycogen into fuel, blood glucose levels increase
GI activity: Decreased movement, decreased secretions, decreased blood flow to tract
Sphincter: bladder sphincter contracts
Blood flow: Vasoconstriction, flow increases to where the body needs it

28
Q
Parasympathetic effects on
Heart
Lungs
Pupil reaction
Glucose
GI activity
Sphincter
Blood flow
A

Heart: rate decrease, pump decrease, BP decrease
Lungs: rate decrease, depth decrease, increased secretions, bronchoconstriction
Pupil reaction: constrict
Glucose: Glucose becomes storage, lower blood glucose levels
GI activity: Increased motility, increased secretions
Sphincter: Sphincters relax
Blood flow: Vasodilation, goes to periphery