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Pharmacollogy > ANS > Flashcards

ANS Flashcards

1
Q

Alpha 1 receptors actions

A

contraction :6
facilitate neuro m
increase total perip. R

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2
Q

Alpha 2 receptors actions

A

Dec. CNS
Dec symp.
Dec. renal

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3
Q

Presynatpic Beta actions

A

increase of noradernlaine

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4
Q

postsynaptic BETA 1 actions

A

Inc 4

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5
Q

Postsynaptic Beta 3 actions

A

increase lipolysis & thermogensis

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6
Q

postsyanptic beta 2 actions

A 
Generlized VD
Increase glucogenolysis
increase insulin relase 
increase of reuptake of K 
Skeletal muscle twitches
relaxation of uterus , GIT , UB
Bronchodiltation
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7
Q

where is alpha 1 receptors

A

mainly postsynaptic

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8
Q

absorption of adernaline

A 
not well by oral 
S.C
inhalation
glucoma 
caridac
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9
Q

Fate of adernaline

A

80 & metabilized
18& reuptake
2& uncharged

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10
Q

does adernaline pass BBB

A

no

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11
Q

Local actions of adernaline

A

Skin
MM
EYE *
Inhalataion

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12
Q

systemic actions of adernaline

A 
CNS * 
EYE
BV: 3
Heart:  3
BP 
Respiratory
GIT *
UB 
Uterus *
SK muscle : 3 *
Anti-allergic effect :2
endocrine * : 1
Metabolic : 4-3
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13
Q

Therupatic uses of adernaline

A 
Bronchal asthma
Acute hypoglycemia
Glucoma
Acute allergy *
Cardiac arrest 
hemosatatic *
with local anesthesia *
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14
Q

ADVERSE AFFECTS OF adernaline

A 
twitches
Heart problems
EYE : 2 *
CNS : 2 *
VC :3 *
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15
Q

what is drug that used at heart problems treatment of adernaline

A

B1 blocker : atenlol

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16
Q

what is drug that used for VC treatment of adernline

A

Alpha blocker : parazosin

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17
Q

Contradictions of adernaline

A 
Heart problems
Thyrotoxicosis
Around fingers & toes
hypertension
hemorrhage shock
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18
Q

what is cause of hemorrhage shock

A

renal V.C

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19
Q

Drugs interaction of adernaline

A

General anesthesia
Digitalis
Non-selective beta blockers :2

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20
Q

example for non-selective beta blockers

A

propanolol

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21
Q

what is source of adernaline

A

A cells of dura matter (80%) & some neurons at CNS

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22
Q

what happen if adernaline exposed to air or light or aqueous or alkaline medium

A

oxidized–>Adernochrome : very toxic & red

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23
Q

why at blood adernaline cant be oxidized

A

reducing agents : glutathione & aschrobic acid

24
Q

what is source of nor-adernaline

A

postganglionic sympathateic
Adernal medulla (20%)
phenocytochroma (90%)
CNS

25
Q

How can noradernaline be adminstrated

A

I.V injection

26
Q

Dynamics of noradernaline

A 
CNS
EYE
BV
CVS : 2 *
GIT,UB
UTERUS*
METABOLSIM:2*
27
Q

Therupatic uses of noradernlaine*

A

Acute hypotension

Local anesthesia

28
Q

Adverse affects of noradernaline

A 
Gnagrene
Arryhtemia & badycardia
cerebral hemorhage
abortion
sudden stop-->sever hypotension
29
Q

what are contradictions of noradernaline

A 
hypertension
hemorrhage shock
coronary heart disease
around finger or toe
general anesthesia
extra vasation
sudden stop
30
Q

what are kinetcs of dropamine

A

similar to noradernaline

31
Q

what are dynamics of dopamine

A

small dose : Stimulate D : V.D
Moderate dose : stimulate D & B1 : VD+ inc. COP
large dose : VC

32
Q

therupatic uses of dopamine

A

Resistant heart failure

Shock : 3

33
Q

adverse affects of dopamine

A

vomiting & nausea

hypertension & HEART problems

34
Q

talk about kinetcs of epipherine

A 
Absorbed orally , mm, paraentral
Pass BBB
not metabolized by MAO
excreted uncharged 
acidifcation
35
Q

talk about dynamics of epipherine

A

Dual or mixed
idirect actions
similar to adernaline but weaker , slower,tachypylaxis
stimulate CNS

36
Q

talk about amphetamine

A

more potent to CNS than ephedrine

addiction

37
Q

what are actions of beta blockers

A 
Heart *
BV
Anti-hypertensive*
bronchi
IOP
metabolsim*
ISA
CNS
Na channel block
38
Q

what are uses of beta blockers

A 
increase heart problems *2
Hypertension
\+alpha blockers in phenocytocroma 
glucoma
headche
anxiety & panic syndrome
tremors
39
Q

difference between two types of angina

A

angina of work : can be used with b blocker

variant (prinzmetal) angina : X be used with b blocker

40
Q

what is drug responsible for badchardia , heart failure

A

virpamil

41
Q

what are contradictions of beta blockers

A 
Variant angina + decrease heart prbolems
P.V.D
hypotension
alone with phenocytochroma
brocnho asthma
paractolo
WITH insulin 
hyperkalemia 
inc. TAG or dec, HDL
sediation
sudden stop
42
Q

Talk about non selective beta blocker

A

Heart
Liver
Limbs *
Lung

43
Q

Cardio selective beta 1 blocker

A

small dose with caution

44
Q

example for Cardio selective beta 1 blocker

A

atenlol
bisprolol
metoprolol

45
Q

example for alpha + beta blocker

A

carvilol + latenolol

46
Q

What are central sympathoplegic drugs

A

alpha 2 agonists
imidozlone I1 agnoists
reserprin

47
Q

example for alpha 2 agonists

A

alpha methyl dopa + colidene

48
Q

example for imidozline I1 agonists

A

moxondine

49
Q

who leads to more sedation

A

reserprin

50
Q

2 enzymes of alpha methyl dopa

A

decarboxylase & beta hydroxylase

51
Q

affects of alpha methyl dopa

A 
Maintain RBF
Inhibit catecholamines & serotonin
Fluid , constipation , dry mouth
CNS
Allergy
Hepatotoxcity
hemaolytic anemia
badicardia 
postural hypotension 
decrease renin
52
Q

uses of alpha methyl dopa

A

hypertension : 3

53
Q

can we use colidene at pregenency

A

no

54
Q

what are disadvantages of colidene

A

large dose or iv : intial hypertension

sudden stop : sevre hypertension

55
Q

what is treatment of sudden stop of colidene

A

alpha + beta blocker

Pharmacollogy (6 decks)

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