acute angle closure glaucoma Flashcards

1
Q

what is glaucoma

A

a group of disorders characterised by optic neuropathy duet to raised intraocular pressure (in most patients)

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2
Q

what causes a rise in intra ocular pressure in acute angle closure glaucoma

A

rise in intra ocular pressure secondary to impairement of aqueous outflow

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3
Q

what are the 3 factors predisposing to AACG

A
  1. hypermetropia (long-sightedness)
  2. pupillary dilatation
  3. lens growth associated with age
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4
Q

what are the features of AACG

A

severe pain - ocular or headache
decreased visual acuity
hard, red eye
haloes around lights
semi-dilated non- reacting pupil
corneal oedema results in dull or hazy cornea
systemic upset may be seen - nausea and vomiting even abdominal pain
mydriasis (dilated pupils that do not respond to change in light)

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5
Q

what are systemic features of AACG that can be seen

A

nausea vomiting

abdominal pain

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6
Q

how is AACG managed?

A
  1. emergency. requires prompt urgent referral to opthalmologist
  2. emergency treatment required to reduce intra ocular pressure acutely before definitive surgery
  3. definitive management = laser peripheral iridotomy - creating a tiny hole in peripheral iris
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7
Q

give an example regime for managing AACG

A
  1. combination of eye drops = direct parasympathomimetic, beta blocker, a2 agonist
  2. iv acetazolamide
  3. definitive surgery = laser peripheral iridotomy
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8
Q

give example of a direct parasympathomimetic and its mode of action

A

pilocarpine = causes contraction of the ciliary muscle = opening of trabecular meshwork and increases outflow of the aqueous humour

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9
Q

give example of a beta blocker used in AACG and its mode of action

A

timolol

decreases aqueous humour production

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10
Q

give example of a alpha -2 agonist and its mode of action in AACG

A

APRACLONIDINE

dual action
acts to decrease aqueous humour production and increases uvesceral outflow

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11
Q

describe the action of intravenous acetazolamide in treating AACG

A

reduced aqueous secretions

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