Abnormalities of teeth Flashcards

1
Q

Incomplete or defective enamel formation

Various causes; variety of environmental influences: Turner hypoplasia; antineoplastic agents; fluorosis; syphilis; etc

Restorative treatment as indicated

A

Enamel Hypoplasia

Ameloblasts are very easily affected/influenced by environment: high fever at young age

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2
Q

“Physiological” wear due to tooth to tooth contact during occlusion

A

Attrition

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3
Q

Tooth structure loss secondary to external agent; variety of patterns

A

Abrasion

ex – toothbrush abrasion

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4
Q

Cupped-out depression of occlusal surfaces or cusp tips; associated with regurgitated gastric acid or dietary acid

A

Erosion

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5
Q

Wedge-shaped defect limited to cervical area; bruxism associated

A

Abfraction

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6
Q

Treatment for attrition, abrasion, erosion, abfraction

A

Early diagnosis and intervention

Construction of occlusal guards

Inform patients regarding tooth loss from acidic foods, reflux, etc.

Lost tooth structure- replaced with variety restorative procedures

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7
Q

Relatively rare

Destruction of tooth structure accomplished by cells located in the dental pulp

May be idiopathic or subsequent to trauma

Teeth may appear pink

A

internal resorption

Treatment: Endo therapy prior to perforation  once communication with PDL, poor prognosis

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8
Q

Relatively common

Destruction of tooth structure accomplished by cells located in the PDL

Causes: chronic inflammation, cysts, neoplasms, trauma, re-implantation of avulsed tooth, impactions, orthodontic forces, idiopathic.

A

external resorption

tx - depends on extent, extraction may be necessary

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9
Q

Surface accumulation of exogenous pigment which can typically be removed by prophylaxis.
ie- tobacco, food and beverages, bacterial, medications, etc

A

extrinsic stains

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10
Q

Endogenous material is incorporated into developing teeth  Deposition of circulating substances including drugs (tetracycline) and blood pigments (Rh incompatibility)  Cannot be removed by prophylaxis

A

Intrinsic stains

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11
Q

Reduced number of teeth

Most common dental developmental anomaly; uncommon in deciduous dentition

Genetic vs. environmental etiology

Patient should be evaluated to determine the cause

A

Hypodontia

Most often affects: third molars, second premolars, lateral incisors

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12
Q

lack of 6 or more teeth

A

Oligodontia

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13
Q

total lack of tooth development

A

Anodontia

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14
Q

Presence of “supernumerary teeth”

Occurs in 1-3% of the population  more frequent in permanent relative to deciduous dentition.  95% maxilla/mandible?,
 Terms: mesiodens; distodens; paramolar

A

Hyperdontia

95% maxilla, usually anterior

May affect occlusion, hygiene; may be unesthetic

 Tx - removal may be indicated to avoid complications related to normal eruption and occlusion

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15
Q

Small teeth, may also affect shape

Most commonly affected teeth:

 Tx – restorative dentistry as indicated

A

Microdontia

maxillary laterals (peg) and third molars

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16
Q

large teeth  Uncommon  associated with various syndromes and systemic disease

A

Macrodontia

17
Q

Partial division of single tooth bud; crown appears split  Usually seen in anterior teeth  Tooth count is normal

A

Gemination

central is normal coming out of gingiva then splits into two at incisal edge

18
Q

2 tooth buds merge to form single large tooth  fused teeth have separate root canals, but share cementum and dentin  Reduced number of teeth in arch

A

Fusion

2 different roots coming out of gingiva, but crowns together with dentin and cementum

19
Q

Uncommon condition; characterized by the fusion of two teeth by cementum alone 

May be difficult to detect radiographically  More frequent in posterior _  Clinically significant if one of the involved teeth must be extracted

A

Concrescence

More frequent in posterior maxilla

20
Q

Rare condition that primarily affects the maxillary incisors  Basically an exaggerated cingulum

A

talon cusp

1/2 to CEJ it is a talon cusp
Not half way it is a cingulum

 Significant if the talon cusp interferes with occlusion  Gradual removal to allow for secondary dentin formation

21
Q

Extra cusp in central developmental groove  Premolar teeth most commonly affected; mandibular predominance

Tooth often becomes non-vital, presumably due to attrition or trauma to cusp

A

Dens Evaginatus

22
Q

Dens Evaginatus - Extra cusp in central developmental groove

most commonly found where

A

Premolar teeth most commonly affected; mandibular predominance

23
Q

Uncommon condition that primarily affects the maxillary lateral incisor “Tooth within a tooth”, perhaps due to invagination of tooth bud Quite variable in severity Affected tooth often becomes non-vital shortly after eruption

A

Dens Invaginatus aka dens in dente

24
Q

Enamel Pearl
 Droplets of ectopic enamel
where do we find them?

 Pearls may have dentin and pulp horns  May be detected by probing, if mistaken for calculus, can lead to exposure

A

 Furcations of maxillary or mandibular molars

Will have a dome shaped radiolucency - because PDL attachs to cementum not enamel

25
Uncommon; radiographic finding  Enlargement of the body and pulp chamber of a multirooted tooth; apical displacement of pulpal floor and furcation  Similar to bovine dentition  Result of chromosomal alterations; associated with various syndromes
Taurodontism Effects usually all multi-rooted teeth in quad
26
Hypercementosis  Unusual; incidental radiographic finding  Adults  Asymptomatic deposition of excessive cementum  Local and systemic factors  most cases are sporatic  strong association with _ Disease of Bone
Paget’s hypercementosis - No treatment necessary  Occasional complications with extraction
27
Unusual; radiographic finding  Curvature/bend of tooth roots  Etiology: trauma to developing tooth  Complications: Extraction or RCT may be difficult
dilaceration
28
Group of uncommon genetic disorders affecting enamel of teeth  Varied patterns of inheritance  Weak enamel is easily lost  Affects both primary and permanent teeth
Amelogenesis Imperfecta Clinical implications and management vary according to the subtype and severity  Dental restorative care to address aesthetics, sensitivity, caries, loss of vertical dimension
29
Amelogenesis Imperfecta _ | inadequate deposition of enamel matrix
Hypoplastic 1st step in forming enamel
30
Amelogenesis Imperfecta _ incomplete mineralization This type affects primary and permanent
Hypomaturation enamel matrix laid down correctly, but not maturation
31
Amelogenesis Imperfecta _ | no significant degree of mineralization
Hypocalcified does mature at all
32
Amelogenesis Imperfecta Enamel looks abnormal clinically? radiographically?
 Clinical: rough, smooth, pitted, pigmented, or “snow-capped”  Radiographic: thin enamel of normal or decreased density; normal root and pulp morphology
33
Uncommon  Autosomal dominant inheritance, Due to mutation of DSPP gene  Affects both primary & secondary dentition  Results in abnormal dentin formation
Dentinogenesis Imperfecta Due to mutation of DSPP gene  “dentin sialophosphoprotein” – one of the major non-collagenous proteins of dentin Management depends on the severity of gene expression  May require little restorative care, however implants and/or overdentures are necessary in many cases
34
Dentinogenesis Imperfecta clinically? radiographically? dentin is very soft Enamel chips off and a lot of attrition
Clinical:  Teeth appear translucent; “Opalescent teeth”  Enamel is normal, but poorly supported; rapid attrition of dentition is often seen  Radiographic:  Bulbous crowns and obliteration of pulps, constriction at cervical CEJ Note: Similar dental changes may be seen with osteogenesis imperfecta
35
Dentin Dysplasia Autosomal dominant inheritance, 2 types _ type : roots are very short “rootless teeth”, obliteration of pulp (crescent-shaped remnant in crown), periapical radiolucencies
Type I: Radicular Dentin Dysplasia | ~Teeth may appear normal clinically~
36
Dentin Dysplasia Autosomal dominant inheritance, 2 types _ type : Thought to be related to dentinogenesis imperfecta  Enlarged pulps with “thistle tube” appearance, pulp stones
Type II: Coronal Dentin Dysplasia | ~Teeth may appear normal clinically~