Abnormal Flashcards

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1
Q

What are the criteria of defining abnormality? x

A

A. Statistical deviation from the norm

B. Social norms criteria

C. Rosenhaun & Seligman’s criteria for abnormality - MIS VOUV

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2
Q

What are Rosenhaun & Seligman’s criteria for abnormality - MIS VOUV? x

A

Maladaptiveness - Behavior which makes life more difficult

Irrationality - The person is unable to communicate in a rational manner that is understood by others

Suffering - The behavior causes suffering

Vividness / unconventionality - The person experiences reality in a unconventional way

Observer discomfort - The behavior makes other people uncomfortable

Unpredictability - The person’s behavior is erratic and difficult to predict

Violation of moral standards - The behavior violates accepted standards for right and wrong

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3
Q

Why may deviations from statistical norms be faulty in assessing abnormality? x

A

The statistical deviation from the norm criteria is simple, reliable, and seemingly objective - it is based on statistics, not on values or beliefs. However, it is far from adequate, because not all statistically unusual behavior is considered undesirable or problematic.

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4
Q

Why may deviations from social norms be faulty in assessing abnormality? x

A

The social norms criteria, much like statistics-based definitions, is also simple and reliable. Most of us would agree on whether behavior has violated social norms or not. However, social norms vary a great deal between one country and the next, and also evolve over time

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5
Q

Why may MIS VOUV be faulty in assessing abnormality? x

A

The criteria is more subjective than the others, as two people may come to different conclusions.

It is not clear how many of the seven items must be met for a behavior to be considered abnormal.

Many of the items are clearly related to social norms and judgments, which vary across time and place.

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6
Q

What are the assumptions of the medical model in abnormality? x

A

-Abnormal behavior is a symptom of a mental disorder

-Mental disorders are related to biological abnormalities in the brain. For instance, depression is thought to be related to a lack of serotonin. Mental disorders are often thought of as a “chemical imbalance” in the brain

-Mental disorders are no different than other diseases. Just as your heart or liver can carry a disease, so can your brain

-Mental illness can be treated by correcting the biological abnormalities that caused the disorder. For example, antidepressant drugs increase levels of serotonin in the brain, thereby treating depression

-Normal behavior is simply the absence of any problems in the brain

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7
Q

Pros and Cons of the medical model in abnormality x

A

PROS

Removes blame. If someone was suffering from a broken leg, you wouldn’t blame them for not being able to run very fast. Similarly, the idea that mental disorders are real, biological illnesses, just like any other illness, removes much of the blame from the patient.

Enables research into causes and treatments. Once psychological disorders are understood as real illnesses - rather than just character flaws - scientists can begin studying the causes of these disorders, and most importantly, how they can be treated

CONS

Uncertainty about which disorders are “real”. A controversial example is the huge increase in the number of children (mainly boys) who have been diagnosed with Attention Deficit Hyperactivity Disorder (ADHD) in the United States.

No way to objectively diagnose mental disorders. For most mental disorders, there is no way to objectively test who has the disorder, and who doesn’t. This makes the process of diagnosis rather subjective and unreliable, and raises questions over what behavior should be considered abnormal.

Diagnosis can lead to stigmatization. Being labeled with a mental disorder can cause employers, family, friends or romantic partners to perceive you differently.

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7
Q

Pros of the medical model in abnormality x

A

PROS

Removes blame.
If someone was suffering from a broken leg, you wouldn’t blame them for not being able to run very fast. Similarly, the idea that mental disorders are real, biological illnesses, just like any other illness, removes much of the blame from the patient.

Enables research into causes and treatments.
Once psychological disorders are understood as real illnesses - rather than just character flaws - scientists can begin studying the causes of these disorders, and most importantly, how they can be treated

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8
Q

Cons of the medical model in abnormality x

A

CONS

Uncertainty about which disorders are “real”.
A controversial example is the huge increase in the number of children (mainly boys) who have been diagnosed with Attention Deficit Hyperactivity Disorder (ADHD) in the United States.

No way to objectively diagnose mental disorders.
For most mental disorders, there is no way to objectively test who has the disorder, and who doesn’t. This makes the process of diagnosis rather subjective and unreliable, and raises questions over what behavior should be considered abnormal.

Diagnosis can lead to stigmatization.
Being labeled with a mental disorder can cause employers, family, friends or romantic partners to perceive you differently.

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9
Q

What are the ethical issues of diagnosis? x

A

Stigmatization. - Being called “mentally ill” can have profound consequences for how others perceive and respond to you

Stickiness of labels. - One of the consequences of diagnosis is that it is very difficult to completely shed the label of being mentally ill

Confirmation bias. - Once someone has been labeled as mentally ill, there is a tendency to see evidence of their mental illness in all of their actions

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10
Q

Rosenhan 1973 x

A

Aim: To investigate the experience of being labeled mentally ill, and the ethical issues that are raised by psychiatric diagnosis

Procedure:

8 healthy adults were recruited to take part in the experiment. They all checked themselves into mental hospitals, claiming (falsely) that they heard voices saying words like “empty”, “dull”, and “thud”

After being admitted, the “pseudo-patients” acted normally, and reported that the voices had stopped. During therapy sessions, the patients answered truthfully about their life

Results

All participants were diagnosed with schizophrenia, admitted to hospital, and forced to take psychiatric medication

Patients were kept in the mental hospital in average of 19 days (in one case, 52 days) despite showing no symptoms of mental disorders after being admitted

At no point did doctors or nurses suspect that the patients did not, in fact, suffer from a mental disorder. In fact, hospital staff interpreted many examples of normal behavior as signs of mental illness, demonstrating confirmation bias.

When participants were released, they were diagnosed with “schizophrenia in remission” rather than being considered as cured

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11
Q

How does Rosenhan 1973 fall short in modern times? x

A

In the 1970’s, a large proportion of mentall ill people were kept in mental health facilities, which became notorious for poor conditions and mistreatment of patients. Nowadays, most people diagnosed with mental illness remain in their homes and communities, and are encouraged to integrate in society to the greatest extent possible

At the time of Rosenhan, knowledge of mental illness was still in its infancy, and certainly far less developed than it is nowadays. The current DSM V has far more detail on specific disorders and symptoms than the DSM II version that was used at the time of Rosenhan’s study

Psychiatry has developed better methods of diagnosis, such as structured interviews, which check for specific symptoms. These methods of diagnosis had still yet to be developed in the early 1970’s

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12
Q

Langer and Abelson 1974 x

A

Aim: Investigate how stigma, labels and confirmation bias affects perceptions of the mentally ill

Procedure

The participants in this study were two groups of clinical psychologists. The first group were analytic psychologists, who tend to view mental illness as a consequence of internal conflicts and childhood trauma. The second group were behavioral psychologists, who tend to focus more on identifying and changing negative patterns of behavior, in the here and now

Participants watched a video of a man being interviewed about his feeling and experiences concerning his past work

Half of the participants were told that the man was a “job applicant”, while the other half were told that the man was a “patient”. The label given to the man was the independent variable in this study

Participants then rated the man according to how “disturbed” or “well-adjusted” he was. The rating of the man’s mental health was the dependent variable in this study

Results

The behavioral psychologists tended to rate the man as fairly normal, regardless of the label

The analytic psychologists, on the other hand, rated the man as significantly more disturbed when they were told that he was a “patient”

Same man who was described as “realistic”, “unassertive”, “attractive”, “innovative” became “tight, defensive”, “dependent, passive aggressive” and had a “conflict over homosexuality” when labeled a patient

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12
Q

Weisman et al 2005

A

Aim: To investigate the prevalence of depression in different countries

Procedure

This study was carried out in 10 countries across the world, including a range of different cultures - the United States, Canada, Puerto Rico, France, West Germany, Italy, Lebanon, Taiwan, Korea, and New Zealand.

Participants in each country were randomly selected using telephone registries. A trained interviewer would call the people selected and interview them about their mental health history. A total of 38,000 participants were interviewed

Results

Rates of depression varied tremendously in different countries. The lifetime prevalence of depression was only 1.5% in Taiwan, but as high as 19% in Beirut, Lebanon

Surprisingly, the rate of depression in France (16.4%)

In spite of the large variation in depression rates, some patterns were noticed across the world. In each country, the rate of depression in women was 2-3 times higher than in men. And people who were separated or divorced had much higher rates of depression than people who were currently married

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13
Q

Why may depression prevalence vary?

A

Different socioeconomic conditions - In some countries, many people struggle with poverty, constant stress, the threat of violence or war, or long periods of unemployment. Living in such difficult conditions may lead to higher rates of depression than living in stable, safe and prosperous countries

Different rates of urbanization - As countries develop economically, more people tend to move out of villages and small towns, and into crowded, stressful cities. Some researchers believe that urban living is linked to higher rates of depression, since city dwellers are more likely to be stressed and socially isolated, living busy, chaotic lives in the midst of thousands of anonymous strangers

Differences in cultural stigma - In some countries, mental illness is still considered a shameful topic, one which is kept hidden behind closed doors. In these countries, more people suffer depression in secret, without ever being diagnosed. If they do seek help, they may report physical symptoms only (like being unable to sleep, or muscle pain), and hence may never be given the correct diagnosis

Differences in how depression is diagnosed - In some countries, psychiatrists may be more likely to diagnose depression than others, depending on the culture of mental health treatment. The line between mild depression and ordinary sadness is somewhat blurry, and so rates of depression could vary because of different mental health training and practices

Discussion points may include, but are not limited to:
* age (for example, higher prevalence rates for depression in the elderly population and
increasing rates among youth)
* gender (for example, higher prevalence for depression in women)
* risk factors such as exposure to conflicts, traumatic events, physical or psychological
abuse
* social and cultural factors (for example, poverty, social, and cultural norms)
* availability of mental health services and treatment (for example, social determinants in
relation to access to treatment and health services)
* Diagnostic criteria and classification systems
* Risk of relapse once the research is over
* Methodological and ethical considerations related to the research into prevalence rates
of disorders.

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14
Q

What is the serotonin hypothesis

A

According to this hypothesis, the cause of depression is low levels of serotonin in the brain. Low levels of serotonin make it more difficult for signals to be passed from one neuron to the next, slowing down the flow of brain activity. This results in the tell-tale signs of depression - such as low energy, lack of enjoyment of life, and feelings of sadness. The best treatment for depression, therefore, are antidepressant drugs, which work by raising the level of serotonin in the brain’s synapses. These drugs are known as SSRI’s. They work by inhibiting the reuptake of serotonin, causing more of the neurotransmitter to remain in the synapse

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15
Q

What is the The diathesis-stress model

A

The diathesis-stress model explains how genes and the environment interact to determine mental health. According to the model, some people have genes that predispose them to depression (diathesis). But not all of these people will become depressed. As long as life goes relatively smoothly, these people will be able to function normally. However, if stressful life events happen - perhaps a loved one dies suddenly, or the person goes through a difficult breakup or divorce - the combination of high levels of stress and genetic vulnerability will make depression more likely.

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16
Q

Caspi et al 2003

A

Aim: Investigate how genetic vulnerability and negative life experiences can interact to cause depression

Method: Quasi - lognitudinal

Procedure

Genetic testing was carried out on (1,037) of New Zealand children to determine which version of the serotonin transporter gene (5HTT) they carried

There are two alleles (versions) of the 5HTT gene: a short version, and a long version. The long version of the gene is associated with higher levels of serotonin in the synapse

The particpants filled out a life callander on how many stressful life events they had experienced (including financial, employment, health, and relationship). They were assed for depression using DSM-IV criteria, and on informant reports (‘someone who knows you well’)
Findings

Participants who had experienced few stressful life events had low rates of depression, regardless of their genes

However, participants with at least one short version of the 5HTT gene who also had experienced three or more stressful life events had the highest rates of depression

Participants with two long 5HTT alleles had low rates of depression, no matter how many stressful life events they had experienced

Conclusion:
The gene does not increase number of stressful life events (not niche picking), but rather it is an interaction between genes and thhe evironment.

[GENE-ENVIRONMENT CORRELATION AND INTERACTION]

17
Q

Arguments for and against the serotonin hypothesis

A

Points in favor of the serotonin hypothesis:

Antidepressant drugs (SSRI’s) work by increasing levels of serotonin, and they are effective in treating depression for many people (although far from everyone)

Research by Caspi suggests that different alleles of the serotonin transporter gene (5HTT) determine risk of depression, suggesting that serotonin plays a significant role

Points against the serotonin hypothesis:

Antidepressant drugs increase levels of serotonin in the brain immediately, but most people don’t report feeling better for 3-4 weeks after taking the drugs. If depression was only about serotonin levels, then why does it take so long for improvements in mood to take place?

Antidepressants only seem to be effective for about 60% of people. If depression was really caused by a lack of serotonin, then why aren’t antidepressants effective for everyone?

Even if increasing serotonin levels helps treat depression, that doesn’t necessarily mean that low serotonin causes depression. For example, we know that aspirin helps treat headaches, but that doesn’t mean that headaches are caused by low levels of aspirin

18
Q

Strengths and limitations for Aaron Becks’s cognitive theory about depression

A

Strengths

The “negative triad” accurately describes how most depressed people think about themselves, the world, and their future. Depressed people often do, in fact, think in a distorted, illogical manner, and this contributes to their helplessness and lack of motivation

Beck’s theory of depression suggests that it may be possible to change negative thought patterns through therapy, and in doing so, effectively treat depression. In fact, there is evidence that Cognitive Behavioral Therapy (CBT), a method of therapy focused on recognizing and changing illogical negative thoughts, is an effective treatment for depression

Limitations

It is not clear why certain people develop negative thoughts in the first place. Genetic factors, or negative life experiences, may predispose a person to a negative thinking style. If biology does shape a person’s thinking style, then perhaps genetic factors are the true “root causes” of depression

It is not clear whether automatic negative thoughts and cognitive distortions are the cause of depression, or a symptom of depression. An alternative explanation of depression could be that biological factors (like levels of neurotransmitters) can affect one’s emotions, and negative emotion leads to negative thought patterns, and so biological factors are the ultimate cause of depression

19
Q

Cognitive Theory of Depression

A

According to psychologist Aaron Beck, depression is caused by cognitive distortions and illogical thinking processes. These patterns of faulty, negative thinking lead to a grim, hopeless view of the world, and often result in depression.

According to Beck, cognitive distortions can result in frequent, automatic and seemingly uncontrollable negative thoughts about the self, the world, and the future, referred to as the negative triad.

20
Q

3 illogical thinking processes

A

Selective attention - when you only focus on the negative aspects of an event

Magnification - when you exaggerate the importance of negative life events

Overgeneralization - when you draw broad conclusions on the basis of a single, negative life event

21
Q

What is the negative triad?

A

Negative thoughts about oneself, such as “I can’t do anything right”, “I’m worthless, or “I wish I was different”

Negative thoughts about the world, such as “Nobody cares about me”, “Everybody is selfish”, or “It’s a cruel world”

Negative thoughts about the future, such as “I’ll never achieve my goals” or “My future is hopeless”

22
Q

Alloy et al 1999

A

Aim: To determine if cognitive styles can predict who will become depressed

Procedure

A large number (347) of American college freshman, with no prior disorders, were given a questionnaire to determine their thinking style - which was categorized as either being “positive” or “negative”

Participants were then followed for 6 years, and rates of depression were compared between the participants who had a positive cognitive style and a negative cognitive style

Results

Participants with a “negative” cognitive style were far more likely to become clinically depressed in the 6 years of the study

23
Q

Haefel et al

A

Aim: Investigate whether thinking style and risk for depression can be influenced by the people around you

Procedure

Participants were 103 college students, who were randomly assigned a roommate during their first year in college

Participants completed an online questionnaire to measure cognitive vulnerability and depressive symptoms after 1 month of arriving on campus, and again after 3 months and 6 months

Findings

Participants who were randomly assigned to a roommate with high cognitive vulnerability seemed to “catch” the negative thinking style and become more vulnerable themselves

Furthermore, the students that developed increased cognitive vulnerability after 3 months had nearly twice the level of depressive symptoms after 6 months compared to students who didn’t show an increase

24
Q

What makes up the Vulnerability model?

A

Protective factors decrease the risk of depression, and can help a person overcome life challenges and stressful events. Examples of protective factors include:

-Close bonds with other people, who can offer understanding, support and assistance during difficult times

-Strong sense of community, such as active involvement in a religious community, can provide a sense of meaning, a foundation of emotional support, and a wealth of connection with others

Vulnerability factors increase the risk of depression, and can cause a person to be especially susceptible to life challenges and stressful events. Examples of vulnerability factors include:

-Unemployment, which can create financial strain and a loss of self-confidence

-Social isolation, which can leave a person with nobody to confide in or seek support from

-Three or more young children at home, which can contribute to high stress and a lack of time for adult relationships

Provoking agents are stressful life events, such as the death of a family member, the loss of a job, or the dissolution of a relationship. Whether provoking agents trigger the onset of depression depend on how many protective or vulnerability factors are present.

25
Q

Brown and Harris 1971

A

Aim: Investigate the role of social factors and stressful life experiences in depression

Procedure

Carried out a survey on 458 women in South London

The participants were asked questions regarding whether they had suffered any episodes of depression in the past year, and were asked to describe any difficult life events

Results

Around 8% of the women had experienced an episode of depression in the past year

Of the women who became depressed, nearly all of them (90%) had experienced stressful life events, such as the death of a close family member, or the loss of a job. ​By contrast, 30% of the women who did not become depressed also experienced stressful life events

Furthermore, social class played an important role in depression risk, especially for women with children. Lower-class women with children were four times as likely to develop depression as middle-class women with children

26
Q

Homes and Rahe 1967

A

Aim: To determine which events are most likely to lead to illness

Procedure

Medical records of 5,000 patients were reviewed to investigate correlations between stressful life events and illnesses

Results

Positive correlations were found between a number of life events and subsequent illness

In general, events which caused a greater degree of “life change” were associated with the greatest risk of illness. Therefore, Homes and Rahe developed a scale to rank the effect of life events. According to this measure, the most stressful life events are:

​​Death of a spouse or child (100)
Divorce (73)
Marital separation (65)
Imprisonment (63)
Death of a close family member (63)
Personal injury or illness (53)
Marriage (50)
Dismissal from work (47)
Marital reconciliation (45)
Retirement (45)

It is interesting to note that even positive events (such as marriage) can increase the risk of illness, because they are associated with major life changes

27
Q

Strengths and limitations for a social cultural explanation of depression

A

Strengths

There is considerable research showing an association between stressful life events (like the death of a spouse, or job loss) and the onset of depression.

Social explanations for depression are consistent with the diathesis-stress model, which predicts that a combination of biological vulnerability and stressful life events cause depression

Limitations

Most of the research on social factors in depression is correlational, and so cannot establish which factors cause depression.

Many people experience negative life events without becoming depressed. Perhaps cognitive factors - how a person thinks about a life event - matters more than the event itself. For instance, some people might find a divorce to be devastating, while others might find it liberating - all depending on how they choose to think about the event.

28
Q

How do SSRI’s work?

A

SSRI’s work by blocking the reuptake of serotonin back into the presynaptic neuron. This causes more serotonin to remain in the synapse, leading to changes in mood, activity levels, and appetite. The use of SSRI’s is based on the serotonin hypothesis of depression. If depression is indeed caused by low levels of serotonin in the synapse, then increasing levels of serotonin is the most direct way to cure the disorder

29
Q

Elkin et al 1989

A

Aim: Compare effectiveness of different treatments for depression

Procedure

The study involved 280 depressed people, treated by 28 clinicians
Participants were randomly assigned to one of the following treatment groups:​
​A) An antidepressant drug (Imipranine)
B) Interpersonal therapy
C) Cognitive behavioral therapy (CBT)
D) A sugar pill (placebo)​
The study was double blind, meaning neither the clinician nor the patient knew if they were receiving a real antidepressant or a sugar pill

Results

Participants on imipranine showed the fastest improvement, but after a few weeks the participants on therapy also showed signs of improvement
After several months, just over 50% of participants in each treatment group recovered from depression, regardless of what kind of treatment they received
In contrast, just 29% of participants in the placebo group recovered

30
Q

Kirsch et al 2008

A

Aim: Evaluate the effectiveness of SSRI’s in treating depression

Procedure

A meta-analysis was carried out of all clinical trials of SSRI’s submitted to the FDA (Food and Drug Administration), 47 trials in total
Included studies whose findings were not previously reported

Findings

For patients with mild to moderate depression, the difference between SSRI’s and placebo was relatively small. At least 75% of the improvement seen with SSRI’s was also seen with the placebo

For patients with severe depression, there was a larger difference between SSRI’s and placebo

Around half of studies failed to find a statistically significant difference between SSRI’s and placebo

31
Q

Strengths and Limitations of using SSRIs

A

Strengths

Research has consistently shown that SSRI’s are better at treating depression than a placebo, and the difference is especially pronounced for those with severe depression

SSRI’s are relatively cheap and easy to prescribe. They don’t require long hours of expensive therapy

According to some research, patients given SSRI’s show faster improvement than those who receive therapy. In order for therapy to be effective, an open and supporting relationship between therapist and patient needs to develop, which can take considerable time

Not everyone feels comfortable sharing their deepest thoughts, fears and insecurities with a therapist. For patients unable or unwilling to discuss very personal issues with a professional, SSRI’s provide the best option

Limitations

SSRI’s don’t work for everyone. Nearly half of patients are not helped by SSRI’s. Furthermore, there are many different SSRI’s available, and no way to determine which medication will work for which patient

SSRI’s are associated with many side effects, such as nausea, weight gain, insomnia, and loss of sexual desire

Patients who stop taking SSRI’s are at a high rate of relapse (becoming depressed again). Some argue that therapy teaches life-long skills that help people overcome depression in the long run, while SSRI’s only provide a temporary solution that only lasts as long as medication is taken

While SSRI’s can help alleviate the symptoms of depression, they may not actually be treating the “root cause” of depression

32
Q

Riggs et al 2007

A

Aim: Study the effectiveness of CBT, both on its own and in combination with an antidepressant

Procedure

The study involved 126 teens who suffered from depression and substance use disorders

Participants were randomly assigned to either receive CBT and a placebo, or CBT and an SSRI (selective serotonin reuptake inhibitor)

The study was double-blind, meaning that neither the participants nor the therapists knew who received the placebo and who received the SSRI

Results

67% of participants who received the CBT and placebo were rated as “very much” or “much” improved

76% of participants who received CBT and a SSRI were rated as “very much” or “much” improved

33
Q

Strengths and limitations of CBT

A

Strenghts

There is considerable research evidence (eg. Elkin, Riggs) that CBT is significantly more effective than a placebo in the treatment of depression, and about as effective as antidepressant drugs (without the side effects). Furthermore, the combination of CBT and antidepressants is even better than either treatment alone

In contrast to other forms of therapy (particularly psychoanalysis), CBT does not require endless years of therapy, or digging deep into questionable childhood memories. Instead, it is focused, efficient (requiring only 10-12 sessions), and results-oriented

CBT can help patients develop important life-long cognitive skills - like identifying and managing automatic negative thoughts - which can help prevent further episodes of depression, reducing the risk of relapse

Limitations

Some research (eg. Elkin) suggests that antidepressant drugs result in faster improvement than CBT. For severely depressed or suicidal patients, it may be risky to wait several months in hopes that CBT will deliver improvement, rather than beginning a course of antidepressants immediately

CBT can only be effective if a positive, trusting relationship between a therapist and patient is established. For patients who are not comfortable sharing their darkest thoughts with a therapist, CBT may be of limited use

CBT requires around 10-12 hours of one-to-one therapy with a qualified, professional therapist, which may be expensive. CBT may not always be covered by insurance, hence may not be practical for low-income patients

34
Q

What is CBT composed of?

A

CBT is a common form of therapy used for depression. It usually consists of 10-12 one hour long sessions with a therapist. There are two components to CBT: cognitive restructuring and behavioral activation.

Cognitive restructuring involves:

Helping the patient become more aware of their negative thoughts, and identifying cognitive distortions, such as overgeneralization and magnification, in the patient’s thought processes
​​
Negative beliefs are then challenged by the therapist, who encourages the patient to consider other ways of thinking or interpreting events

The patient is then encouraged to replace negative thoughts with more positive, rational thoughts

Behavioral activation is an equally important component of CBT. Patients work with therapists to get re-engaged in positive and rewarding activities, which helps break the cycle of depression.

Behavioral activation involves:

Help in planning enjoyable activities, ideally with others

Overcoming obstacles (logistical, financial) in taking part in enjoyable activities

35
Q

What are the cultural barriers in treating depression?

A

Cognitive barriers include beliefs that seeking professional psychological treatment is unnecessary, a sign of weakness, or ineffective.

Affective barriers include feelings of shame at seeking psychological help, or anxiety at being judged by others.

Sociocultural barriers include the reluctance to share personal and family problems with a stranger, particularly someone from a different culture.

36
Q

Kinzie et al 1987

A

Aim: Investigate cultural barriers to treatment for depression

Procedure

Carried out blood tests on 41 South-east Asian patients with depression who had been prescribed tricyclic antidepressants in U.S. clinics

The blood tests were intended to measure compliance with prescribed treatment

Results

No sign of medication usage was detected in the blood of 61% of the patients

Only 6 of the patients (15%) had therapeutic levels of antidepressants in their blood, indicating they were taking the medication often enough for it to be helpful

After a discussion session was held with patients, in which the benefits and side effects of medication were explained, compliance rates increased

37
Q

What is indigenous therapy?

A

Indigenous therapy is carried out by someone within the patient’s culture, who is sanctioned within that culture to be a therapist. The therapy is carried out in the patient’s language, without the involvement of cultural outsiders

Indigenous therapy incorporates beliefs and practices that are rooted within the patient’s culture. It may incorporate spiritual beliefs, ritual, and ceremony. There is often more emphasis on collective healing, involving the family and wider community

38
Q

Prevalance rates (2)
Biological explanation for depression (2)
Cognitive explanations for depression (2)
Social cultural explanations for depression (2)
Effectivness of treatment and bio/psych treatment (4)
Role of culture in treatment (2)

A

Prevalance rates:
-Brown and Harris 1971
-Weisman et al 2005

Biological explanation for depression:
-Caspi et al 2003
-Kendler et al 2006

Cognitive explanations for depression:
-Haefel et al
-Alloy et al 1999

Social cultural explanations for depression:
-Brown and Harris 1971
-Homes and Rahe 1967

Effectivness of treatment:
-Elkin et al 1989
-Kirsch et al 2008
-Riggs et al 2007
-Hollon et al 2005

Role of culture in treatment:
-Kinzie et al 1987
-Griner and Smith 2006

38
Q

Kendler et al 2006

A

Aim: To investigate the heritability of depression

Method: Correlation (interviews; twin study)

Participants: 42000 twins from Sweden’s twin registry

Procedure: The participants were interviewed and assessed for major depressive disorder in accordance with DSM-IV

Results: The researchers estimated from the concordance rates in mono- & dizygotic twins that the heritability of depression is ≈ 38%- The rate was higher in females. (MZ male - 31%: MZ female - 44%: DZ males - 11% DZ females - 16%)

39
Q

Griner and Smith 2006

A

Aim: Examine effectiveness of culturally adapted treatments of mental disorders

Method: Meta analysis of 76 studies cultural adaptations ranged from consultations with individuals familiar with clients culture to cultural sensitivity for staff

Participants: 25000

Procedure:
-Moderately strong benefit of culturally adapted interventions
-Therapist speaking native language more effective than therapist speaking english
-Benefit 4 times stronger for same race groups than mixed race

40
Q

Hollon et al 2005

A

Aim: Asses relapse rates of MDD after different treatment methods

Participants: 180 (104 responded) patients with moderate to severe depression aged 18 to 70

Procedure:
1: patients who responded positively to cognitive therapy and were withdrawn for 12 months
- 2: patients who responded to medication and continued medication
- 3: patients who responded to medication and continued to take a placebo

Findings: Relapse rates
1: CBT - withdrawn 31%
2: meds - continued 47%
3: meds - placebo 76%