ABGs Flashcards

1
Q

Normal pH

A

7.35-7.45

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2
Q

normal Pao2

A

75-100

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3
Q

normal paCo2

A

35-45

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4
Q

normal hco3

A

22-28

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5
Q

normal sao2

A

94-100

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6
Q

normal arterial vol

A

15-22%

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7
Q

normal venous vol

A

11-16%

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8
Q

critical pH values

A

<7.25, >7.6

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9
Q

Critical PaCo2

A

<20, >60

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10
Q

critical HC03

A

<10, >40

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11
Q

Critical PaO2

A

<40

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12
Q

O2 sat critical

A

<75%

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13
Q

What should you perform prior to ABG?

A

allen’s test

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14
Q

PaCO2 is elevated in

A

respiratory acidosis
metabolic alkalosis

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15
Q

PaCO2 is decreased in

A

respiratory alkalosis
metabolic acidosis

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16
Q

Majority of the CO2 in the blood is

A

HCO3 - regulated by the kidneys
elevated in metabolic alkalosis, decreased in metabolic acidosis

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17
Q

indirect measure of O2 in arterial blood

A

PaO2

decreased in: oxygen diffusion issues, premature mixing of arterial/venous blood (CHD), overperfusion (Pickwickian syndrome)

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18
Q

% of hemoglobin saturated w/ oxygen

A

O2 saturation
if carbon monoxide poisoning –> put O2 on

19
Q

amount of oxygen in the blood

A

O2 sat * Hgb * 1.34 * PO2 * .003

20
Q

base excess/deficit calculated by

A

pH, PaCO2, Hct, measuring amount of buffering anions
negative = metabolic acidosis
positive = metabolic alkalosis or compenstaed resp acidosis

21
Q

When to draw an ABG?

A

resp failure, acid-base disorders, monitoring critically ill patients, evaluation of organ function, guiding therapy decisions

22
Q

interpret ABG

A

1) check pH
2) analyze paCO2
3) analyze HC03
4_ compensatory mechanisms

23
Q

high PaCO2, low pH

A

respiratory acidosis from COPD, asthma, overdose

24
Q

low PaCO2, high pH

A

respiratory alkalosis from hyperventilation, anxiety, pain

25
Q

low HCO3, low pH

A

metabolic acidosis from diabetic ketoacidosis, renal failure

26
Q

high HCO3, high pH

A

metabolic alkalosis, vomiting, diuretic use

27
Q

CIs to ABG

A

absence of pulse, infection, - allen test, AV fistula proximal to site of acess, severe coaguloopathy

can cause arterial occlusion, nerve injury

28
Q

metabolic alkalosis can be from

A

hypokalemia
hypochloremia
chronic + high vol gastric suction
chronic vomiting
aldosteronism

29
Q

respiratory alkalosis can be from

A

CHF, cystic fibrosis, CO poisoning, anxiety, pain, preganncy

30
Q

metabolic acidosis can be from

A

keto acidosis, lactic acidosis, severe diarrhea, renal failure

31
Q

respiratory acidosis can be from

A

resp failure

32
Q

Tx: respiratory acidosis

A

improve ventilation

33
Q

Tx: respiratory alkalosis

A

treat underlying cause

34
Q

Tx: metabolic acidosis

A

treat underlying cause

35
Q

Tx: metabolic alkalosis

A

correct electrolyte imbalances, treat vomiting

36
Q

COPD
oversedation
head trauma
over-oxygenation in patients w/ COPD causes

A

increased PaCO2

37
Q

hypoxemia
PE
anxiety
pain
pregnancy

A

decreased PaCO2

38
Q

chronic vomiting
aldosteronism
use of mercurial diuretics
COPD

A

increased HC03

39
Q

chronic or severe diarrhea
chronic use of loop diuretics
starvation
DKA
AKI

A

decreased HCO3

40
Q

polycythemia
increased oxygen
hyperventilation

A

increased PaO2 + O2 content

41
Q

anemias
mucus plug
bronchospasm
pneumothorax
pulmonary edema
ARDS

A

decreased PaO2 + O2 content

42
Q

anion gap

43
Q

high anion gap

A

lactate, ketones, renal, toxins

44
Q

normal anion gap

A

chloride, acetazolamide, addisons
GI causes - diarrhea ,vomtiing, fistulas