A2 biopsychology Flashcards

1
Q

what is localisation theory?

A

different parts of the brain perform different tasks and are involved with different parts of the body

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2
Q

what is lateralisation?

A

physical and psychological functions are controlled by a particular hemisphere

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3
Q

give detail on the motor area

A

located in the back of the frontal lobe and controls body movement on the other side of the body

damage results in a loss of control over fine movements

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4
Q

give detail on the auditory area

A

located in the temporal lobe and analyses speech based production
info from the left ear goes to the right hemisphere

damage may result in hearing loss

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5
Q

give detail on the somatosensory area

A

at the front of the parietal lobes where sensory info from the skin is represented

the amount of somatosensory area given to a particular body part indicates how sensitive it is
e.g face and hands occupy over half of somatosensory

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5
Q

give detail on the visual area/cortex

A

in the occipital lobe
the eye sends info from the right visual field to the left visual cortex and vice versa

damage to the hemisphere can produce blindness in the opposite eye

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6
Q

What are brain imaging techniques

A

These allow doctors to research and view activity in the human brain

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7
Q

explain fmri

A

used to measure brain activity whilst someone performs tasks

regions of the brain that are rich in oxygenated blood are active
these 3D images show which part of the brain is responsible for a task

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8
Q

explain EEG

A

tracks brainwave patterns by putting electrodes on the scalp

detects problems with electrical activity that may be responsible for certain brain disorders

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9
Q

explain ERP

A

stimulus is presented to participant and the researcher looks for activity triggered by the stimulant through electrodes on the scalp

ERPs are linked to cognitive processes like attention and memory

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10
Q

explain post mortem examinations

A

brain is analysed after death to determine if certain behaviours were due to brain abnormalities

often conducted on people with rare disorders#

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11
Q

what is the difference between temporal resolution and spatial resolution

A

temporal: measurement w respect to time
spatial: the size of the smallest object that can be detected in an image

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12
Q

weakness of FMRI?

A

doesnt provide a direct measure of neural activity, just measures blood flow.
cant infer causation and say that part of the brain is responsible for a particular function

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13
Q

strengths of fmri

A

good spatial resolution (1-2mm),greater than others
can determine activity of regions with greater accuracy

non invasive. does not use radiation or involve instruments directly into the brain, its risk free so more patients can undertake and more data can be found

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14
Q

weaknesses off ERP

A

Erp has poor spatial resolution
only detect activity in superficial regions of the brain, not deeper regions

erp is uncomfortable as electrodes are attached to the scalp. discomfort may affect cognitive response

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15
Q

strengths of erp

A

good temporal resolution because it has readings every millisecond, accurate measurement of electrical activity when doing a task

non invasive. no radiation or instruments directly in the brain

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16
Q

strength of eeg

A

important in diagnosis of epilepsy, random burst in brain activity

good temporal resolution, readings every millisecond

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17
Q

weakness of EEG

A

electrical activity is detected in several regions of the brain simultaneously. difficult to pinpoint exact region and draw causal relationship.

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18
Q

post mortem strength

A

improve medical knowledge, broca and wernicke relied on pms

can access areas like the hypothalamus and hippocampus, which scanning techniques cannot, insight into deeper regions of the brain

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19
Q

weakness of post mortems

A

issue of causation, deficit a patient displays during their life may not be linked to the brain but another illness like mental health problems

ethical issues patients may not be able to provide consent

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20
Q

explain Brocas area

A

left hemisphere, makes up part of the language centre.
areas in the frontal lobe responsible for speech production

damage causes brocas aphasia: speech that is slow, laborious and lacks fluency

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21
Q

explain wernickes area

A

in the temporal lobe responsible for language comprehension

damage produces wernickes aphasia where patients can produce language but their speech is meaningless and they produce nonsense words

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22
Q

studies against localisation

A

evidence against localisation, lashley removed parts of rats brains (10-50%) whilst they learnt a maze. no area was more important, the learning process required every part of the cortex and wasnt localised. however rats learning a maze cant be compared to the complex learning we see in humans

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23
Q

study to support localsisation

A

phinneas gage had a metal rod forced through his head when dynamite exploded at his job, permanent damage to left frontal lobe, His friends believe his personality changed so personality is localised to the left frontal lobe. HWVER, this is too unique to generalise and the trauma of the incident could’ve been what caused the change

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24
Q

what is plastcicty

A

the brains ability to change and adapt as a response to new learning and experiences

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25
Q

AO1 brain plastcity

A

in infancy the brain has rapid growth in the number of synaptic connections with 15,000 at 2-3 as children are constantly taking in new information which is twice as much as adult brain

synaptic pruning: rarely used connections are deleted whereas frequently used connections are strengthened

neural connections can change or be formed at any time in life

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26
Q

exp-lain AO2 research into plasticity

A

maguire et al studied the brains of london taxi drivers and found more volume of grey matter in the posterior hippocampus than the matched control group

this part of the brain is associated with development of spatial and navigational skills

their brain adapted as a result of new learning

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27
Q

AO3: strength of plasticity

A

its may be a lifelong ability
bezzola et al shows how 40hrs of golf training leads to changes in the neural representation of movement in participants age 40-60. Fmri showed increased activity in motor area than a control group. suggesting NP is lifelong

however adults may find it harder to learn than children as albert et al suggests that general NP is much stronger in children (more synaptic connnections)

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28
Q

A03 : negative of plasticity

A

plasticity has maladaptive behavioural consequences. evidence has shown the brains adaptation to prolonged drug use leads to poorer cognitive functioning later in life as well as an increased risk of dementia.

shows the brains ability to adapt to damge may not always be positive but still supports the fact that it can adapt.

29
Q

what is functional recovery?

A

a from of plasticity

after damage the brain is able to redistribute functions that are usually performed by the damaged area to a non damaged area of the brain

30
Q

A01 for functional recovery

A

after damage unaffected areas can adapt and compensate for damaged areas

this can occur spontaneously then slow down after weeks or months

the brain rewires and reorganises itself by forming new synaptic connections close to the area of damage. secondary neural pathways that wouldn’t typically be used are activated to allow functioning to continue

31
Q

What is reformation of blood vessels?

A

brain deals with indirect physical effects of trauma e.g damaged blood vessels

32
Q

what is axonal sprouting?

A

the growth of new nerve endings which connect with undamaged nerve cells to form new pathways

33
Q

what is recruitment of homologous areas?

A

areas on the opposite side of the brain carry out functions of the damaged area

34
Q

weakness of functional recovery?

A

individual differences like education level influence recovery.
Schneider et al found that the more time people with brain damage spent in education the greater their chance of disability free recovery.
40% of people who achieved DFR had more than 16 years in education.
not everyone benefits from functional recovery equally.

35
Q

strengths of functional recovery?

A

evidence to support from animal studies.
Tajiri et al found stem cells given to rats after brain trauma show a clear development pf neuron like cells in the area of injury. shows the ability of the brain to form new connections.

real life application in development of neuro rehabilitation. uses motor therapy and electrical stimulation to counter the deficits in motor and cognitive functions following accidents and strokes. e.g constraint induced movement therapy for strokes (practice using the affected part of the body whilst the other side is constrained)

36
Q

what is hemispheric lateralisation?

A

the idea that two hemispheres of the brain are functionally different and certain mental processes are mainly controlled by hemisphere than the other

37
Q

what does the corpus collosum do?

A

allows info received from one hemisphere to be sent to the other

38
Q

explain sperrys procedure

A

image or word is shown to the right eye with left eye closed. and then the same image/word is shown to the left eye with the right eye closed.

in the normal brain the corpus collosum would share any info between hemispheres

in split brain patients info couldn’t be conveyed from one hemisphere to another

39
Q

explain sperrys results

A

when an image was shown to the right eye they could easily describe what was seen.
when shown to the left eye they couldnt describe it. this is due to lack of language centres in the right hemisphere and no corpus collosum to relay information

40
Q

weaknesses of lateralisation and split brain research

A

differences in function may be overstated. Sperry says left =speech and right = recognition and drawing. however functional recovery suggests functions of the damaged area may be taken over by non specialised areas in the opposite hemisphere e.g lang controlled by right

cant establish causal relationship w/ sperry. compared 11 split brain patients w epilepsy to neurotypical control group who had no epilepsy. having epilepsy is a confounding variable and any differences could be a result of this

41
Q

strengths of split brain and lateralisation

A

research: in connected brains 2 hemispheres process info differently. fink et al pet scans to see what areas of the brain were active during a visual processing task. ptps w connected brains had to attend to global elements of an image(e.g a whole forest), regions of the right hemisphere were much more active. when focusing on finer details (individual trees)the specific areas of the left hemisphere dominated

support from more split brain research. split brains performed better than connected controls on certain tasks e.g faster identifying the odd one out than controls. in a normal brain the left hemispheres better cognitive strategies were weakened by the inferior right hemisphere

42
Q

what is a circadian rhythm ?

A

patterns of behaviour that occur every 24 hours and is set and reset by environmental light levels

43
Q

name 3 types of circadian rhythms?

A

the sleep wake cycle
core body temperature
hormone production

44
Q

how do the circadian rhythms work?

A

circadian rhythms are driven by our body clocks and synchronised by the SCN in the hypothalamus.

45
Q

how does light affect circadian rhythms

A

light provides the primary input to this system, setting the body clock to the correct time which is photoentrainment.

in mammals light sensitive cells in the eye act as brightness detectors and send messages about environmental light levels to the scn.
the SCN uses this to coordinate activity of the whole circadian rhythm.

46
Q

what is the SCN?

A

an endogenous (internal) pacemaker in the hypothalamus of each hemisphere of mammals, acts as a master clock

47
Q

explain Siffre’s cave study

A

he spent time underground to study his bio rhythms

he was deprived of exposure to natural light and sound but had access to food and drink. he resurfaced in mid September and believed it was mid august. a decade after he spent 6 months in the cave again. his free running bio rhythms was just beyond 24 hrs he fell asleep and woke up on a regular schedule.

this suggests we need external factors like light

48
Q

ao3 evaluation of circadian rhythms

A

practical application shift work and the adverse affects. reduced concentration in the morning and more mistakes. poorer health e.g 3x more likely heart disease. economic implications

PA: education, shift in the timing of the school day. researchers recommended school day start later as its harder for hormonal teens to get sleep theyre sleepier in skl. schools aiming to improve performance will take circadian rhythms into account

used to improve medical treatments. CR coordinate a number of the bodys basic processes like heart rate. they rise and fall during the day so treatments are administered to fit a bio rhythm. E.g asprin for heart issues is best taken at night as heart problems occur in the morning so this timing matters

49
Q

what is an exogenous zeitgeber

A

external cues that may affect or entrain our biological rhytms such as the influence of light on the sleep cycle

50
Q

what is an endogenous pacemaker?

A

the internal body clocks that regulate our biological rhythms such as the influence of the SCN on the sleep cycle

51
Q

Explain the process of melatonin being released or induced

A

the SCN receives info on light levels via the optic nerve.
the SCN signals to the pineal gland to increase production of melatonin at night and decrease it as light levels increase in the morning

52
Q

how does melatonin induce sleep

A

by inhibiting the brain mechanisms that promote wakefulness

53
Q

What happens to the body clock in the absence of external cues?

A

the free running biological clock that conrols the sleep cycle continues to tick in a distinct cyclical pattern, its then brought into line in environmental cues

54
Q

state and explain 2 types of exogenous zeitgebers

A

light - it can reset the SCN and plays a role in the maintenance of sleep cycle

social cues - all babies start with a random sleep cycle, schedules are then imposed by people like meal times and bed times

55
Q

A03 weakness of EP and EZ

A

influence of exogenous zeitgebers is overstated. miles studied man blind from birth who had an abnormal circadian rhythm of 24.9 though hes exposed to social cues like sleep and meal times.

exogenous zeitgebers dont have the same effect in all environments. people in the arctic circle have similar sleep patterns all year round. though they spend 6 months in total darkness. sleep cycle is controlled by endogenous p that override exz

56
Q

A03 strengths of EZ and EP

A

research to support exogenous zeitgebers on sleep wake. Campbell and murphy found lights detected by skin receptor sites. had 15 ptps woken at diff times and light shone on the back of knees. produced deviation in ptps usual sleep cycle of up to 3 hrs. Light is a powerful zeitgeber on not j eyes.

research to support animal studies. DeCoursey destroyed SCN in brains of chipmunks and put them back in the wild, observed for 80 days. their sleep cycle dissapeared so they were killed by predators. SCN is important. No SCN = no sleep cycle. HWVR unethical do findings outweigh procedure?

57
Q

What is an infradian rhythm?

A

type of biological rhythm w a frequency of less than one cycle in 24 hours e.g menstruation and seasonal affective disorder

58
Q

what is an ultradian rhythm?

A

type of biological rhythm with a frequency of more that one cycle in 24 hours e.g stages of sleep

59
Q

how is the menstrual cycle an infradian rhythm?

A

Its an endogenous system thats governed by the monthly changes in the hormone levels

typical cycle takes 28 days to complete

60
Q

how is the menstrual cycle affected by other exogenous factors?

A

diet
stress

61
Q

what is SAD and what are the symptoms

A

It is a depressive disorder which has a seasonal pattern of onset.
persistent low mood and lack of activity and interest in life

62
Q

How may SAD be classed as a circadian rhythm?

A

SAD is triggered during winter where theres less daylight hours. during winter lack of light in morning and evenings means secretion of melatonin is extended which has a knock on effect on the production of serotonin in the brain.

63
Q

A03 evaluation 2 strengths of infradian rhythms

A

SAD has practical application in creating light therapy. light box shines light in the morning and evening to reset melatonin levels in people w SAD, works in 60%. shows external cues affect melatonin which affects sleep wake. HWVER, higher relapse rate than those recieving CBT.

Menstrual cycle can be explained by natural selection. synchronisation of cycles has an evolutionary value. in ancestors time it may have been good for women to menstruate together because babies who lost the mother in childbirth can access milk and survive.

64
Q

A03 weakness of infradian rhythms

A

studies support the menstrual cycle is influenced by exogenous factors like synchronisation. confounding variables like diet and stress affect the cycle. weakness as synchronisation may have been due to chance.no causal relationship

65
Q

what does stage 1 and 2 of the sleep cycle consist of?

A

light sleep, easily awoken, brain wave patterns are slower

66
Q

what happens at stage 3 and 4 of the sleep cycle?

A

deep sleep or slow wave sleep, more difficult to rouse someone at this point.

67
Q

what happens at stage 5 and REM sleep?

A

body is paralysed but brain activity speeds up. rapid eye movement which is associated with dreaming

68
Q

A03 evaluation one strength of ultradian rhythms

A

improved understanding of age related changes in sleep. sleep scientists have observed slow wave sleep(when growth hormone is produced) reduces with age.
sleep deficit explains issues with old age like reduced alertness. this led to the use of relaxation medication

69
Q

A03 evaluation one weakness of ultradian rhythms

A

significant variations between research. Tucker found big differences between ptps in terms of duration of each sleep stage specifically 3 and 4 which are possibly biologically determined suggesting its difficult to describe ‘normal sleep’ in a meaningful way.