8 - Occlusal Trauma Flashcards
the periodontium attempts to adapt to what
occlusal forces
effects of occlusal forces are influenced by what
- magnitude
- direction
- duration
- frequency
increased magnitude of occlusal forces on peridontium results in what
- WIDENED PDL space (increase # and width of PDL fibers)
- INCREASED DENSITY of alveolar bone
PDL best accomodates forces along what axis of tooth
LONG AXIS
___ and ___ can INJURE the periodontium (in terms of direction)
LATERAL FORCES (horizontal) and TORQUE (rotational) can injury the periodontium
what forces are more damaging than intermittent forces
constant forces (increased frequency is more damaging)
what are microscopic alterations of PDL that result in increased tooth mobility
occlusal trauma
tissue injury to ___, ___, and ___ results when occlusal forces exceed the adaptive capacity of periodontium
PDL, alveolar bone, and cementum
tissue injury is also called what
occlusal trauma
what is the INJURY to periodontal tissues (PDL, alveolar bone, cementum) from excessive occlusal forces
occlusal trauma
is occlusal trauma visible histologically
YES
what is the “effect”? what is the “cause”?
effect = occlusal trauma
cause = traumatic occlusion or traumatic occlusal force
what is the FORCE the produces injury to the periodontal tissues
traumatic occlusion or traumatic occlusal force
what does this show
necrosis of PDL (chronic occlusal trauma)
what does this show
cemental tear (acute occlusal trauma)
are dental implants capable of adapting to occlusal forces
NO
does dental implant have PDL
no
in dental implant, there is [horizontal OR vertical] crestal bone loss with traumatic occlusal forces
VERTICAL
acute occlusal trauma results from what type of occlusal impact
ABRUPT
what are examples of acute occlusal trauma
tooth pain, sensitivity to percussion or cold, increased tooth mobility
a patient with a recent restoration on #3, hyperocclusion, and reports exterme sensitivity to biting pressure and percussion has what type of trauma?
treatment?
acute occlusal trauma
tx: occlusal adjustment (tissue injury heals and symptoms subside)
what trauma is more common that acute
chronic occlusal trauma
what develops gradually from occlusal changes
chronic occlusal trauma
what are examples of chronic occlusal trauma
tooth attrition
drifiting
extrusion
bruxism
clenching
what is periodontal inury to a tooth with PREVIOUSLY HEALTHY PERIODONTIUM
primary occlusal trauma
in primary occlusal trauma, does the tooth have loss of attachment of bone loss?
NO!
primary occlusal trauma occurs when occlusal trauma is a result of what?
alterations in occlusal forces
T/F: in primary occlusal trauma, occlusal forces are excessie
TRUE
what is periodontal injury to a tooth with bone loss or attachment loss
secondary occlusal trauma
in what trauma does the tooth have a reduced ability to withstand occlusal forces
secondary occlusal trauma
in secondary occlusal trauma, do previously well-tolerated forces become traumatic
YES
T/F: in secondary occlusal trauma, occlusal forces may be normal or excessive on the tooth
true
what is this an example of:
patient has previously healthy periodontium, new restoration was in hyperocclusion
primary occlusal trauma
what is an example of:
missing posterior teeth, no bone loss, excessive occlusal forces applied to premolars, teeth #4 and #5 are moblie
primary occlusal trauma
what is this example of:
toth #8 have been loss and cannot withstand normal occlusal forces, #8 is mobile and extruded, pathologic tooth migration present
secondary occlusal trauma
what type of occlusal trauma
primary
what type of occlusal trauma
secondary
what type of occlusal trauma
secondary
what are signs of occlusal trauma
- widened PDL
- angular (vertical) defects in bone)
- moibile teeth
- thick lamina dura
- root resorption
- fremitus
what is the vibration of movement felt on crown of tooth as patient occludes
fremitus
how to check for fremitus
- place finger on facial of tooth
- ask pt to gently tap up and down
- feel for vibration or movement of tooth
what is the mobility scale (each class)
Class I = <1 mm
Class II = tooth moves B-L 1-2 mm
Class III - tooth moves B-L >2 mm or is depressible in socket
what are the stages of tissue response to increased occlusal forces
Stage I - injury
Stage II - repair
Stage III - adaptive remodeling
what stage of tissue response:
tissue injury produced by excessive occlusal forces
stage I - injury
what is the most susceptible to Stage I injury from excessive occlusal forces
FURCATIONS
Stage I (injury) results in what
DECREASED:
- mitotic activity
- proliferation of fibroblasts
- collagen formation
- bone formation
can repair occur if forces are diminished or if tooth drifts away from forces?
YES
what stage of tissue response:
damaged tissues removed, new CT fibers, bone, cementum are formed
stage II: Repair
what stage of tissue response:
forces remain traumatic only as long as damage produced exceeds the reparative capacity of the tissues
stage II: repair
what is buttressing bone
response to excessive occlusal forces
what stage of tissue response:
peridontium is remodeled in an effort to create a relationship in which forces are no longer injurious to tisseus
adaptive remodeling
results of adaptive remodeling
Widened PDL, funnel- shaped at osseous crest
Angular defects in bone with no pocket formation
Mobile tooth
what is insufficient occlusal force
hypofunction
what is hypofunction
open bite, absence of tooth antagonist
what happens with hypoofunction
Thinning of PDL
Atrophy of PDL fibers
Osteoporosis of alveolar bone
Reduced bone height
is occlusal trauma reversible
yes
what must occur for occlusal trauma repair to occur
injurious force must be relieved
presence of imflammation from plaque impairs what?
reversibility of traumatic lesions
dental biofilm initiates what
gingivitis and periodontitis
is the marginal gingival affected by occlusal trauma
no
does occlusal trauma cause periodontal pockets or gingivitis
no
occlusal trauma can increase periodontal destruction induced by___
periodontitis
does occlusal trauma cause pocket formation
no
in the presence of plaque induced periodontitis, can traumatic occlusion accentuate periodontitisu
YES
in occlusal trauma, it is important to eliminate ___ AND ___
inflammation and traumatic occlusion
what are factors involved in pathologic tooth migration
- Weakened periodontal support
- Changes in forces exerted on teeth (occlusion, pressure from lips, cheek, tongue)
is drifting the same as tooth migration
NO
does drifting result in destruction of periodontal tissues
NO
what is a common sequela when missing teeth are not replaced
drifting
what is an example of drifting pathway
open contacts -> food impaction -> periodontal disease
what are the genetic disorders associated with periodontitis
- Leukocyte Adhesion Deficiency - Defective transendothelial migration
- Chédiak-Higashi syndrome - Impaired killing of microorganisms by neutrophils
- Papillion-Lefèvre syndrome
- Down syndrome
- Ehlers-Danlos syndrome
- Cyclic Neutropenia
- Hypophosphatasia
is there a genetic component to chronic periodontitis
yes some studies show
twin studies: periodontal conditions of identical twins were often similar
do the genes and enviornment interact to affect periodontitis
yes
is periodontitis a multi-factorial disease
yes
what can be used to determine perio genetics
- oral DNA labs
- MyPerioID report
- interleukin genetics
- perio predict
what do perio positive test results show
patients who test positive overproduce IL-1beta, which triggers heightened inflammation
what are limitations of perio genetic testing
small effects of IL-1 polymorphism makes up tiny fraction of genetic component risk
do smokers have increased expression of clinical inflammation?
NO!! they have DECREASED expression
do smokers have increased or decreased BOP
DECREASED
what is the major risk factor for perio prevalence, extent, and severity
smoking
what is more prevalent and severe in smokers compared to non-smokers
pocket depth, attachment loss, bone loss, tooth loss
former smokers are ___ times more likely to have periodontitisu
1.7
smokers (</= 9 cigs/day) are ___ times more likely to have periodontitis
2.8
heavy smokers (>/=31 cigs/day) are ___ times more likely to have periodontotis
6 times
smoking associated with generalized aggressive periodontitis occurs in what age
young adults (19-30)
on average, smokers are ___times more likely to have periodontitis than non-smokers
4 times
risk for periodontitis [increases or decreases] with the increasing number of years since quitting smoking
DECREASE
are smoking cessation programs part of periodontal therapy
YES
are cigar and pipe smoking effects similar to cig smoking
yes (the disease severity is intermediate between cig smoker and non smoker)
what to e-cig liquid contain
E-liquid contains: Nicotine, flavorings,
propylene glycol or vegetable glycerin
inhalation of flavorings in e-cig are associated with what
Inhalation of flavorings (diacetyl)
associated with bronchiolitis obliterans
(irreversible; “popcorn lung”)
do e-cig promote a unique microbiome
yes
do e-cigs provide a more faborable perio tx response compared to non-smokers? how?
NO! less favorable!
- more sites with PD >/= 5 mm
- similar response to tx as smokers
how do e-cigs lead to oral health harms
may lead to tobacco use in adolsecents who try e-cig
can the benefits of quitting tobacco smoking outweigh oral health harms caused by short-term used of e-cig
yes
frequent recreational cannabis use is associated with what
- deeper DP
- increased CAL
- increased odds of severe periodontitis (compared to non-cannabis users)
topical application of CBS (cannabidiol) can results in what
- decreased periodontal inflammation (by downregulating TNF-alpha)
- may promote gingival enlargement by increasing gingival fibroblast production and inhibiting MMP
what is associated with oral leukoplakia and carcinoma
chewing tobacco
chewing tobacco has increased risk of what
severe periodontitis
what occurs at the site of chewing tobacco placement
localized attachment loss and recession
what this
recession from chewing tobacco
smokers may have increased numbers of what organisms
- Tannerella forsythia
- A.a.
- Porphyromonas gingivalis
- Treponema denticola
- Fusobacterium nucleatum
- Prevotella intermedia
- Parvimonas micra
what are the effects of smoking on immune response
- Impaired neutrophil chemotaxis, phagocytosis, and killing
- Decreased lgG2 production
- Increased release of tissue-destructive enzymes by host
what are examples of tissue-destructive enzymes by host
Tumor necrosis factor-a
Prostaglandin E2
MMP-8 (collagenase)
how is the physiology of smoking affected
- Constriction of gingival blood vessels
- Decreased blood flow to gingival tissues
- Smokers have less oxygen concentration in gingival tissues
what are effects of smoking on response to periodontal therapy
- Less pocket depth reduction following scaling and root planing
- Less bone fill in vertical defects following bone graft
- Increased risk of implant complications and failure (double the risk)
do smokers respond well to perio therapy as non-smokers
NO
what are the 5 A’s to smoking cessation
- Ask (smoking status in med hx)
- Advise (smokers of associations between oral disease and smoking)
- Assess (pt interest in and readiness to quit smoking)
- Assist (brief advice, NRT, rx)
- Arrange (follow-up visit, referral to professional smoking cessation services)
smoking cessation approahces
- willpower
- selfhelp materials
- brief intervention by entist
- nicotine replacement therapy
- varenicline or bupropion
- counseling, cognitive behavioral therapy, hypnosis, acupuncture
occlusal trauma is an injury to what
periodontium
how can primary occlusal trauma be treated
occlusal adjustment
secondary occlusal trauma results from what
inadequate bone support to resist normal or excessive occlusal forces
what genetic disorders is periodontitis associated with
LAD, Trisomy 21, etc.
what is the #1 modifiable risk factor associated with periodontitis
smoking