7 - Role of Dental Calculus and Local Predisposing Factors Flashcards

1
Q

primary cause of gingival inflammation

A

dental biofilm in a susceptible host

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2
Q

local predisposing (secondary) factor of gingival inflammation

A
  1. Calculus
  2. latrogenic Factors
  3. Malocclusion
  4. Complications Associated with Orthodontics
  5. Self-inflicted Injuries
  6. Chewing tobacco
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3
Q

when is mineralized bacterial plaque that forms on teeth, implants, and prostheses (dentures, partials, crowns, retainers)

A

calculus

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4
Q

what color is supragingival calculus

A

white and yellow

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5
Q

what are the common areas to find supragingival calculus

A
  1. buccal of maxillary molars
  2. lingual mandibular incisors
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6
Q

what empties near buccal of maxillary molars

A

stenson’s duct (parotid gland)

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7
Q

what empties near lingual of mandibular incisors

A

wharton’s duct (submandibular gland) and bartholin’s duct (sublingual gland)

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8
Q

what is the source of mineralization for supragingival calculus

A

saliva

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9
Q

what is located below marginal gingiva, not visible, detectable with an explorere or probe, dark brown or green/black, firmly attached, difficult to remove

A

subgingival calculus

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10
Q

when does subgingival calclulus become supragingival

A

if gingiva recedes

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11
Q

what does removal of subgingival plaque and calculus do

A

Reduces gingival inflammation
Reduces probing depths
Gains in clinical attachment (Decrease in clinical attachment level/loss)

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12
Q

can calculus be visible on a radiograph? can this be used to indicated the bottom of the pocket?

A
  • yes
  • location does not indicate bottom of pocket
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13
Q

what is the inorganic composition of calculus (70-90%)

A

Calcium phosphate - largest component
Calcium carbonate
Magnesium phosphate - traces
Crystalline structures

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14
Q

what are crystalline structures found in inorganic calculus and location

A
  1. Hydroxyapatite - most common
  2. Magnesium whitlockite - posterior
  3. Octacalcium phosphate
  4. Brushite - mandibular anterior
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15
Q

what is organic composition of calculus

A
  1. protein-polysaccharide complexes
  2. epithelial cells
  3. leukocytes
  4. microorganisms
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16
Q

what are salivary proteins that are found only in SUPRAgingival calculus

A

protein-polysaccharide complexes

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17
Q

what does calculus attach to on tooth surface

A

organic pellicle on cementum or enamel

18
Q

how does calculus attach to tooth surface

A
  • mechanical locking to surface irregularity
  • close adaptation to depressions of cementum
  • penetration into cementum
19
Q

calculus is plaque that has undergone what

A

mineralization

20
Q

mineralization starts how many days after plaque formation

A

1-14 days

21
Q

plaque hardens thru precipitation of what crystalline salts

A

crystalline calcium phosphate salts

22
Q

do separate foci of calcification increase in size and coalesce to form solid masses of calculus

A

yes

23
Q

does mineralization of plaque generally start intra or extra cellulary

A

Mineralization of plaque generally starts extracellularly.
Some organisms calcify intracellularly.

24
Q

pH is elevated by what

A

pH elevated by CO2 and ammonia produced by bacteria in plaque
Protein degradation
Stagnation

25
Q

how does the precipitation of phosphate salts occurs

A

lowering precipitation constant

26
Q

source of mineralization for:
supragingival calc
subgingival cal

A

supragingival: saliva
subgingival: serum transudate (GCF)

27
Q

what is in anti-tartar toothpaste? how does it work

A

sodium pyrophosphate
- Sodium pyrophosphate is soluble
- Calcium phosphate not yet bonded to tooth will be attracted to sodium pyrophosphate
- Soluble compound forms instead of calcium phosphate (calculus)

28
Q

what is a contributing factor, covered by layer of non-mineralized plaque, and provides nidus for plaque accumulation and retains it close to the gingiva

A

calculus

29
Q

iatrogenic factors in gingival inflammation

A

inadequate dental procedures:

Overhanging margins
Overcontoured restorations
Subgingival margins
Roughness of restoration in the subgingival area
Inadequate marginal fit (short margin)
Open contacts

30
Q

does PRDP favor accumulation of plaque? what does this increase?

A

YES! increases: tooth mobility, gingival inflammation, and perio pocket formation

31
Q

does tooth crowding make plaque control more difficult

A

yes duh

32
Q

does malocclusion have prominent roots of teeth in buccal/lingual version with SMALL quantities of attached gingiva

A

YES

33
Q

is malocclusion susceptible to gingival recession

A

YES

34
Q

how can ortho create dehiscene

A

ortho may move tooth facially thru thin bone

35
Q

malocclusion can be followed by what

A

recession

36
Q

what are complications associated with ortho

A
  • ortho increases plaque retension
  • may lead to gingival inflammation and enlargement
37
Q

what are examples of self-inflicted injuries

A
  1. toothbrush trauma
  2. tobacco pouchkeratosis
  3. chewing tobacco associated with recession
  4. oral jewelry
38
Q

what injuries are associated with oral jewelry

A

recession, bone loss, fractures enamel and porcelain restorations

39
Q

what is the primary etiological factor in gingivitis and periodontitis

A

edntal biofilm

40
Q

plaque calcifies thru precipitation of what

A

calcium phosphate