8 Flashcards

1
Q

High osmolarity causes:

A

Thirst  increased water intake

ADH release  water reabsorbed from urine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Low osmolarity causes:

A

Lack of thirst  decreased water intake

Decreased ADH release  water lost in urine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Capillary pressure - pushes

A

water out of cap, perssure goes up water gos out

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Capillary colloidal osmotic pressure pulls

A

water back into capillaries

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

interstitial fluid pressure -

A

stops movement of fluid from capillary

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

tissue colloidal pressure pulls

A

water from capillary into tissue spaces

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Hydrostatic pressure can be thought of as………. and osmotic pressure can be thought of as ……pressure.

A

.. “pushing pressure,”.., ….“pulling”

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Edema-

A

is fluid in the interstitial space

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Pedal edema-

A

sweeling of the feet

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

dec capillary colloidal osmotic pressure –

A

loss of proteins - albumin is the smallest, decreased proteins control *decreased permeability *

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

inc capillary permeability -

A

increase pores

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q
  • lymphedema
A

obstruction of lymph flow

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Sodium (Na+) - controls

A

ECF(Extracellular) osmolality

most abundant cation - enters in GI - leaves by kidney

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Why would “retaining sodium” cause high blood pressure?

A

Sodium bonds to water

When you have sodium and water being retain when it goes in the veins.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Hypodipsia –

A

drink less elderly

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Polydipsia-
True
False
Psychogentic

A

True, symptomatic- hypothalamus tell you to drink cuz your need water you truly need it.

False, secondary(causes by something else), inappropriate(false), heart is holding water, fluid is not going out to the body hypothalmas thinks you need water.
Kidney-filters , kidney flails , ie excrete all fluids

Psychogenic, compulsive- psychological, SZ Sx drink a lot

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

First response of a fluid hormone is to

A

retain fuild!

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

ADH – anti diuretic hormone –

A

Vasopressin, we should be 70% water! (will also retain Sodium) retains sodium(Na) kick potassium(k). Happens when pain trauma stress
Vasopressin- contracts vessels

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Increase ADH - caused by

A

pain, trauma, nausea, surgery, narcotics, nicotine

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

alcohol lowers

A

ADH- thus why we pee more when we drink

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

diabetes insipidus-(has nothing to glusoce) –

A

frequent peeing unable to concentrate urine - does not respond to ADH usually deficiently

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

syndrome of inappropriate ADH (SIADH) -

A

failure of negative feedback system, pee too much

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Aldosterone supports…. secretion and suports … retention

A

Supports K+ secretion

supports Na+ and H2 retention

24
Q

stress, trauma inc cortisol which increases aldosterone so increase transfer of

A

IntraCellFluid K+ to ECF –results in hypokalemia (excreted)

25
Addison’s)
deficiency in aldosterone effect – (Addison’s) prevents K+ excretion treat with glucose to move K+ back to ICF
26
hypovolemia
dec in ECF
27
inc in ECF
retention (na and h20 resorbed) | Renal failure, heart failure
28
Hyponatremia - < 135 mEq/l – Chart 31-6
if inc h2o, then dec Na+ | Muscle cramps, weakness
29
hypernatremia - > 145 mEq/l – Chart 31-7
if inc sodium in serum, then cell dehydration results
30
Second most abundant cation - major in ICF
K+(Potassium)
31
hypokalemia -
cardiac sx, but not serious Ch 31-8 not usally ACUTE | Slowing down heart rate, less O more tired.
32
hyperkalemia -
if cardiac sx, potentially fatal stops performing – Chart 31-9. too much K HR to much heart contractility. Will have Heart firing in other spots. Cardiac arrhythmia abnormal heart beat.
33
Hyperkalemia raises
resting potential toward threshold Cells fire more easily When resting potential reaches threshold, Na+ gates open and won’t close
34
Calcium
absorbed from GI (intestine) - stored in bone - excreted by kidney
35
Calcium, controls (stable)
neural messages
36
Hypocalcemia
to much firing
37
Hypomagnesemia
``` (Helps with Calcium nerve controlling) - symptoms only if severe malnutrition, malabsorption, laxative abuse usually hypo Ca, hypo K diuretics sx inc neuro excitability ```
38
hypermagnesemia -
rare renal failure, elderly hyporeflexia, dullness
39
``` Acid (H+) Blocks Controls Byproducts of “Food” ```
Blocks Na+ gates Controls respiratory rate Individual acids have different functions: Byproducts of energy metabolism (carbonic acid, lactic acid) Digestion (hydrochloric acid) “Food” for brain (ketoacids)
40
Acidosis - | alkalosis -
DEC in pH | increase pH
41
metabolic - alt bicarb (base) in ECF dec pH, dec bicarb = inc pH, inc bicarb =
=acidosis | =alkalosis
42
respiratory - alt CO2 (acid) dec pH, inc CO2 = inc pH, dec CO2 =
=acidosis | =alkalosis
43
Metabolic acidosis | Increased levels of
ketoacids, lactic acid, etc. | Decreased bicarbonate levels
44
Metabolic alkalosis | Decreased
H+ levels Increased bicarbonate level
45
Co2is toxin in
is toxic to brain
46
Dec pH, dec bicarb | inc respiratory rate to release CO2
Metabolic Acidosis
47
lactic acidosis
-inadequate oxygen,ketoacidosis unavailable carbs so fatty acids mobilized - become ketones if excess
48
lactic acidosis
-inadequate oxygen
49
,ketoacidosis
unavailable carbs so fatty acids mobilized - become ketones if excess
50
CO2 + H2O   H2CO3   H+ + HCO3-
get money
51
work with and explain action potential thresholds and if you have less NA orK+
get money
52
``` Dec pCO2, dec H2CO3, Inc pH hyperventilation lightheaded, dizzy paresthesias short periods of apnea treat with rebreather ```
Respiratory alkalosis
53
Byproducts of energy metabolism Digestion “Food” for brain
Byproducts of energy metabolism (carbonic acid, lactic acid) Digestion (hydrochloric acid) “Food” for brain (ketoacids)
54
Respiratory acidosis
``` Inc pCO2, H2CO3, dec pH acute - narcotic overdose, lung injury chronic - emphysema HA, blurred vision, inc HR and BP - flushed - irritable, muscle twitching ventilation ```
55
``` Dec pCO2, dec H2CO3, Inc pH hyperventilation lightheaded, dizzy paresthesias short periods of apnea treat with rebreather ```
Respiratory alkalosis
56
Byproducts of energy metabolism Digestion “Food” for brain
Byproducts of energy metabolism (carbonic acid, lactic acid) Digestion (hydrochloric acid) “Food” for brain (ketoacids)
57
Respiratory acidosis
``` Inc pCO2, H2CO3, dec pH acute - narcotic overdose, lung injury chronic - emphysema HA, blurred vision, inc HR and BP - flushed - irritable, muscle twitching ventilation ```