7. The Athletic Heart Flashcards

1
Q

What is the HR’s response to dynamic exercise?

A

Roughly a tripling of HR

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2
Q

How does training affect SV and HR?

A
  • Increased SV
  • Lower resting HR
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3
Q

What is SV’s response to dynamic exercise? How?

A
  • Increased filling due to:
    • Venous return
    • Preload
  • Increased emptying due to:
    • Frank Starling Law
    • Increased contractility
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4
Q

How does peripheral resistance respond to the tissues’ increased demand for O2?

A

Decreased peripheral resistance to flow

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5
Q

How is blood flow to the organs redistributed w/ increasing exercise intensity?

A
  • Brain always gets constant amount
  • Flow to the heart and muscles increases proportional to exercise intensity
  • Flow to the kidneys, liver, stomach, intestines, etc. decrease as exercise intensity increases
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6
Q

Arterial and venous pO2 at rest vs. during exercise

A

At rest:

  • Less O2​ extraction from blood
  • More oxygenated blood goes to veins

During exercise:

  • Increase in O2 extraction from blood
  • For same amount of O2 entering arteries, less O2 goes to veins
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7
Q

What is CV drift?

A

A syndrome of CV destabilization during prolonged exercise that occurs w/ high workloads in hot environments

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8
Q

What causes CV drift?

A

Decrease in preload associated w/ relocation of central blood volume to the skin to dissipate metabolic heat

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9
Q

How does CV drift affect SV and systemic and pulmonary arterial pressure?

A
  • Decreased SV
  • Decreased pulmonary arterial pressure
  • Decreased systematic arterial pressure
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10
Q

What are the hemodynamic responses to dynamic exercise?

A
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11
Q

What are the hemodynamic responses to static exercise?

A
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12
Q

How does plasma volume change w/ training? Significance?

A

Increased plasma volume makes blood less viscous and easier to flow

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13
Q

How do HR and BP adapt w/ training?

A

Decreases due to alterations in the balance b/t sympathetic and parasympathetic tone

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14
Q

How does SV adapt w/ training? Why?

A

Increased SV due to:

  • Reduced afterload (aortic pressure)
  • Increased diastolic filling time (slower HR)
  • Increased pressure of right atrial blood (volume load)
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15
Q

What are the benefits of a slower HR?

A

​Allows more time for diastolic filling and coronary perfusion

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16
Q

How is functional reserve altered w/ training?

A

Increased: can do same workload w/ less effort

17
Q

Define orthostasis.

A

Maintaining upright posture

18
Q

What is orthostatic hypotension? Symptoms? Cause?

A
  • Shift of blood from head and heart to veins in legs upon standing
  • Feeling of faintness due to sudden decrease in cerebral perfusion pressure due to reduced arterial blood pressure
  • Most likely due to increased parasympathetic drive in trained athletes which leads to a slight delay in the autonomic nervous response upon standing
19
Q

What is concentric hypertrophy of the heart? Causes?

A

Response heart makes to chronic pressure loading (hypertension) in which the walls start to move toward center

20
Q

What is eccentric hypertrophy?

A

Wall thickens in proportion to increase in blood volume due to increased preload

21
Q

How are sarcomeres added in response to pressure load? What type of hypertrophy does this promote?

A

In parallel –> concentric hypertrophy

22
Q

How are sarcomeres added in response to volume load? What type of hypertrophy does this promote?

A

In series –> eccentric hypertrophy

23
Q

What constitutes normal, athletic hypertrophy of the heart?

A
  • Adaptive
  • Symmetric wall growth
  • Reversible training effect
  • Diastolic function is preserved