5.5 Plant and animal responses Flashcards

You may prefer our related Brainscape-certified flashcards:
1
Q

Define abiotic components

A

components of an ecosystem that are non-living

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Define alkaloids

A

Organic nitrogen-containing bases that have important physiological effects on animals; including nicotine, quinine, strychnine and morphoine.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

Define biotic components

A

components of an ecosystem that are living.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

define pheromone

A

any chemical substance released by one living thing, which influences the behaviour or physiology of another living thing.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

define tannins

A

pherolic compounds, located in cell vaculoes or in surface wax on plants.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

define tropism

A

a directional growth response in which the directionof the response is determined by the direction of the external stimulus.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

give some examples of some of plants tropisms

A

Phototropism - shoots grow towards light, which enables them to photosynthesise

geotropism - roots grow towards the pull of gravity. This anchors them in the soil and helps them to take up water, which is needed for support (to fill vacule to stay tugid) as a raw material for photosynthesis and to help cool the plant. there will also be minerals, such as nutrate in the water needed for the synthesis of amino acids.

Chemotropism - on a flower, pollen tubes grow down the style, attracted by chemicals, towards the ovary where fertilisation can take place.

thigmotropism - shoots of climbing plants, such ivy, wind around other plants or solid structure to gain support.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Explain the effect of cytokinins

A

promote cell division
delay leaf sequence
overcome apical dominance
promote cell expansion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

explain the effect of abscisic acid

A

Inhibits seed germination and growth

Causes stomatal closure when the plant is stressed by low water availabilty

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

explain the effect of auxins

A

Promote cell elongation
inhibit growth of side-shoots
inhibit leaf abscission

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

explain the effect gibberellins

A

promote seed germination and growth of stems.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

explain the effect of ethene

A

Promotes fruit ripening

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

define apical dominance

A

inhibition of lateral buds further down the shoot by chemicals produced by the apical bud at the tip of a plant shoot.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

define auxins

A

plant hormones which are reponsible for regulating plant growth.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

define gibberellins

A

plant hormones which are reponsible for control of stem elongation and seed germination.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

State the obsivation when an shoot tip is broken of the plant.

A

the plant starts to grow from the lateral buds that were previously dormant.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

State the first theory of apical domiance

A

the apical bud produces auxins which inhibits lateral buds from growing, and so when removed latical buds can grow.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Suggest how the scientist tested the effect of oxygen to the apical cut.

A

the oxygen could of produced a hormone which promotes lateral bud growth. to get around this they placed a ring of auxin transport inhibitors below the apex of the shoot. The lateral bud grew.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Explain the suggested season for apical domiance with the evidance from the auxin ring inhibitor.

A

when normal auxin level in lateral buds inhibits growth, whereas low auxin levels promote growth,

However a third variable may be the reason, years later it was discovered that auxin levels increase when apex is cut off, and is caused by two other hormones, Abscisic acid and cytokinins.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

explain the current understanding of apical dominance

A

it was discovered that auxin levels increase when apex is cut off, and is caused by two other hormones, Abscisic acid and cytokinins.

Abscisic acid inhibits bug growth. high auxin in the shoot may keep abscinic acid levels high in the bud. when the bud is removed Abscisic acid conc drops, and lateral starts to grow,

Cytokines - promotes bud growth, directly apling cytokinin to buds can override the apical dominance effect,the apex is a sink for cytokinins produced by roots. when apexs is removed spreads evenly around the plant,

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

describe how a fungus caused disease causes cell elongation.

A

the fungal compounds involed are gibberellins and include gibberellic acid(GA3),

It made rice, maize peas rosette all grow taller.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

explain why cell elongation might not be caused by GA3 in nature.

A

An expirmient which uses concentrations of gibberellins within natural ammounts.

Researcher compared GA1 levels of tall pea plants and dwarf pea plants, which were otherwise genetically identical. GA1 values were higher in taller plants/

It was found that the Le gene (tall gene) causes the production of an enzyme that converts GA20 to GA1.

then a pea plant with a mutation that blocks gibberellin production between ent-kaurene and GA12-aldehyde in the pathway. those plants produce no gibberellin and grow only about 1cm tall.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

describe the synthesis pathway for gibberellins

A
ent-kaurene -> 
GA12-aldehyde ->
GA12 ->
GA52 ->
GA44 ->
GA19 ->
GA20 -> (enzyme produced by Le gene)
GA1
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

define geotropism

A

a directional growth response to gravity`

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

define phototropism

A

a directional growth response to light

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

State what kind of plant cells that can grow and how,.

A

meristem as they are immature and are still capable of dividing.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

state where are apical meristem is found

A

at the tips or apices of roots and shootsm and are responsible for the roots and shoot getting longer

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

State where are lateral bud meristems found.

A

found in the buds these can give rise to side shoots.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

state where the lateral meristem found.

A

it forms a cylinder near the outside of roots and shoots are responsible for roots and shoots getting wide.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

State what causes growth between nodes.

A

intercalary meristem which are located between nodes.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Define autonomous nervous system

A

Part of the nervous system repensible for controlling thge involuntary motor activities of the body.

32
Q

Define CNS

A

The central part of the nervous system composed of the brain and spinal cord.

33
Q

define Peripheral nervous system

A

The sensory and motor nerves connecting the sensory receptors and effectors to the CNS.

34
Q

Define Somatic nervous system.

A

The motor nerurones under conscious control.

35
Q

State the commercial uses of auxins

A

Taking cuttings - Dipping the end of a cutting in rooting powder before planting it encourages root growth. Rooting powder contains auxins and talcum powder!

Seedless fruit - Treating unpollinated flowers with auxin can promote growth of seedless fruit (parthenocarpy), Applying auxin promotes ovule growth, which triggers automatic production of auxin by tissues in the developing fruit, helping to complete the developmental process.

Herbicides - Auxins are used as herbicides to kill weeds. Because they are man0made, plants find them more difficult to break down, and they act within the plant for longer. They promote shoot growth so much that the stem cannot support itslef, buckles and dies.

36
Q

State the commercial uses of cytokinins

A

can be used in tissue culture to help mass-produce plants.
Cytokinins promote but and shoot growth from small pieces of tissue taken from a parent plant. This produces a short shoot with a lot of side branches, which can be split into lots of small plants.

37
Q

state the commercial uses of gibbeellins

A

Gibberellins delay senescence in citrus fruit, extending the time fruits can be left unpicked, and making them available for longer in the shops.
Gibberellins acting with cytokinins can make apples elongate to improve their shape.
without Gibberellins bunches of grapes are very compact; this restricts the growth of individual grapes. with gibberellins, the grapes stalks elongate they are less compacted and the grapes get bigger.

38
Q

state the commercial uses of ethene

A

Speeding up fruit ripening in apples, tomatoes and citrus fruits
Promoting fruit drop in cotton, cherry and walnut
promoting female sex expression in cucumbers reducing chance of self polenation.
promoting lateral growth in some plants.

39
Q

List the functions of auxins

A
  • Involved in tropic responses e.g IAA
  • control cell elongation
  • suppress lateral buds to maintain apical dominace
  • promote root growth in rooting powder
40
Q

Explain why shoots show positive phototropism

A
  1. Indoleacetic acid IAA diffuses to shaded side of shoot tip.
  2. As IAA diffuses down shaded side, it causes active transport of H+ ions into cell wall.
  3. Disroption to H-bonds between cellulose molecules And action of explansins make cell more permeable to water (acid growth hypothesis)
  4. Cells on shaded side elongate faster due to higher turgor pressure.
  5. Shoot bends towards light.
41
Q

Explain why roots show positive gravitropism

A
  1. Gravity causes IAA to accumulate on lower side of root.
  2. IAA inhibits elongation of root cells.
  3. Cells on the upper side of the root eloinate faster, so the root tips bends downwards.
42
Q

How do hormnones stimulate stomata to close?

A
  1. Abscisic acid binds to complementary receptors on guard cell membrane, causing Ca2+ ion cahnnels on tonoplast to open. Ca2+ ions diffuse from vacuole into cytosol.
  2. Postitive feedback triggers other ion chennels to oepn other ions E.G K+ diffuse out of guard cells.
  3. Water potential of guard cell becomes more positive water diffuses out via osmosis.
  4. Guards cell become flaccid so stomata close.
43
Q

What is apical dominance.

A

Phenomenon where during the growth of the shoot, the growth of side shoots does not take place. Maintained by the action of auxin, abscisic acid and cytokinins.

44
Q

Explain the experimental evidence that auxins maintain apical dominance.

A

Auxin production in apex maintains high levels of abscisic acid.
Inhibits growth of side shoots.

When apex is removed.
A) Auxin levels drop, causing abscisic acid levels to drop.
b) cytokinins (initially concentrated near auxin reserve in bud) diffuse evenly to promote bud growth in other parts of plan = lateral buds.

45
Q

Explain the experimental evidence that gibberellins control stem elongation and germination.

A

Stem elongation: Tall plants have higher gibberellin concentrations than dwarf plants.

Germination: mutant seeds with non-functional gibberellin gene do not germinate unless gibberellin is applied externally. Inhibtors of gibberellin porudction prevent germination.

46
Q

How auxins and cytokinins used commercially?

A

Auxins: Rooting powder, growing seedless fruit, herbicides, low concentrations prevent lead And fruit growth, high concentrations promote fruit drop.

Cytokinins: prevent yellowing of lettuce leaves, promotes shoot growth.

47
Q

How are gibberellins and ethene used commercially?

A

Gibberellins: delay senescence in citrus, elongation of apples and grapes stalks, brewing beer for malt production, increase sugar cane yield, speed up seed formation in conifers, prevent lodging.

Ethene: speeds up ripening, promotes lateral growth, promotes fruit drop.

48
Q

Outline the gross structure of the ammalian nervous system.

A
split into 
Peripheral
- somatic (volentary)
- autonomous (involentary)
{
sympathetic,
parasympathetic
}

Central

  • Spinal cord
  • brain
49
Q

Name the two main divisions of the nervous system

A

Structural organisation.

  • Central nervous system (comprised of brain and spinal cord. Specialised system of nerve cells processes stimuli and propagates impulses.)
  • Peripheral nervous system ( all neurons that are not part of the CNS).
50
Q

Name the two main divisions of the peripheral nervous system.

A

Functional organisation:

  • Somatic
  • autonomic
51
Q

Name the two main divisions of the autonomic nervous system.

A

Sympathtic: oftern stimulates effectos (fight or flight response), neurotransmitter noradrenaline, ganglia near CNS.
Parasympatheic: ofter inhibits effectors (rest/digest repsonse), nerualtransmitter acetylchlione, ganglia far from CNS.
Act antagonistically to regulate response of effectors.

52
Q

Descibe the gross structure of the brain.

A

2 hemispheres jopined by band of nerve fibres (corpus callosum.) divided into lobes.

  • Parietal lobe at the top of the brain: movement, orientation, memory, recognition.
  • Occipital lobe at the back of the brain: visual cortex processes signals from the eye.
  • temporal lobes beneath the temples: processes auditory signals.
53
Q

Identify the location fo function of the cerebellum

A

Identify the lcoation and function of the cerebellum

  • controls execution (not initiation of movement e.g timing, balance, coordination, posture.
  • Possible role in cognition eg attention and language.
54
Q

Identify the location and function of the medulla oblongata

A

located at the base of the brain.
Controls a range of autonomous functions, including breathing and heart rate (location of acrdioacceleratory/ deceleratory centres).

Accelerans Nerve, increase heart rate,
Vagus nerve, decrease heart rate.

55
Q

Identify the location and function of the cerebrum

A

uppermost part of the brain is organised into loves which control voluntary functions, e.g. initating movement speech, thought.

56
Q

Identify the location and function of the hypothalamus.

A

Includes anterior pituitary gland (secrete metabolic and repordictive hormones).
Involved in thermo and osmoregulation.

57
Q

Outline wht happens in a simple reflex arc.

A

Receptor detects stimulus -> sensory neuron -> relay neuron in CNS coordinates response -> motor neruton -> response by effector.

Survical benefit: rapid response to potentially dangerous stimuli since only 3 neurons involved, instinctive.

58
Q

Describe the knee jerk reflex.

A

Important for maintaining posture and balance.
1. tapping patterlar tendon stimulates stretch-mediated receptors.
2. Impulse travels sensory -> motor (no interneuron). quadriceps contract. inhibits antagonistic hamstring contraction.
diagnostically useful: multiple kicks = symptom of cerebellar disease, lack of reflex = nervous problems.

59
Q

Describe the blinking reflex

A

Brain stem reflex. Consensual response: Both eyelids close rapidly when just 1 cornea is stimulated by bright light / touch.

Seonsry neuron of trigeminal nerve -> spinal nucleus of triugenminal nerve -> iterneurons -> facial motor nerve -> effector muscle orbicularis oculi.

60
Q

What is a fight or flight response.

A

If brain perceives threat, it stimulates stress responses involing adrenaline.
Triggers physiological changes to prepare body:
pupil dilation, inhibition of digestive system, higher heart rate and stroke volume, greater blood flow to bain for mental awareness, faster metabolic rate.

61
Q

use the secondary messenger model to explain how adrenaline works.

A
  1. adrenaline 1st messenger. Hormone-receptor complex forms.
  2. conformational change to receptor activates G-protein.
  3. Activates adenylate cyclase, which converts ATP to cyclic AMP cAMP.
  4. cAMP 2nd messenger. Activates protein kinase A pathway.
  5. Results in glycogenolysis.
62
Q

Descibe the 3 types of muscle tissue.

A

Striated skeletal muscle consists of multinucleated cells. Antagonistic muscle pairs enable movement.

Smooth involuntary muscle enables walls of blood vessels and intestines to contract.

cardiac muscle consists of branched uninucleated cells. myogenic contration = heartbeat.

63
Q

Describe the gross structure of skeletal muscle

A

Muscle cells are fused together to form bundles of parrallel muscle fibres (myofibrils).

Arrangement ensures there is no point of weakness between cells.

~Each bundle is surrounded by endomycium: loose connective tissue with many cappilaries.

64
Q

describe the microscopic structure of skeletal muscle.

A

Myofibrils: site of contraction.

Sarcioplasm: shared nuclei and cytoplasm with lots of mitochondria and endoplasmic reticulum.

Sarcolemma: folds inwards towards sarcoplasm to form transverse (T) tubules.

65
Q

describe the ultrastructure of a mysofibril.

A

Z-line: boundary between sarcomeres.
I-Band: only actin
A-band: overlap of actin and myosin.
H-zone: only myosin.

66
Q

How is muscle contraction stimulated.

A
  1. Neuromusclar junction: action potential = voltage-gated Ca2+ channels open.
  2. vesicles move towards and fuse with presynaptic membrane.
  3. Exocytosis of acetylcholine (ACh), which diffuses across synaptic cleft.
  4. ACh binds to receptors on Na+ channel proteins on skeletal muscle cell membrane.
  5. influx of Na+ = depolarisation.
67
Q

Explain the role of Ca2+ ions in a muscle contration

A
  1. Action potential moves through T-tubules in sarcoplasm = Ca2+ channels in sarcoplasmic retuculum open.
  2. Ca2+ binds to troponin, triggering conformational change in tropomyosin.
  3. Exposes binding sites on actin filaments so actinomyosin bridges can form.
68
Q

Outline the sliding filament theory.

A
  1. myosin head with ADP attached forms cross bridge with actin.
  2. Power stoke: myosin head changes shape and loses ADP, pulling actin over myosin.
  3. ATP attaches to myosin head, causing it to detach from actin.
  4. ATPase hydrolyses ATP-> ADP + Pi so myosin head can return to original postion.
  5. Myosin head re-attaches to actin further along filament.
69
Q

How does sliding filament action cause a myofibril to shorten

A
  • Myosin heads flex in opposite directions = actin filaments are pulled towards each otehr.
  • Distance between adjacent sarcomere Z lines shortens.
  • sliding filament action occurs up to 100 times per second in multiple sarcomeres.
70
Q

Explain the role of creatine phospate in muscle contarions

A

phophorylation ADP directly to ATP when oxygen for aerobic respiration is limited e.g. during vigorous exercise.

71
Q

State the name and location of the 2 nodes involed in heart contraction.

A
Sinoatrial node (SAN): within the wall of right atrium.
Atrioventricular node(AVN): near lower end of right atrium in the wall that separates the 2 atria.
72
Q

Name the receptors involved in changing heart rate and state their location.

A

Baroreceptors (detect changes in blood pressure):Carotid body

Chemoreceptors (detect changes in pH due to increase in CO2 concentration): carotid body aortic body.

73
Q

How does the body respond to an increase in blood pressure.

A
  1. Baroreceptors send more impulses to cardioinhibitory centre in the medulla oblongata.
  2. More impulses to SAN down vagus nerve VIA parasympathetic nervous system.
  3. Stimulates release of acetylcholine, which decreases heart rate.
74
Q

How does the body respond to a decrease in blood pressure.

A
  1. Baroreceptors send more impulses to cardioacceleratory cenre in the medulla oblongata.
  2. More impulses to SAN via sympathetic nervous system.
  3. stimulates release of noradrenaline, which increases heart rate and strength of contraction.
75
Q

How does the body respond to an increase in CO2 concentration?

A
  1. chemoreceptors detect pH decrease and send more impulses to cardioacceleratory centre of medulla oblongata.
  2. more impulses to SAN via sympatheric nerous system.
  3. Heart rate increase, so rate of blood flow to lungs increases = rate of gas exchange and vetilation rate increases.
76
Q

Describe the structure of nerumusclar junction.

A

synaptic cleft between a presynaptic motor neuron and a skeltal muscle cell.
Act as end of nerual pathway and always stimulates an excitary response.