25. MI Flashcards

1
Q

sx indicating MI

A
  • chest discomfort: heavy, crushing, pressure in the left retrosternal area across the chest; radiating in neck, jaw, L arm/shoulder, epigastrium, or between shoulder blades
  • associated sx: nausea, emesis, diaphoresis, dyspnea
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2
Q

which populations are of particular concern for a silent or atypical MI

A

diabetics, elderly women

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3
Q

pathophysiology of an MI

A

narrowing of coronary arteries d/t erosion, fissuring, or rupture of plaque causing thrombus formation

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4
Q

what can a partial occlusion of coronary arteries cause

A

unstable angina (new and worsening CP) or NSTEMI

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5
Q

describe the progression of CAD/ACS

A

normal heart –> stable angina –> unatable angina –> NSTEMI –> STEMI

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6
Q

what would an ECG indicating subendocardial injury show?

epicardial ischemia/ infarction?

A

R waves normal, ST segment elevated, T wave peaked

diminishing amplitude of R wave, marked ST elevation

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7
Q

what is ischemia and how does it show up on ECG

A
  • deficient blood supply, impairing repolarization

- inverted T wave

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8
Q

what is cardiac injury and how does it show up on ECG

A
  • deficient blood supply, impairing ability to fully polarize
  • ST segment shift
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9
Q

what is infarction and how does it show up on ECG

A
  • dead tissue, unable to depolarize

- widened Q waves

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10
Q

when are troponin levels detectable, when do they peak and for how long can levels be elevated in relation to cardiac necrosis

A
  • detectable 1-4 hours after onset of AMI
  • peak 10-24 hours
  • persist 5-14 days

*renal failure can also cause false positive

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11
Q

how can a NSTEMI be diagnosed

A
  • elevated troponin, CK, CK/MB

- ST depression of T wave inversion in contigious leads

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12
Q

a subendocardial infaction is basically the same thing as ___

A

NSTEMI

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13
Q

how much is the ST segment elevated in STEMI (men vs women)

A

elevated 2+ mm in men (1.5+ mm in women) in absence of LVH

or 1+ mm in more than 2 contigious leads

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14
Q

what other cardiac issue may obscure ST elevation analysis

A

new LBBB, may need serial tracings to determine

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15
Q

ischemia causes ___ d/t ___

A

inverted T waves d/t altered repolarization

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16
Q

muscle injury causes ___

A

ST elevation

17
Q

infarction causes ___ d/t ___

A

Q waves d/t absence of depolarization current from dead tissue and opposite currents from other parts of the heart

18
Q

during recovery, which part of the ECG is the first to return to normals

A

ST segment, followed by T wave

19
Q

anterior wall infarction is caused by occlusion of what artery?

in what leads is this seen?

A

anterior interventricular a

V2-V4 (Q waves and ST elevation)
technically V1-V7; V1 and V2 are septal leads

20
Q

inferior wall infarction (RV infarction) is caused by occlusion of what artery?

in what leads is this seen?

A

right coronary artery

2, 3, AVF (Q waves and ST elevation)

21
Q

lateral wall infarction is caused by occlusion of what artery?

in what leads is this seen?

A

circumflex artery

1, AVL (Q waves and ST elevation)
also V5, V6

22
Q

posterior wall infarction is caused by occlusion of what artery?

in what leads is this seen?

A

posterior interventricular artery

V1-V3

23
Q

how do you determine if a Q wave is significant

A
  • Q wave is = 1/3 amplitude of entire QRS complex

- 1+ mm wide, lasting 0.4 s or more (more than 1 box wide)

24
Q

large Q waves in the absence of ST changes indicates what?

A

old MI

25
Q

what ECG findings indicate a posterior MI

A

large R waves and ST depression in V1-V3