17 - Muscle Function Flashcards

1
Q

How do we asses muscle function clinically?

A

Manual muscle testing (structural unit)

Electromyography (EMG) and nerve conduction studies (functional unit)

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2
Q

How do we do manual muscle testing? What does this test?

A

Typically this tests groups of muscles, not an isolated muscle.

  • Agonist, antagonist, and syngerists
  • Planes of movement
  • Types of muscle contraction
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3
Q

What do electromyography and nerve conduction studies test?

A

Detailed evaluation of muscle and nerve function/interaction.

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4
Q

What are agonists? What is an example?

A

“Primary mover”

Responsible for initiation and execution of a specific action at a joint.

Often considered as a functional muscle group: “elbow flexors” - brachalis

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5
Q

What are antagonists? What are synergists? Give examples of each for the group elbow flexors.

A

Antagonists: oppose of reverse the action of a primary mover.

-the triceps is an antagonist of the elbow flexors

Synergists: assist the prime mover in its action - biceps brachii

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6
Q

How do agonists and antagonists work together to facilitate joint movement? What is the purpose of this?

A

Reciprocal inhibition: skeletal muscles typically function in pairs for maximal muscle effifiency, speed, and control.

  • agonist increases its tone in preparation for full activation
  • antagonist prepares to slow down/stop intended function

The muscle pair needs to coordinate their contractions to avoid injury.

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7
Q

Describe the pathway of reciprocal inhibition in neurologically intact patients?

A
  1. Ia afferent enters the sp cd, synapses on the alpha motor neuron, and causes the agonist to contract
  2. At the same time, the other branch of the Ia afferent synapses on the Ia inhibitory interneuron which synpase on the alpha motor neuron of the antagonist, preventing contraction of that muscle group
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8
Q

Describe the pathway of reciprocal inhibition in neurologically compromised patients?

A

LAck of reciprocal inhibition (lack of descending inhibition) causes spasticity

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9
Q

What are the three planes of movement? Describe each.

A

Frontal/coronal: divides body into anterior and posterior; abduction and adduction movements occur in this plane

Sagittal: divides body into right and left; flexion and extension occur in this plane

Transverse/horizontal: divides the body into top/bottom; internal and external rotation occurs in this plane.

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10
Q

Do the motions for shoulder external rotation, internal rotation, extension, flexion, and posterior reach (internal rotation).

A
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11
Q

Do the motions for external and internal rotation of the hips when they are extended and when they are flexed.

A
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12
Q

Do the motion for heel inversion and eversion. Do the motion for foot supination and promation.

A

Supination of the foot: inversion, adduction, and plantar flexion.

Pronation of the foot: eversion, abduction, and dorsiflection.

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13
Q

You are seeing a 42 yo left handed male with right shoulder pain that started after a fall. His pain is in his lateral shoulder when he reahes above his head. He has some neck pain, but no numbness or tingling. What exams should be performed?

A
  • Range of motion
  • Strength
  • Reflexes
  • Special tests
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14
Q

For the mans functional muscle assessment, what are the shoulder abductors (agonists) and shoulder adductors (antagonights)?

A

Shoulder abductors: deltoid and supraspinatus (first 30 degrees)

Shoulder adductors: Latissimus dorsi, pec major, teres major.

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15
Q

How would you use planes of movement to determine the cause of a winged scapula from a shoulder injury?

A

Planes of movement:

Increased winging with abduction = spinal accessory/trapezius injury

More prominent winging with flexion: long thoracic nerve/serratus anterior injury

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16
Q

What are the two different types of contraction?

A
  1. Isotonic: muscle length changes
  • Concentric, muscle shortens
  • Eccentric: muscle elongates
  1. Isometric: muscle length remains the same
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17
Q

Which type of muscle contraction is capable of producing the greatest amount of force? Why is this?

A

Eccentric contractions

  • require less metabolic energy
  • with max contraction, they can generate much higher tension levels conpared to concentric contractions
  • up to 50% higher

Important concent in rehab of tendinopathies because it generates more force and stimulates collagen growth.

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18
Q

How would you treat a 20yo runner with achilles tendinopathy?

A

Eccentric exercises (muscle elongates).

This is becasue the tendon is damaged and you want to stimulate collagen growth factors.

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19
Q

How would you treat a 75 yo male with coronary artery disease who just suffered a myocardial infarction?

A

Avoid isometric exercises (muscle length remains the same) because you don’t want them to increase your blood pressure and increase preload on the heart.

20
Q

What types of diseases will result in an alteration of the normal recorded electrical signals and recruitment patterns? How can these be diagonsed?

A

Nerve compression, hereditary or acquired diseases, or myopathy.

With EMG and nerve conduction studies.

21
Q

What do EMG and nerve conduction studies test?

A

EMG: skeletal msucle fibers (mostly type 1, slow twitch)

Nerve conduction studies: motor, sensory, and mixed nerves (standard evaluate Ia large myelinated nerve fibers(

22
Q

What is the functional element of the neuromuscular system? What are the component?

A

The motor unit

Lower motor neuron (anterior horn cell), axon, NMJ, and muscle fibers.

23
Q

What is an innervation ratio? How does this vary?

A

of muscle fibers that belong to a single axon (innervated by a single axon)

  • Varies between muscles

Low for fine motor - extraocular muscles

High for gross motor - soleus

24
Q

The higher the innervation ratio, the _____ the force generated by that motor unit. What type of muscle fibers does a single motor unit innervate?

A

Higher innervation ratio = greater force generated

All muscle fibers innervated by one motor unit are of the same muscle fiber.

25
Q

What results from individual axon depolarization? What is the foundation for electromyography?

A

When an individual axon is depol., an AP propagates down the nerve.

Depolarization of the fibers in a motor unit creates an electrical potential called motor unit action potential (MUAP).

Analysis of MUAPs and assessment of baseline electrical signal at rest serves as the foundation for electromyography.

26
Q

How are motor unit action potentials (MUAPs) generated by conraction of an individual muscle recorded? How can this information be used?

A

Using a surface or needle electrode.

This info can be used to monitor that muscles activity during a certain action and asses the integrity of that muscle and the nerve ssupplying it.

27
Q

What is the henneman size principle?

A

Motor untis are recruited from smallest to largest.

Patterns of recruitment indicate neuropathic (reduced) vs myopathic (early) conditions.

28
Q

What are nerve conduction studies? What are two different components that can be studied?

A

A component of electromyography that assess the integrity of the peripheral nervous system.

  • Compound motor APs
  • Sensory nerve APs
29
Q

Describe compound muscle action potentials in nerve conduciton studies? What does the amplitude, latency, and duration tell you? What type of nerve does it test?

A

Amplitude: # of fibers activated

Latency: fastest conducting motor fibers

Duration: synchrony of firing fibers

This is done to test motor nerves.

30
Q

Describe sensory nerve action potentials of nerve conduction studies. How do they differ from compound muscle action potentials?

A

They measure action potentials of sensory nerves and their wave forms are different from compound muscle action potentials.

31
Q

How can planes of movement be used in muscle testing to help with a diagnosis? Use the example of testing ankle eversion/inversion.

A

PRedominance of one or the other is abnormal.

Different plave of movement being utilized by pt indicates problem.

32
Q

What is the goal of nerve conduction studies? What is this depicting?

A

Evaluation of peripheral nerve function

The image shows testing of the median motor (Abductor pollicus brevis) and the median sensory (index finger) nerves.

33
Q

What are the two methods that can be used for electromyography (EMG)?

A

Surface electrodes and needle electrodes (tests type I fibers)

34
Q

What type of contraction is shown in this image?

A

Isometric - shows co-contraction of wrist flexors and extensors (ie making a fist)

35
Q

What causes an upper motor neuron syndrome?

A

A lesion of the upper motor neuron in the CNS: brain or spinal cord

36
Q

What are the positive and negative signs associated with an upper motor neuron syndrome? Describe the associated muscle problem.

A

Positive signs: hyperrefleia/spacticity - overactivity

Negative signs: weakness - underactivity

No inherent muscle atrophy.

37
Q

Describe the spacticity associated with an UMN syndrome.

A

Velocity-dependent increase in tonic stretch reflex (muscle tone) - due to loss of descending inhibition

Hyperexcitability of the stretch reflex

Decreased reciprocal inhibition.

38
Q

What are the agonists of wrist flexion?

A
  • Flexor carpi radialis
  • Flexor carpi ulnaris
  • Flexor digit profundus
  • Flexor digit sublimis
39
Q

What are the antagonists of wrist flexion?

A
  • Extensor carpi radialis longus and brevis
  • Extensor ulnaris
  • Extensor digitorum
40
Q

What are the agonists of hip flexion?

A
  • Iliopsoas
  • Rectus femoris
  • Add long/brev
  • Pectineus
  • Gracilis
  • Sartorius
  • Tansor fasciae latae
41
Q

What are the antagonists of hip flexion?

A
  • Biceps femoris
  • Semitendinosis
  • Semimembranosis
  • Gluteus maximus
  • Adductor magnus
42
Q

What is clonus? When is it seen?

A

A hyperactive stretch reflex with repetitive contract/relax due to lack of reciprocal inhibition.

Seen with UMN syndrome.

43
Q

How can botulinum neurotoxin be used to help muscles?

A

As intramusclar injections to reduce focal muscle overactivity.

  • Produces denervation by pre-synaptically blocking the release of Ach by cleaving SNAP-25 protein.
44
Q

What are some FDA approved uses for using botulinum toxin?

A

Spasticity, dystonia, migraine headaches, neuropathic pain, hyperhydrosis, sialorrhea (hyper salivation), blepharospasm (tight closure of eyelids), and GU.

45
Q
A