12] Burns Flashcards
Who is at risk for burns
Kids
Elderly
Young males with high risk jobs
Types of thermal contact
Flame
Hot liquid
Steam
Thermal-factors influencing severity
Contact time
Temperature
Type of insult
Wet heat from steamor fluid transfers
more heat to the body than a flame
Scald
Exposure to acids, bases, industrial accidents,
assaults
Chemical burns
Factors influencing severity of chemical burns
Contact time
Chemical concentration
Type of chemical
Due to direct damage to DNA
Radiation burn
Recurrent exposure tolow doses of radiation produce erythema, edema and severe induration
Radiation burns
Thermal injury to skin and effect of electrical current
Electrical burn
Electrical burn- dry skin increases risk of skin injury but decreases risk of
Internal injury
Factors influencing electrical burn
Contact time
Voltage
Risk increased by peripheral artery disease, Raynaud’s disease, DM, smoking, use of betablockers, ETOH
Frostbite burn
What areas are injured with frostbite
Nose
Ears
Fingers
Toes
Most severe injury ->irreversible cell injury
– May expand 48 hrs after burn
Zone of coagulation
Less severe injury ->reversible cell injury; sluggish blood flow
Zone of stasis
Inflamed area but expected to fully recover
within 7-10 daysof injury
Zone of hyperemia
4 depth categories of burns
1- superficial
2- superficial partial thickness
3- deep partial thickness
4- full thickness
damage to epidermis with
erythema, pain,
edema
Superficial burn
Ex] sunburn
Superficial burn
Should heal spontaneously in afew days without
scarring
May exfoliate
Superficial burn
Injured epidermis sloughs within a few days
May have itching as it heals
Superficial burns
damage to epidermis
& slightly into papillary Dermis
Superficial partial thickness burn
Caused by scalds, brief contact and brief
flame
Superficial partial thickness burns
Erythema, extreme pain, moderate edema And Blistering (up to 5 days)
Intact sensory receptors
Blanches to pressure with quick capillary
refill
Large amounts of drainage
Should heal in 10-14 days with minimal
scarring
Superficial partial thickness burn
damage to epidermis & deep into dermis with less pain,
edema and eschar
– Extends into reticular layer ofdermis
Deep partial thickness burn
Mottled red and white areas
– Decreased pinprick but intact pressure sensation
Deep partial thickness burn
Blanches to pressure with slow capillary refill
– Heals in 3 weeks with scarring and pigment changes
Deep partial thickness burn
damage to epidermis, dermis, and
subcutaneous tissue
with little to no pain
Full thickness burn
Risk of infection increases – Will not heal on own or would take three to four months – Grafting necessary – May have contractures
Full thickness burn
Mottled appearance
Dry, rigid, leathery eschar
Lack pain, pressure and temperature sensation
Full thickness burn
How do you calculate size of burns
Rule of 9s
Charts used with
assigned percentages to body areas
Used for infants and
young children
Lund and browder method
Goals of subacute phase
Prevent contractures and neuropathies
Maintain function
Ambulate
3 phases of burn care
Emergent
Subacute
Rehab
3 dressings used in burn treatment center
Antibacterial creams
Silver impregnated products
Enzymatic debriders
2 types of antibacterial creams
Silvadene/Silver sulfadiazine
Sulfamylon
2 types of silver impregnated products
Acticoat
Aquacel Ag
1 Type of enzymatic debridement for burns
Santyl
Treatment of superficial burns
Don’t need topical anti microbials
Treatment for superficial partial thickness burns
Can use topical antimicrobial ointment such as bacitracin
Treatment for deep partial thickness burns
Can use topical antimicrobial ointment such as bacitracin
Ace wraps Tubigrip Isotoner/edema gloves Coban Pressure garments Silon/elastomer
Interventions for scarring
Should not mobilize on
Intact scabs
stabilize two areas along a scar while pulling the scar perpendicular
Plucking- scar mob
grasping a segment of the scar
and pulling the skin parallel to the incision
Rolling - scar mob
Overrepair type: Rise above the level of Surrounding skin
Hypertrophic scar
More persistent, tend
to regrow if excised,
and extend beyond the area of injury
Keloid
Loss of inhibition onfibroblasts and has
increased collagen
accumulation
Keloid overrepair